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  • Pathophysiology of CHF

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    Imae Mayo
    5K просмотров1 страница
    Heart failure occurs due to non-modifiable risk factors like age, gender, and valvular disease as well as modifiable lifestyle factors such as smoking, alcohol, obesity, and hypertension. This leads to increased left atrial pressure and blood backing up into the lungs, reducing stroke volume and tissue perfusion while increasing pulmonary capillary pressure and fluid buildup in the lungs causing pulmonary edema, hypoxia, and activation of the renin-angiotensin-aldosterone system to increase blood volume and pressure. Signs include dyspnea, cough, fatigue, and reduced kidney function.

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    Pathophysiology of CHF

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    Heart failure occurs due to non-modifiable risk factors like age, gender, and valvular disease as well as modifiable lifestyle factors such as smoking, alcohol, obesity, and hypertension. This leads to increased left atrial pressure and blood backing up into the lungs, reducing stroke volume and tissue perfusion while increasing pulmonary capillary pressure and fluid buildup in the lungs causing pulmonary edema, hypoxia, and activation of the renin-angiotensin-aldosterone system to increase blood volume and pressure. Signs include dyspnea, cough, fatigue, and reduced kidney function.

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    Attribution Non-Commercial (BY-NC)
    Скачайте в формате DOCX, PDF, TXT или читайте онлайн в Scribd
    Отметить как неприемлемый контент
    5K просмотров1 страница

    Pathophysiology of CHF

    Загружено:

    Imae Mayo
    Heart failure occurs due to non-modifiable risk factors like age, gender, and valvular disease as well as modifiable lifestyle factors such as smoking, alcohol, obesity, and hypertension. This leads to increased left atrial pressure and blood backing up into the lungs, reducing stroke volume and tissue perfusion while increasing pulmonary capillary pressure and fluid buildup in the lungs causing pulmonary edema, hypoxia, and activation of the renin-angiotensin-aldosterone system to increase blood volume and pressure. Signs include dyspnea, cough, fatigue, and reduced kidney function.

    Авторское право:

    Attribution Non-Commercial (BY-NC)
    Скачайте в формате DOCX, PDF, TXT или читайте онлайн в Scribd
    Отметить как неприемлемый контент
    из 1

    Pathophysiology

    Non-modifiable risk factors: - Age - Gender - Valvular heart disease Reduced myocardial contractility Increased cardiac workload Decreased diastolic filling Obstruction of left atrial emptying Modifiable risk factors: - Lifestyle - Smoking - Excessive alcohol consumption - Obesity - hypertension

    Increased left atrial pressure

    Blood dams back into the pulmonary capillary bed

    Decreased stroke volume

    Decreased tissue perfusion Pressure of blood into the pulmonary capillary bed increases

    Fluid shift into the intraalveolar space and interalveolar space

    Increased cellular hypoxia

    Decreased blood flow to the kidney

    Pulmonary edema

    RAAS stimulation

    Signs and symptoms: Dyspnea Paroxysmal nocturnal dyspnea Orthopnea Rales/crackles Moist cough Blood tinged frothy sputum Dizziness Fatigue Weakness

    Vasoconstriction and reabsorption of sodium and water

    Increased blood volume Increased systemic BP

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