Oral medicine

1. Aphthous ulcer, compared with herbes ulcer is: a. More characteristic in histology. b. Leaves scar. "! c. Less response to stress. d. #ccur in lining mucosa. $$$ %yphilis &irst appearance: a. Multiple vesicle. *)('+ b. ,rythematous reaction. -./+ c. 0lcer. $$$ d. 1ullae. 234 child with vesicle on the hard palate with history o& malaise &or 5 days what is the possible diagnosis: 16 herpes simple7. $$$ 86 erythema multi&orm 9hich virus is present in the patient:s mouth all his Li&e; a. <erpes %imple7 b. <erpes =oster c. >arecilla >irus d. ?one o& the above $$$ <erpes %imple7 @ %ymptoms, Areatment and Brevention Cold sores are caused by the <erpes %imple7 >irus. #nce in&ected, they plague the patient &or li&e. <erpes can be treated but not cured. %ymptoms appear brie&ly and then disappearD the disease lies dormant in nerve cells, but it may be reactivated by stress or illness. %hingles %hingles is caused by the varicella =oster virus, which also causes chicEenpo7. F& you have had chicEenpo7, the varicella virus remains in a group o& nerves in your central nervous system, but doesn:t cause any symptoms. Ahis is called a dormant virus. Ahe central nervous system consists o& the brain and spinal cord, which are connected to the nerves in the body. 9hen the virus becomes active again, it causes the symptoms o& shingles. >aricella@=oster virus G>H>I is the cause o& chicEenpo7 and herpes =oster Galso called shinglesI. G<erpes %imple7 G<%>1 *\ :GPZMN STQ) [I JKLMN O . PQRMN STQ) UVMN WXYKZMN Calcium channel blocEers cause increase saliva secretion. e. Arue. &. ]alse. $$$

Ahe most common type o& biopsy used in oral cavity is: g. ,7cisional biopsy. $$$ h. Fncisional biopsy. i. Aspiration through needle. ^. Bunch biopsy. 1_ years old child present with bilateral swelling o& submandibular area, what could be the disease: E. ]ibrous dysphasia. l. Cherubism $$$ m. Bolymorphic adenoma. Cherubis`.the most common presenting sign is a painless,&irm,bilateral enlargement o& the lower &ace. Ft develops n early child hood Ahe scienti&ic evidence in dictating that oral Lichen planus is a apremalignant Lesiona is: a. >ery strong b. ?on@e7istent c. Moderately strong d. 9eaE. $$$ @ Ahe premalignant potential o& oral lichen planus and the malignant trans&ormation rate is cited as _.b@5.5 per cent. 9hether or not patients with atrophic or erosive &orms o& oral lichen planus are more susceptible to malignant change has yet to be proved by long@term prospective studies. cood prognosisD rare malignant trans&ormation G_.de5fI #ral lesions o& lichen planus usually appear as: 9hite striae. $$$ ged plahue. %hallow ulcers. Bapillary pro^ections. 1uilae.

a. b. c. d. e.

Ahe oral lesions o& the lichen planus: a. Are usually pain&ul. b. garely appear be&ore lesion elsewhere on the body. c. May be part o& a syndrome in which lesions also appear on the sEin, con^unctiva and genitalia. $$ d. #&ten appear in nervous, high@strung individuals. e. <eals with scarring. i %Ein sites: &orearm, shin, scalp, genitalia. ,tiology: i 0nEnown i Autoimmune A cellemediated disease targeting basal Eeratinocytes. Lichen Blanus >ariants: reticular Gmost common oral &ormI @ erosive Gpain&ulI @ atrophic, papular, plahue typesD bullous GrareI.

All o& the &ollowing are oral &eatures o& achuired immunode&iciency syndrome AFj% ,kC,BA:

a. b. c. d. e.

Candidiasis. ,rythema multi&orm. $$$ <airy leuEoplaEia. gapidly progressing periodontitis. laposi:s sarcoma.

<airy trichoglossia may be caused by: n. 1road spectrum antibiotic. o. <8o8 mouth wash. p. %ystemic steroid. h. <eavy smoEers. r. All o& the above. $$$ .mnKQMN m)*/ZMN mL(MNX *.opMN pqY. rX"!X stuMN v3w xM yz) Fn hairy tongue, which taste buds increase in Length: ]ili &orm. $$$ mK{K|MN ]ungi &orm. mKn/TMN ]oliate. mK}~*MN Circumvallates. mKTMN

s. t. u. v.

>errucous carcinoma: mKM*MMN mwYQMN w. Malignant. $$$ 7. 1enign. Batient comes to your clinic complaining that the denture become tight, during e7amination you notice nothing, but when the patient stand you notice that his legs bowing GcurvedI. 9hat you suspect: AI Bagets disease.. $$$ Bt come &or checE up, no complaining, a&ter radiograph u see well circumscribe radiolucent area in related to mandibular molar the periphery was radiopahue which not well de&ined border the diagnosis is: simple bone cyst. $$$ %callopped border above in&erior alveolar canal between roots o& mandibular molars, this lesion is: aI solitary cyst. bI aneurysmal bone cyst. cI traumatic bone cystGsimple bone cyst. $$$ @ Ahis is the radiographic &inding &or the the trumatic bone cyst gadiographically, these lesions tend to appear as smoothly outlined radiolucencies that scallop around the roots o& the teeth. Ahey do not displace teeth or resorb roots, and the lamina dura is le&t intact. Ahey may range &rom very small G1 cmI to very large Ginvolving most o& the mandibleI. Ahey tend to occur above the in&erior alveolar canal @ A traumatic bone cyst is not a true cyst since there is no epithelial lining. ]ound mostly in yong people, asymptomic. gadiolucency which appears to scallop around the roots o& teeth. Aeeth are usually vital.

Cyst between roots o& mandibular molars , above the mandibular canal has %callopped border above in&erior alveolar canal, on inspiration straw liEe e7udate, the teeth were vital, no periodontal involvment this lesion is: a. static bone cyst b. sta&ne bone cyst c. aneurismal bone cyst d. unicameral bone cyst$$$ gadiographic radioulucency in the interradicular area: a. Fnvasion o& &urcation. ~XVRMN YZ. m3{!. w b. Beriodontal abcess. ! r*+ NY c. Beriodontal cyst. m/2N QwI mK! r*+ mQKqI Mental &oramen appear in radiograph as radiolucent round area to the area o&: y. Mandibular premolars. $$$ =. Mandibular incisors. aa. Ma7illary canine. gadiographic diagnosis o& a well@de&ined, unilocular radiolucent, area between vital mandibular bicuspias is more liEely to be: a. gesidual cyst. 3p. Kq b. Ahe mental &oramen. $$$ mK!}VMN mp3MN c. A radicular cyst. d. #steoporosis. PtMN |u|" e. ?one o& the above. 9hich cyst is not radiolucent; a. clobuloma7illary cyst. UXYq U*u2 T4 Kq b. ]ollicular cyst. p)Y c. jentigerous cyst. $$$ S -u2 UX+ d. ?asopalatine cyst. mKZwN mKT!LMN J!3MN .Ku *x4 S U*L) w ]emale . %welling in le&t o& mandible, slowly increasing , radio opahue surrounded by radiolucent band: a. #steoma. b. #ssi&ying &ibroma. c. Cementoblastoma. $$$ d. #steosarcoma. gadiolucent are cover the pericornal part o& the 5rd molar is: a@ jentigerous cyst b@ Central Cyst in 7@ ray: 1. gadiolucent with bone e7pansion. 8. gadiolucent with bone resorption $$$ Cyst growth

