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Specific Phobia
A. B. C. Traumatic experience and conditioning Heritability and neuroticism Phobic conditioning Morers two-factor model
1. Classical conditioning neutral stimuli (CS) with an unconditioned stimuli (UCS) 2. Operant conditioning
D.
II.
Social Phobia
A. Two-factor model
1. 2. Classical conditioning Operant conditioning
B.
C. Cognitive factor: focus on negative self-evaluation (do not believe in themselves; unrealistic negative consequences in response to their behaviour)
III.
3.
Medications
a)
b) Beta blockers (block adrenaline effects that increase the heart rate)
IV.
Panic Disorder
A. Neurobiological factors
1. Locus ceruleus major source of neurotransmitter norepinephrine in the brain which triggers the sympathetic nervous system activity 2. Misfire of the fear circuit system
B.
Classical conditioning
1. Interoceptive conditioning arousal of internal bodily sensations
C.
Cognitive factors
1. 2. 3. Perceived control no control over somatic changes Fear of the bodily sensations Agoraphobia (etiology) a) Fear-of-fear hypothesis driven by negative thoughts about the consequences of having a panic attack in public
V.
2. Stop from seeing sensations as loss of control and see them as harmless sensations that can be controlled
B.
Medication
1. SSRI antidepressants (selective serotonin reuptake inhibitor)
VI.
A. Worry is reinforcing because it distracts people from more powerful negative emotions and images B. Decreased physiological signs of arousal
2. Training in detecting cues to anxiety and strategies to