%everal mechanisms are described &or cyst growth, including: i epithelial proli&eration i internal hydraulic pressure i bone resorption. 9hich o& the &ollowing lesions has more tendency to show well de&ined multilocular radiolucency: a. Lateral periodontal cyst b. %huamous cell carcinoma o& ^aw bones c. Brimordial cyst. d. Ameloblastoma. $$$ e. #steomylitis o& the mandible. 3!MNX PtMN sxMN Bt come &or checE up, no complaining, a&ter radiograph u see well circumscribe radiolucent area in related to mandibular molar the periphery was radiopahue which not well de&ined border the diagnosis is: simple bone cyst gadiographic diagnosis o& bone destructive in the mandible without evidence o& bone &ormation is: a. #steomyelitis. b. Malignancy. $$$ c. ]ibro@osseous lesion. d. ]racture. e. osteoradionecrosis. Malignant lesions destroy bone uni&ormly. Fn ost eomyelitis, areas o& radiographically normal@appearing bone are &rehuently seen between the areas o& destruction. %ehuestra are not present in malignant lesions. A _@year@old man has been treated &or a Gt8nomoI shuamous cell carcinoma by radical radiotherapy. <e has a history o& chronic alcoholism and was a heavy smoEer. %i7 years a&ter treatment, he develops a pain&ul ulcer in the alveolar mucosa in the treated area &ollowing minor trauma. <is pain worsens and the bone became progressively e7posed. <e is treated by a partial mandibular resection with gra&t. Ahe diagnosis is a. Acute osteomylitis b. cerre,s osteomylitis c. #steoradionecrosis $$$ d. Chronic osteomylitis #steoradionecrosis is more in a: ma7illa b: mandible. $$$ c: no di&&erence

gadiographic diagnosis o& bilateral e7pansible radio opahue areas in the canine premolar region o& the mandible is: a. <ematoma. b. gemaining roots.

c. Aours mandibularis. $$$ d. Fnternal oplihue ridge. e. cenial tubercle. Fn radiographs, which disease cause multiple radiolucencies: a. <ypothyroidism. }~ MN ~*(3MN b. <yperparathyroidism. $$$ ~ MN NY4N J) c. gicEet disease. Ahe &ollowing are multilocular radiolucencies in 7@ray ,kC,BA: a. Ameloblastoma. b. #dontogenic Eeratocyst. !/MN mK! mwY3. mQKq c. Adenomatoid #dontogenic cyst. $$$ U MN ~*M Kp !/MN ! Kq d. My7oma. |. ~X central giant cell reparative granuloma @ aneurismal bone cyst @ brown : z) NYRLMN J t. tumor o& hyperparathyroidism @ #dontogenic My7oma6My7o&ibroma e Cherubism @ @ <ypopharyn7 Abscess %imple 1one Cyst: %olitary bone cyst, traumatic bone cyst, hemorrhagic NYRLMN J t. N ~w bone cyst, hemorrhagic cyst, idiopathic bone cavity, unicameral bone cyst e ]ibrous jysplasia @ Mucoepidermoid Carcinoma :JYZ. X J t. 55 years old &emale BA come with slow growing swelling in the angle o& the mandible. gadiograph show radio@opahue with radio@lucent border diagnosis: a. #steoma. b. #steosarcoma. c. Cementoblatoma. $$$ Batient came to your clinic with severe pain, on 7@ray the right side o& the mandible has a radiolucency with a radiopahue border that resembles the sunshine rays. our diagnosis is : AI ossi&ying &ibroma 1I osteosarcoma. $$$ CI acute osteomyelitis Cortical integrity, periosteal reaction and so&t tissue %low@growing lesions o&ten cause e7pansion with cortical bowing, while cortical destruction denotes aggressive in&lammatory or neoplastic lesions. Bresence o& periosteal reaction and so&t tissue is also suggestive o& an in&lammatory or malignant aetiology. %ome types o& periosteal reactions are huite speci&ic, liEe the sunburst type in osteosarcoma.

Beriapical and occlusal radiography and orthopantomography revealed di&&use bone destruction on the le&t side o& the mandible due to the presence o& a lesion o& variable appearance, presenting dense radiopahue, mi7ed and completely radiolucent areas. Ahe

lesion was e7tensive and poorly delimited, with the periosteum showing the classical asunraya reaction on occlusal and periapical radiographs. #steomyelitis most in: 3!MNX PtMN sxMN a. Ma7illa b. Mandible. $$$ JY)oMN m)XYMN pQ N N ~w U*utM Child 18 years old with swelling in the mandibular premolars area, &irst premolar clinically missing, in k ray e7amination we &ound gadiolucent is cover the percoronal part o& the 5rd molar is: 1. jentigerous cyst. $$$ #steomyelitis more common a. Ma7illa b. Mandible $$$ c. Hygoma d. ?asal septum e. Condyle jiabetic patient came to clinic with pain swelling enlarged mandible, on radiograph it showed mouth eaten appearance, your diagnosis is : aI acute osteomyelitis. $$$ bI &ocal sclerosing osteomyelitis. cI di&&use sclerosing. #%A,#M ,LFAF% : gadiographically the amoth@eatena appearance is huite characteristic Batient su&&ering &rom pain in the area o& the mandibular molars with paresthesia in the lower lip. 1y clinical and radiographic e7amination your diagnosis: AI Acute osteomyelitis. $$$ #ral paresthesia may be caused by manipulation or in&lammation o& a nerve or tissues around a nerve, direct damage to a nerve or tissues around a nerve, tum or impinging on or invading a nerve, pnmary neural tumor, and central nervous system tumor. Ahe 7 ray show scattered radiopahue line in the mandible ^aw, the diagnosis will be: A@ Baget disease. $$$ 1@ carres syndrome C@ ]ibrous dysplasia j@ #steosarcoma a. b. c. d. Ahe most common type o& malignant bone tumor o& the ^aws is: #steochondrosarcoma. mK4XYz mK/2 .*q~ #steosarcoma. $$$ Leiomyosarcoma. Qu. mKuz2 .*q~ Chondrosarcoma.

<1> can be transmitted by transplacental: a. Arue. $$$ b. ]alse.

1leeding o& the socEet &ollowing tooth e7traction: 1. Fs always a capillary bleeding in nature. 8. AaEes not less than hal& e day in normal individual. 5. Fs always &avorable i& it is primary type. b. Can be due to the presence o& a nutrient vessel. a. b. c. d. 1 and 8 are correct. 1, 8 and 5 are correct. 1, 5 and b are correct. $$$ All are correct.

Fn sicEle cell anemia, #8 is decreased in oral mucosa: Arue. $$$ ]alse. jestruction o& g1C may cause anemia and it is due to de&ect in cell membrane: Arue. $$$ ]alse. Fmmuno&luorecent test and biopsy are used to diagnosis pemphigus: 3ZMN vK|M |Q) o|MNX 2!/MN MMN ~pN Arue. $$$ ]alse. Measuring blood pressure is one o& vital signs important in medical compromised: a. Arue. $$$ b. ]alse. Cell o& chronic in&lammation: a. Lymphocytics. $$$ )XZ/uMN b. BM?. O*!MN N t. c. ?eutrophils. [tMN Amalgam tattoo is an oral pigmentation lesion: p(" Arue. $$$ ]alse. #ral and perioral cyst &ormed &rom epithelial rest o& serres: Arue. ]alse. $$$

j?A only in&ect human but g?A doesn:t in&ect human: Arue. ]alse. $$$

parotid j0CA is opposite to ma7ilary premolar ma7ilary 1st molar ma7ilary 8nd molar $$$ mandibular 1st molar Ahe parotid duct, also Enown as %tensen:s duct, is the route that saliva taEes &rom the parotid gland into the mouth. Ft passes through the buccal &at, buccopharyngeal &ascia, and buccinator muscle then opens into the vestibule o& the mouth ne7t to the ma7illary second molar tooth. Ahe buccinator acts as a valve that prevents in&lation o& the duct during blowing. gunning along with the duct superiorly is the transverse &acial artery and upper buccal nerveD running along with the duct in&eriorly is the lower buccal nerve Ahe most common benign tumor in oral cavity is: bb. ]ibroma. $$$ZKM ~X cc. Bapilloma. dd. Lipoma./L ~X jental jecEs @ page 1_5_@1_5 ]ibroma: reactive, the most common tumor seen in oral cavity. Ahe most prominent cell in acute in&lammation is: ee. Lymphocyst. &&. Blasma cell. gg. BM?. $$$ hh. ((Z/MN ]lat bone grow by endochondral ossi&ication: ii. Arue. ^^. ]alse. $$$ .4XYzMN [ MN PtM* /!" \X U~N RMN mL{Q/MN tMN S. gadiopacity attached to root o& mandibular molar: EE. #ssi&ying &ibroma. ll. <ypercementosis. $$$ mm. Beriapical cemental dysplasia. ceographic tongue is seen in Bt with: nn. jiabetes. $$$ oo. Fron de&iciency anemia. pp. Bemphigus.3ZMN 1enign migratory glossitis is seen with a &our&old increase in &rehuency in patients with ^uvenile diabetes, possibly due to an increased &rehuency o& elevated amounts o& the <LA@ 11d tissue type. MN Y34X mKQLMNX /)oqNX *YMN -Y. t! 2 MVqX -pMN UYTQMN . wK+ 4NY) 4NYRMN QuMN N -Y. !2 \) w P\NX -mKw*.YxMN NY{[NX KM*ZMN /+ v3!( /MN .N*LMN !2X -K*MN .yN (MN ceographic tongue is always accompanied in patient with: a.jiabetes.

b. ,rythema multi&orm. c.Fron de&iciency. d. Bsoriasis. $$$ ceographical tongue: yXYt. YK pQMN . QuMN -u2 *uMN NY/+ !. S2 J~p2 Z|) X ) ~X ~ YT) . !mMLMN V 4 Y XVMN NV J . MVM ST/) . J t. rK 4 *X S2 mLNX mKu2 m m(} !K+N t 4 STM O M * [LMN Pt. . pM +'. P" [ /L/MN S. STM - mM*Z{MN mu+Y. 4 K+N t 4 JYZM m+'. YK mMLMN V -3p" KLQ/MN S. KM !TM /L/MN YK S. X p) w . Y/tMN (!. 4 -Y/MN psoriasis. 34NY. z) Y! )TM U~* {" XV w -u2 4NYRMN QuMN X p) -Y/MN S. ) tMN !2 . mu)* yN ( MN Ahere may be an association between certain types o& psoriasis Gespecially pustular psoriasisI and geographic tongue. A 81 years old patient who has iron de&iciency anemia, di&&iculty in swallowing, with e7amination o& barium sulphate, you &ound: A. ceographical tongue. 1. 1urning mouth syndrome. $$$ C. ````.. %yndrome j. jiabetic patient Fron de&iciency result in: Candida e LeuEoplaEia @ Aphthous %tomatitis @ %ore tongue Gglossodynia e glossitis e 1urning mouth syndromeI @ Batterson@1rown@lelly syndrome GcA%Ag#F?A,%AF?AL jF%,A%,I Bt came to the clinic complaining &rom soreness in the tongue sore throat the diagnosis is: aI burning mouth syndrome. $$$ bI geographical tongue cI &issure tongue Acute atrophic candidiasis presents as a red patch o& atrophic or erythematous raw and pain&ul mucosa, with minimal evidence o& the white pseudomembranous lesions observed in thrush. Antibiotic sore mouth, a common &orm o& atrophic candidiasis, should be suspected in a patient who develops symptoms o& oral burning, bad taste, or sore throat during or a&ter therapy with broad@spectrum antibiotics. Batients with chronic iron de&iciency anemia may also develop atrophic candidiasis. simple7 herpes KQpMN LMN -Y. !2 z) *) JYR!LMN PMX upMN YQ2 /q

<andicapped Bt with lesion in central nervous system appears to have di&&erent type o& disorder in movement and procedure: hh. %ei=ure.m*w rr. Cerebral palsy. $$$ . u ss. Learning disability. Cerebral palsy`.the disability might involve ``.and uncoordinated or involuntary movements Fntercellular movement o& BM? leuEocytes is called migration: KpMN )YTuM m)*u|MN SK mqYLMN JYRxMN -2 " tt. Arue. $$$

1_

uu. ]alse. cingival hyperplasia related to phenytoin therapy is: Y(MN z. vv. Most common on lingual sur&ace. ww. Most common in older Bt. 77. %trongly related to phenytoin dosage. yy. %trongly related to poor oral hygiene. $$$ 1urning mouth syndrome is a chronic disorder typically characteri=ed by each o& the &ollowing ,kC,BA: ==. Mucosal lesion. $$$ |/MN M m4 aaa. 1urning pain in multiple oral sites. m}Y+ PM bbb. Bain similar in intensity to toothache pain. J M !N PM p) ccc. Bersistent altered taste perception. XVMN mL Y/Q. YK" 9hich o& the &ollowing is the most liEely potential cause o& 1M% G1urning mouth syndromeI: /+N YqN pQMN [ jenture allergy. ddd. %alivary dys&unction. $$$ eee. ?eural dys&unction. &&&. Menopausal changes. /{MN {3wN 9hich o& the &ollowing represents the best pharmacologic therapy &or 1M%: ggg. Antidepressant agents. mTMN Nz. hhh. Corticosteroids. iii. An7iolytic agents. ^^^. Ahere is no therapy o& proven general e&&icacy. $$$mKMt4 F& an underlying cause o& 1M% can be identi&ied GAable 8I, then treatment should be directed toward the source.ab1a 0n&ortunately, treatment based on any o& these possible etiologic &actors is &rehuently ine&&ective,: and no therapy &or 1M% has proven generally e&&ective. Aype F diabetes mellitus can be characteri=ed as: EEE. ?on@insulin@dependent. lll. Adult@ onset. *upM N mmm. letosis@prone. $$$ SKM*QwN -u2 /t. nnn. Accompanied by normal cell activity. Ahe most common &orm o& oral ulcerative disease is: ooo. <%>. ppp. Ma^or aphthous ulcer. OYpTMN mK2'3MN hhh. 1ah^et disease. rrr. Minor aphthous ulcer. $$$ Ahe ma^ority o& primary herpetic in&ections are: mKnuLMN N )N mKpM a. %ymptomatic. b. Asymptomatic. $$$ c. Broceeded by &ever.

11

d. Accompanied by gingival erythema. -./+ e. A, c and d. Aobacco should be considered a risE &actor when planning treatment &or Bt who rehuire: a. Fmplants. b. Beriodontal surgery. c. #ral surgery. d. ,sthetic treatment. e. All o& the above. $$$ child came to u with gray discolouration o& the deciduous incisor also on radiographic e7am. Ahere is dilation o& &ollicle o& the permenant successor what will u do: 1@e7tract the decidous tooth. $$$ 8@start endo 5@observe over time jroplet nuclei containing mycobacterium tuberculosis: mKuQMN JY{Z/MN -u2 m)XLMN NYK{3MN O*w a. jo not cause in&ection. b. %ettle out o& room air huicEly. Y/tQ" c. jo not spread widely in the building. d. gemain airborn &or prolonged period. $$$ mu)* JYZM N*xM 3!" 9hich o& the &ollowing is the cause o& immediat type allergic reaction to late7 products: a. Accelerator. b. Antio7idants. c. Late7 protein. $$$ d. ?icEel. 9hich o& the &ollowing is a benign epithelial neoplasm: PKu U~x ~X a. ghabdomyoma. {|/MN uztMN ~*MN b. ]ibroma. ZKM ~X c. Lipoma. /L ~X d. cranular cell tumor. mKpKpLMN |')MN ~X e. leratoacanthoma. $$$ Y3. T ~X A rhabdomyoma is a benign neoplasm o& striated muscle. Almost all lesions in the oral cavity that are called &ibromas are not true neoplasms A lipoma is a benign neoplasm composed o& &at cells. A granular cell tumor, also called granular cell myoblastoma or AbriEosov:s tumor in the past, is a benign lesion o& the so&t tissues whose origin and nature are not &ully understood. ]or a long time the lesion was considered a benign neoplasm related to muscles. Currently a neurogenic origin seems to be more liEely. A Eeratoacanthoma, also called molluscum sebaceum, is a benign cutaneous lesion that is believed to arise &rom hair &ollicles <istologic e7amination o& a Eeratoacanthoma shows hyperplastic epithelium with carcinoma@liEe &eatures Ahe Eeratoacanthoma: Ft is de&ined as a benign epithelial neoplasm, originated &rom the superior portion o& the sebaceous gland o& the hair &ollicle

18

Ahe &unction o& the periodontal ligament include a. Mechanical &unction b. ]ormative &unction c. ?utritive &unction d. %ensory &unction e. All o& the above. $$$ AnEylosis: a. ?o BjL b. Caused by trauma c. ,7tracted surgically d. All o& the above. $$$ Ahe &ollowing are types o& hamartoma ,kC,BA: 3uI 2 ~XI a. Cementoblastoma $$$. '/MN N~*(. ~X b. Compound odontoma. ! ~X c. Comple7 odontoma. PKu ~X *\X A child came to the clinic with continuous involuntary movement o& his head and e7tremities and di&&iculty in vocal communicationD Ahe condition is described as: a. ,pilepsy. Y b. Cerebral palsy $$$. . u Ahe movement o& water across a selectively permeable membrane is called: a. #smosis. $$$ !MN b. Active transport. rtZMN 3!MN c. ]iltration. "~[N d. ji&&usion. ~w[N #smosis is the net movement o& water across a selectively permeable membrane driven by a di&&erence in solute concentrations on the two sides o& the membrane. Cell that can give more than one type: a. ]ibroblast. KuMN N~*(. b. #dontoblast c. Mesenchymal cell. $$$ Mesenchymal cell : An undi&&erentiated cell &ound in mesenchyme and capable o& di&&erentiating into various speciali=ed connective tissues. Ahe Common disease a&&ecting the submandibular salivary gland is: a. %alivary calculi $$$ b. Bleomorphic adenomas. rTN t. c. >iral sialoadenitis. XYKZMN mKtuMN J MN sxMN d. Fn&ected sialoadenitis. 9hich most common salivary gland neoplasm: ~X Bleomorphic adenoma. $$$ rTN t. U ~X Bleomorphic adenoma is the most common tumor o& the ma^or and minor salivary glands

15

. .*/2 mKtuMN MN K(" MN NY.N /R. S. f_ /)X mKZT!MN J MN K() *\X

ganula is associated with which salivary gland: a@submandiuLar gland. b@sublingual gland. $$$ A ranula is a similar cyst arising in the &loor o& mouth &rom the sublingual gland ganula Can be treated by: a. ,7cision. {3MN @ r(n[N b. Cauteri=ation. TMN c. Fncision. zpMN d. Marsupiali=ation. $$$ K*MN aMarsupiali=ation can be per&ormed be&ore a de&initive e7cisiona Contemporary oral and ma7illo&acial surgery, peterson, bth edition, Bage bb Ahe usual treatment o& ranula is marsupiali=ation`. Ahe pre&erred t7 &or recurrent or persistent ranula is e7cision o& the ranula and sublingual gland. solitary bone cyst management a. anti in&lammatory and &ollow up b. curettage and close c. marsupiali=ation$$$ d. no active management Most common 1enign Aumer in oral cavity is: a. ]ibroma GAmeloblastic &ibromaI. $$$ 9hich o& the &ollowing spaces are bilaterally involved in Ludwig:s angina; a. %ubmandibular masticatory spaces b. %ublingualLat.Bha.space c. %ubmandibularsublingualsubmental. $$$ Ludwig angina remains a potentially lethal disease, rapidly spreading bilateral cellulitis o& the submental, sublingual, and submandibular spaces. Ahe roo& o& mandibular &ossa consist o&: a. Ahin compact bone. $$$ Kq b. %pongy bone. R!Z c. Cancellous bone. R!Z ?eoplasm that spread by Lymphatic &rom the angle o& the mouth reach the: a. Breauricular Lymph nodes. N N*K . b. Mental Lymph nodes. c. %ubmandibular Lymph nodes. $$$ d. Bterygoid ple7us. mK+!RMN JYKZzMN e. ugulo@digastric nodes. SK!{pMN N@mKN*MN 1b

A standard rule o& thumb is that the lymphatic drainage &or any particular region is predicted by the arterial supply o& that region. Ahe lip, cheeE, and anterior gingiva drain to submandibular and submental lymph node groups. Fn addition, the cheeE and upper lip also drain to in&erior parotid nodes. A plastic anemia is caused by: RQ!" 'MN MN Y34 a. Aetracycline. b. Benicillin. c. ,rythromycin. d. %ul&onamide. $$$ P)YpKQMN . Aplastic Anemia goughly hal& o& all aplastic anemias occur as a result o& drugs Gsuch antibiotics as chloramphenicol, sul&onamides, phenylbuta=one 1uta=olidin, and such anticonvulsant agents as mephenytoinI Also: chloramphenicol, phenylbuta=one 1uta=olidin, and such anticonvulsant agents as mephenytoin. Ahe cell primary site o& AAB production is: a. Mitochondria. $$$ N~ 3. b. Lysosomes. mM+ /KQ c. ?ucleus. O*!MN d. ?ucleolus. !*)MN e. >acuoles. N*R4 Ahe organelle most closely associated with the manu&acture o& proteins within the cell: a. gibosome. $$$ Wp)~ b. Lysosome. c. ?ucleolus. d. Cell wall. e. Cell membrane. Ahe pacEing and sorting o& protein is the &unction o&: a. ,ndoplasmic reticulum. mKM*KxMN mTpMN b. colgi apparatus $$$ c. Mitochondria d. ?ucleus Ahe process o& attraction o& neutrophils to a site o& Local tissue in^ury is called: a. Bhagocytosis. m/tu b. jiapedesis. r'QwN c. Chemota7is. $$$ K/Kq sNVRwN d. ,pista7is. y2~

Action o& <istamine: a. >asodilatation. mK2XN *" b. Bermeability. m)*Zw

1d

c. ChemoEinesis. K/Kq oKZL" d. 1roncho. p(} ALL #] A<, A1#>, v}w ~K Cholesterol crystals are &ound in: a. leratocyst. $$$ mwY3. mQKq b. Beriodontal cyst radicular cysts contain brown shimmering &luid because o& the presence o& the cholesterol crystals, whereas odontogenic Eeratocysts contain pale greasy &luid, which may include Eeratotic shuames. Cholesterol crystals are &ound in many odontogenic cysts including gadicular cysts, dentigerous cysts, and odontogenic Eeratocysts. Ahe cyst wall shows nicEs produced by cholesterol crystals, in&lammatory cells, calci&ications and satellite microcysts. Ahe process o& cell engul&ing particle is called: n)oRuM |')MN 'N mKu/2 a. ,ndocytosis. 3MN b. ,7ocytosis. yV} c. Bhagocytosis. $$$ m/tu d. Binocytosis. Q+N Ahe Most common odontogenic cysts in the ^aws are: a. gadicular cyst. $$$ U~V b. leratocyst. Most commonly dentigerous cysts are associated with: a. 0nerupted permanent ma7illary canines b. 0nerupted mandibular third molars. $$$ %pontaneous regression o& bilateral dentigerous cysts associated with impacted mandibular third molars : Article : 1ritish jental ournal Ft is most prevalent in the third molar region and is there&ore a common cause o& a radiolucency associated with the crown o& an impacted third molar 1ilateral jentigerous Cysts @ geport o& an 0nusual Case and geview o& the Literature although it may involve any tooth, the mandibular third molars are the most commonly a&&ected. <istopathologically, dentigerous cyst Lining epithelium may be: J~x m!{p/MN mK!QMN mQKTMN a. Cuboidal in type. TMN ptT. b. %trati&ied shuamous in type. $$$ p {. ZY+ c. geduced enamel epithelium. JY. mK!K. J~x d. All o& the above.

Ahyroglossal duct cysts: mKwQuMN mK}~ MN J!3MN a. Are only &ound in the posterior tongue. b. Clinically present in the Lateral necE tissue. c. May be &ound anywhere along the pathway o& the embryonic thyroglossal duct. $$$ d. Are sometimes called Lympho@epithelial cysts.

Most cyst occur in the midline ,with _f over the thyroid membrane and only 8f within the tongue it sel&. gesidual epithelial elements that do not completely atrophy may give rise to cysts in later li&e. 0nilateral swelling slowly progressing Lesion on the Le&t side o& the mandible. Ahis could be: a. #steoma. /2 ~X b. Cementoblastoma. c. #ssi&ying ]ibroma. $$$ d. #steo@sarcom. Ahe ossi&ying &ibroma is a slow growing ,e7pansile lesion that is usually a symptomatic Ahe body secret antibody against antigen using which cells: A lymphocyte 1 lymphocyte Fn diabetic patient, periodontium a&&ected by which cells: ?eutrophil. -2 " \X pmns Macrophages Cause o& angular cheilitis: a. Loss vertical dimension Bt have complete denture. $$$ b. Autoimmune &actors. !2X UoKu\ uM m)*utMN mKZu|MN !N Kz!" !2X }pN U*/tMN tpMN v3w !2 U~N*(MN mZMN sxMN L) .KQ)X PZMN )NX 4 /RK4 stuMN mMN S. X |MN !/4 mKuZQMN !N 4~ "Atlas Of Oral Medicine " )NX XYq NX !K.KZMNX ) LMN v3wX )*3!tMNX )3tMNX KpMN zKp/M mN !2 L) /q .mK2!/MN NY.NX GmZMN KY" J2I rZN !2 L)X -mKtKp{MN !M U*/tMN tpMN X !N U }4 SK!Q/MN !2 L) /q :Compared to herpetic ulcers...aphthous ulcers are a. %mall si=e. b. Fn mucosa lining. $$$ c. Leave scars. x4 mK2'3MN . - N Y qY) [ 2*!MNX -)YZuM ~Kt. KM m+Y3MN Ypq 4 mK2'3MNX mKnuLMN m+Y3MN SK mw~3/uM .mwY3/MN [ mK|/MN Q!MN 4

?icotine stomatitis: !K"*TK!MN PZMN sxMN a. Balate``.hyper;; b. <yperplasia @ Q!" Y4 c. BricEle cell liEe shape prominent `base`$$$ Q!MN S. mq/ *4 mT ') m3p Areatment o& &ungal in&ections: a. Benicillin b. Aetracyclin c. ?ystatin. $$$

gadiographic diagnosis o& bilateral e7pansile radioopahue areas in the canine premolar area region o& the mandible is aI <ematoma bI gemaining roots cI Aorus mandibularis $$$ dI Fnternal oblihue ridge eI cenial tubercle %yphilis &irst appear as: a. Multiple vesicles b. ,ryhthematous reaction c. 0lcer d. 1ulla 1ody de&ends itsel& by antibodies &rom a. 1 lymphocytes $$$ b. A lymphocytes Blasma cell OY mK(" ) ST/) X cell that produce antibodies a. 1 lymphocytes $$$ b. A lymphocytes Blasma cell %huamous cell carcinoma .mKN~X NY{N e |/MN M NY. e wwN e /MN mt e r*LTMNX pMN :mppQ/MN .N*tMN .m/M. YK \X muL +Y} X zK X NY/+ L)*M T Yx" mK pMN JYTp/MN wYQMN .m/M. p("X mK}X J~p. JY) Q. yN*+ N +Y} T Yx" wYQMN 3X X p" t /K4 -u2 T" PZMN wY S. f_ S. YqI PZMN S/ 2*K YqN }*/MN \ QuMN S. mKZu|MN mKpwRMN yN*LMN GPZMN }X J~XR/MN mK|!QMN m4LMNX QuM mKpwRMN yN*LMN .o)/MN mM t. X K T Jo)/. |')MN mT wY \ PZMN wY S. fd S. Yq %huamous cell carcinoma is derived &rom: sss.,pithelial tissue. $$$ ttt. Connective tissue. a. b. c. d. e. Most common site o& oral shuamous cell carcinoma: Bostero@lateral border o& tongue. $$$ ]loor o& the mouth. 1uccal mucosa. Lip. %Ein. Ahe posterior lateral and ventral sur&aces o& the tongue are the most common sites o& intraoral cancer.

Ahe ma^ority o& introral shuamous cell carcinomas are histologically: a. Boorly di&&erentiated. b. 9ell moderately di&&erentiated. $$$ . LM Jo)/.

c. %pindle cell in type. TMN mKMo. d. Carcinoma in situation. %huamous cell carcinoma is multi&actorial: .N*tMN J t. uuu. Arue. $$$ vvv. ]alse. ,arly shuamous cell carcinoma o& oral cavity present as: a. >esicle. u()*+ b. %essile mass. !2 X q c. An ulcer. d. ged plahue. $$$ e. A white cauli&lower liEe lesion %CC: Clinical appearance Most o&ten seen as a painless ulcer, although may present as a swelling, an area o& leuEoplaEia, erythroleuEoplaEia or erythroplaEia GA reddened patchI, or as malignant change o& long@standing benign tumours or rarely in cyst linings. Bain is usually a late &eature when the lesion becomes superin&ected or during eating o& spicy &oods. ge&erred otalgia is a common mani&estation o& pain &rom oral cancer. Ahe ulcer is described as &irm with raised edges, with an indurated, in&lamed, granular base and is &i7ed to surrounding tissues. .NY/+ 3X m/M. YK m+Y} S. q \ %CC |')MN mKZY+ mwYQMN Z z) QMN v!MN S. .NY/LMN 3pMN *\ pN STM S)~K|MN S)V\ S. Y) . *\ rNQMN NV\ sN* N -mKpKp+ mpu J 23 m mtZ"Y. yN*L oK/) %CC mn|4 Z(MN mK3 . : aA white cauli&lower liEe lesion a mZ(M4 %CB |')MN /KuLMN ~*MN %huamous cell papillomas may present as e7ophytic pedunculated papules with a cauli&lower@liEe appearance. ]irm, &i7ed necE nodes are most to be detected in association with: An ameloblastoma A basal cell carcinoma An odontogenic &ibroma A shuamous cell carcinoma. $$$

a. b. c. d.

%tage Fb disease o& shuamous cell carcinoma: A@A1 ?# M# b@A5 ?# M# c@ A8 ?# M#. $$$ d@Ab ?# M# Atropine : A@ jries secretion such saliva. $$$ 1@ depresses the pulse rate. c @cause central nervous system depression. jrug used to increase saliva is: 1@ anticholinergic. 8@ cholinergic. $$$

5@ antidiabetic b@ anticorticosteroid Fn order to decrease the gastric secretion: a. histamine A antigen ehuivalent b. histamine 1 antigen ehuivalent c. anticholenergic. $$$ d. adrenal steroids jrug used to decrease saliva during impression taEing is: 1. Cholinergic. 8. AntiCholinergic. $$$ 5. Antidiabetic. b. Anticorticosteroid. Atropine: Ft is classi&ied as an anticholinergic drug Fn^ections o& atropine are used in the treatment o& bradycardia Gan e7tremely low heart rateI, asystole and pulseless electrical activity GB,AI in cardiac arrest. Ahis worEs because the main action o& the vagus nerve o& the parasympathetic system on the heart is to decrease heart rate. Atropine blocEs this action and, there&ore, may speed up the heart rate. Atropine:s actions on the parasympathetic nervous system inhibits salivary, sweat, and mucus glands. Atropine induces mydriasis by blocEing contraction o& the circular pupillary sphincter muscle, which is normally stimulated by acetylcholine release @ %copolamine, atropine and ben=tropine are anticolinergic drugs. Ahey decrease @ the &low o& salive.Cholinesterase inhibition is associated with a variety o& acute symptoms such as nausea, vomiting, blurred vision, stomach cramps, and rapid heart rate. :m'|MN U t/MN NY4N )o)X p!MN {p)X stuMN )o) Cholinergic @ U t/MN NY4N v3!"X p!MN )o"X m} LMN *"X stuMN v3!" anticholinergic @ .NYKN SKM*TMN /2 qt)X SKM*q KKN /2 4T) SKXY"N /2 @ pt on long term antibiotic came with systemic Candida: 1@amphotrecin 8@&lucona=ol $$$ 5@nystatin ]lucona=ole d_ mg od is the systemic drug o& choice. C. clabrata, C. Aropicalis, and C. lnusel are &lucona=ole resistant, there&ore, candida subtyping should be per&ormed &or resistant cases. SK"Q!MN Kt*.X -J !t/MN [LM KZ)X U )~X S3+ X + rXw*q*uZMN *\ )x zKp/uM zZ/MN 'tMN . !/ -uN rXw*TK/MNX SQ)Y"*Z.NX - S) KQT\~*uTMN N |N ST/.X v. + X ut. Candida in&ection is a &rehuent cause o&: 1urning mouth Lesion at ^unction between hard and so&t palate and surrounded with psudoepithelium AI hyperplasia in salivary gland. 1I necroti=ing sialometaplasia. $$$

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Areacher collins syndrome characteri=ed by @Bg#c?A<,%FA #] MA?jF1L,. uZ 4 3" @?# ,Ag L#%% @0B9Agj %L0AF?c #] , , @MALAg 1#?, ?#A 9,LL ]#gM,j #g A1%,?C,. $$$ :o!KM*q Yu)Y" m.'. !*MN PtMN ~*/ pQ* uM mT/QMN T underdeveloped cheeE bone @ NY" @ ZM SK!KtMN 'K. *3. !+ @ [N N*K *" @ u|uM uZQMN ZMN. Batient present with de&&ieciency at the malar boneopen bitenormal mental abilitiesD 1@treacher cholins. $$$ 8@cleidocrenial dyspasia 5@eagle syndrome Bt. 9ears complete denture &or 1_ years now he has cancer in the &loor o& the mouth. 9hat is the &irs huestion that the dentist should asE: a@ does your denture is ill &itted b@ smoEing. $$$ G_f o& the cancer o& the &loor o& the mouth is caused by smoEingI c@ Alcohol d@ does your denture impinge the o.mucosa. Gtraumatic causeI Lesion at ^unction between hard and so&t palate and surrounded with psudoepithelium hyperplasia in salivary gland aI ?ecroti=ing sialometaplasia$$$ |')MN :. 3)YZMN vK|MN A &emale patient came to your clinic with dry lips and mouth and bilateral submandibular oedema and ocular dryness. jiagnosis is: aIBolymorphecadenoma bI sialotitis$$$ or: %^gren:s syndrome i& present Ahe compression 6 rela7ation cycle o& e7ternal cardiac compression should be repeated a@ 8 times 6 second b@ _ times 6 minute c@ times 6 second d@ 1__ times 6 minute. $$$ Circulation ]eel &or a carotid pulse. F& it is present, provide 1_ breaths per minute, checEing the pulse &or 1_ sec every 1_ breaths. F& no pulse commence chest compression, at the middle o& the lower hal& o& the sternum, depressing bd cm 1__ times per minute. All these shows honey combed bone radiographically ,kC,BA: a@ Ameloblastoma b@ #dontogenic my7oma cyst c@ #dontogenic Eeratocyst d@ Adenomenated tumor. $$$ Ahe way to remove mucocele is a@ gadiation

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b@ ,7cision $$$ c@ Chemotherapy d@ Cateri=ation Mucocele Fn&rehuently, a mucocele goes away without treatment. 1ut i& some mucoceles remain untreated, they can scar over. our dentist should e7amine any swelling in your mouth. A mucocele usually is removed by surgery. Ahe dentist may use a scalpel or a laser to remove the mucocele. A&terward, the tissue will be sent to a laboratory &or evaluation. Ahere is a chance that a&ter the mucocele is removed another one may develop. %ome doctors use corticosteroid in^ections be&ore trying surgery. Ahese sometimes bring down the swelling. F& these worE, you may not need surgery %chicE test an intradermal test &or determination o& susceptibility to jiphtheria hyper%ensitivity Auberculosis hypersensitivity lepsron Ahe %chicE test, invented between 11_ and 1111 is a test used to determine whether or not a person is susceptible to diphtheria.8 Ft was named a&ter its inventor, 1la %chicE G1e1I, a <ungarian@born American pediatrician. Ahe test is a simple procedure. A small amount G_.1 mlI o& diluted G16d_ MLjI diphtheria to7in is in^ected intradermally into the arm o& the person. F& a person does not have enough antibodies to &ight it o&&, the sEin around the in^ection will become red and swollen, indicating a positive result parotitis with pur0alant e7udate ,what wou will do A. Fmmediate coverage with antibiotic days 1.delay selection antibiotics until Enow culture result C.anti viral drug j.give patint wide spectrum antibiotic until result o& lab. Culture$$$ oung pt came without any complain. juring routine k ray appear between the two lower molar lesion diameter about 8mm e7tend laterally with irregular %hape. 9hats the type o& cyst aI dentigerous cyst bI apical cyst cI radicular cyst Batient come to your clinic complaining that the denture become tight, during e7amination you notice nothing, but when the patient stand you notice that his legs bowing GcurvedI. 9hat you suspect: aI Bagets disease. $$$ bI ``.7 cI ``.7

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cleidocranial dysostosis characteristic: aI supernumerary o& teeth. bI Clevical problems. Fsolation period o& chicEenbo7 should be: @a&ter appear o& rach by weeE @untill vesicle become crusted. $$$ @until carter stage is last *)('LMN Y3O+ Mt) /MN U~ Fts most contagious one day be&ore the onset o& rash and until all vesicle have crusted. A&ter u did upper lower complete denture &or old pt. <e came bacE to the clinic ne7t day complaining o& un com&ort with the denture. A&ter u re checE ,no pain, good occlusion, good pronunciations , but u notice beginning o& in&lammation in the gum and outer margins o& the lips , u will thinE this is due to: 1@ 7erostomia. 8@vit@1 de&iciency 5@ sclero edema kerostomia can cause di&&iculty in speech and eating. Ft also leads to halitosis and a dramatic rise in the number o& cavities, as the protective e&&ect o& saliva:s reminerali=ing the enamel is no longer present, and can maEe the mucosa and periodontal tissue o& the mouth more vulnerable to in&ection Batient comes to your clinic with complete denture &or goutine visit no complenining .juring speech or swallowing or opening the mouth ^ust glossitis Angular Cheilitis and discom&ort increasing while day www. >itamin 1 de&iciency 777. yyy. kerostomia sclero edema

Batient with leuEemia absolute neutrophilic count is 1__ what oral surgeon should do go on the manager a. postpone another day b. worE with prophylactic antibiotic. c. platelets trans&usion

child came to your clinic have leuEaemia number o& neutrophils are G1b__Iwant to e7tract his primary central incisor will you treat him: a. as usual pt@ b. give prophylactic Gantibody I c. give platlets be&ore e7traction@ d. do not e7tract

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KML" (N 'q Q+ 8___ S. } tMN /M J ) K/Kq*M ) M )Y/MN 4 SK3QMN SKMNQuM mpQ!M JKtMN 4 /2 U M 3w *R) '4 mKp Bt came to the clinic complaining &rom pain related to swelling on ma7illary central incisor area with vital to under percussion; 16 periapical cyst 86incisive cystG nasopalatin cystI. $$$ 56 globuloma7illary cyst b6 aneurysmalbone cyst Bt came to the clinic with a lesion con&ined to the middle o& the hard palat, on the clinical e7amination the lesion is &luctuant tender. #n the k@ray radiolucent area btw the two central incisors roots. Ahe diagnosis will be: a@ clubuloma7illary cyst. b@ incisive canal cyst. c@ 1ohn:s nodule. d@ Aneurysmal bone cyst. trigeminal neuralgia treated by carbomi=apine, the ma7 dose per day divided in doses is: a@8__ mg 1@d__mg C@1___mg j@18__mg u. 18__@__ mK.*KMN m2YRMN 0sual Adult jose &or Arigeminal ?euralgia Fnitial dose: 1__ mg orally twice a day Gimmediate or e7tended releaseI or d_ mg orally b .Gtimes a day Gsuspension May increase by up to 8__ mg6day using increments o& 1__ mg every 18 hours Gimmediate or e7tended releaseI, or d_ mg &our times a day. GsuspensionI, only as needed to achieve .&reedom &rom pain. jo not e7ceed 18__ mg6 day .Maintenance dose: b__ to __ mg6day %ome patients may be maintained on as little as 8__ mg6day while others may rehuire as much as 18__ mg6day. At least once every 5 months throughout the treatment period, attempts should be made to reduce the dose to the minimum e&&ective level or to discontinue the drug 9hite lesion bilaterally on cheeE, other member in the &amily has it a @leuEoplaEia b @white sponge nevus. $$$ others 9hite sponge nevus presents as bilateral symmetric white, so&t, spongy, or velvety thicE plahues o& the buccal mucosa @ 9hite sponge nevus, also Enown as Cannon:s disease, <ereditary leuEoEeratosis o& mucosa, and 9hite sponge nevus o& Cannon1 appears to &ollow a hereditary pattern as an autosomal dominant trait.8:_ Although it is congenital in most cases, it can occur in childhood or adolescence. Ft presents in the mouth, most &rehuently as a thicE bilateral white plahue with a spongy te7ture, usually on the buccal mucosa, but sometimes on the labial mucosa, alveolar ridge or &loor o& the mouth

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<ead and necE nevi with multi lesion is: 16,agle syndrome. 86Albert syndrome. $$$ GAlbright syndromeI Albright X Apert S. q *) Albert P m.'. *) [ M*\ "X KuL J*x3MN 3 p) t3 u p("X t. ZKM /2 !(" * S. M" :)NYpM m.'. .[LMN S. f8d 4 X Y "! 2X /(MN jylantin GphynotoinI dont give with : AI aspirin 1Ia=oles CImetronida=ole. $$$ jI all o& the above Bregnant 8d years, bleeding on probing, location on papilla o& anterior area o& the ma7illa, Fsolated: AIgiant cell granuloma 1I pyogenic granuloma Gpregnancy epulisI. $$$ CI giant cell granuloma endocrine and e7ocrine gland is : AI pancreas. $$$ 1I pituitary gland CI thyroid g jI salivary g ,I sweat g Ahe pancreas is a gland organ in the digestive and endocrine system o& vertebrates. Ft is both an endocrine gland producing several important hormones, including insulin, glucagon, andsomatostatin, as well as an e7ocrine gland, secreting pancreatic ^uice containing digestiveen=ymes that pass to the small intestine. Ahese en=ymes help to &urther breaEdown thecarbohydrates, protein, and &at in the chyme Batient complaining &rom pain in the &loor o& the mouth Gbeneath the lower ^awI your diagnosis is related to the salivary glands, whats the best 7@ray to help you: AIpanoramic 1Iocclusal CIsialograph. $$$ G%peciali=ed radiograph &or the %alivary gland disordersI 1ecause the salivary glands consist o& so&t tissue, they cannot be seen on radiographs unless special steps are taEen to maEe them visible. Fn a technihue called sialography. the movement o& polymorphic cells in the gaps o& intracellular to the blood capillary #utside it called: AIporosity mK.Q. 1IslinEing sYQ" CI diapedesis. $$$ r'QwN Child with cle&t palate and cle&t lip with anodontia due to: a@ >an der woude syndrome. $$$

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b@ Areacher Collins syndrome. c@ Baget disease. >an jer 9oude syndrome G>j9%I is a genetic disorder. Beople with >j9% have cle&t lipwith or without cle&t palate, isolated cle&t palate, pits or mucous cysts on the lower lip, andhypodontia. A&&ected individuals have normal intelligence Bt came 8 u with coloration bluish Gor green;; I and blacE in the gingival margins .he said that hi has gasteriointensinal problem.this is caused because o& : a@mercury b@lead c@bismuth. $$$ d@arsen. .m) t/MN m*/LMNX m+Y3MN Nz. KqY" 4 *.opMN '. |Q" Child patient presented with swelling in the buccal and palatal ma7illary anterior area tow days ago, the pathology o& the lesion there is a giant cell, what is the diagnosis: 1. ciant granuloma. 8. <emangioma. 5. `. Central giant cell granuloma GCcCcI is a benign intraosseous lesion o& the ^aws that is &ound predominantly in children and young adults. Although benign, it may be locally aggressive, causing e7tensive bone destruction, tooth displacement and root resorption. Child with anodontia and loss o& body hair, the diagnosis is: 1. jown:s syndrome. 8. ,ctodermal dysplasia. $$$ 5. ]ructose `.. b. jiabetic `. 9hich o& the &ollowing conditions is highly indicated &or the short therapy o& j#A% and Fs directly observed once in the clinic: AI Auberculosis. $$$ 1I <F> CI <1?1 jI Mental Fllness

A huestion about Cleidocranial dysostosis characteristic: Bartly or completely missing collarbones. F& the collarbones are completely missing or reduced to small vestiges, this allows hypermobility o& the shoulders including ability to touch the shoulders together in &ront o& the chest. A so&t spot or larger so&t area in the top o& the head where the &ontanelle &ailed to close. 1ones and ^oints are underdeveloped. Beople are shorter and their &rames are smaller than their siblings who do not have the condition.

Ahe permanent teeth include supernumerary teeth. 0nless these supernumeraries are reabsorbed be&ore adolescence, they will crowd the adult teeth in what already may be an underdeveloped ^aw. Fn that case, the supernumeraries will probably need to be removed to provide space &or the adult teeth. Bermanent teeth not erupting 1ossing GbulgingI o& the &orehead. ypertelorism < pt have lesion in toung which su&aring &rom scar &ever the lesion when remove leave the bleding area under it ...diagnosis is leuEoplahua candida ulcer Child patient with pain&ull herpese simple7 the treatment is acyclovir with a@ >it C b@ Local anesthesia with multi@>itamin c@ Local anesthesia with protine .N KZ. t*/MN Y) |MN *T) } pt PMt{MN rX "!p() KL N m/M. 4N wq N Child 1_ years old came to the clinic with periodontitis associated with the 1ry 8ry dentition with sever generali=ed bone destruction and calci&icationon the general e7amination hyperEeratosis o& hands &eet is noticed the diagnosis is : a. <ypophosphotasia. b. Brepuberty periodontitis. c. Bapillon le&evre syndrome$$$. d. uvenile periodontitis pt. with palm oplanter Eeratosis and periodontitis in permanent dentition is liEely to has: ===. papillon ele&evere syndrome b. downas syndrome c. leuEemia d. hypophosphatesia

jiabatic pt with multiple naevi on the necE and the scalp , and multiple ^aw cyst,,,, ur diagnosis will be: ,agle syndrome. corlin e colt= syndrome$$$ Bierre gobin syndrome. ?on o& the above

Maternal immunity that passed &rom mother to the &etus through the placenta, Fgc, FgA is considered: a@?atural passive immunity.$$$ b@?atural active immunity. c@Achuired passive immunity. d@Achuired active immunity. pt came 8 ur clinic complain &rom his gingiva which bleeds alot with any little pressure, on clincal e7amination u &ound pin point purple dots and general rash liEe o& eccymosis on his body laboratory &inding : highly decrease in platletsGslightly more than 8d___I have a history o& eccymosis and brusing all over his body : @ thrombocytopinic purpura $$ 3u" yo!MN *T) M S. } tMN q N . ~ } ) yo!MN *T) 8d___ S. Yq LKZ(MN 2 q N mKp KML" Kp 'q Q+ U*Z2 gadiolucent structure occupied by a radioopahue structure that &orms a mass o& :disorgani=ed arrangement o& odontogenic tissue $$$a. Comple7 odontome .b. Calci&ying ,pithelial #dontogenic Cyst c. Compound odontome is a white lesion a@ lichen planus b@ cancer c@ heamatoma d@none &ordyce:s spots is on a@ tongue b@ oral mucosa c@ upper lip d@ throat ``Fs an anticoagulant agent a@ aspirin b@ heparin c@ paracetamol d@ eve7 -u2 /t) X 2 q S)~pxMN -{t) BAA juct o& submandibular gland is a@ warton b@ bartholin c@ barvenous d@ stenson leuEoplaEia is present on a@ the mouth b@ eye 8

c@ heart d@ lungs Cranial nerves are a@ 18 nerves b@ 1b c@ 1_ d@ 1 je&iciency o& vit C leads to a@ scurvy b@ anemia c@ ricEet d@ de&ect in blood clotting je&iciency o& vit l leads to a@ scurvy b@ anemia c@ ricEet d@ de&ect in blood clotting je&iciency o& vit j leads to a@ scurvy b@ anemia c@ ricEet tMN SKM d@ de&ect in blood clotting

child years old hearing loss , &lush around his mouth and notches in incisors and bolbuos molars .what is your diagnosis; aI gardner syndrome bI congenital syphilis nicotinic stomatiis : aI acanthosis with Eeratin$$$ bI bricEle cell liEe shape bases pt came with prominence in the &orehead and the potassium sul&ate level increased with curved legs enlarged mandible and ma7illa a. Baget disease patient came to dentist a&ter previous stress&ul procedure complaining o& burning discom&ort o& his lip on e7amination u &ound lesions on the palate, diagnosis is: @ contact dermatitis @ allergy @ aphthous ulcer @ herpes simple7 Gherpetic gingivostomatitisI $$$

/MN tMN ~YT" xQZw NY2N Yq . . Y rN aaaa. aphthous ulcer bbbb. cccc. recurrent herpes ulcer$$$ allergic stomatitis

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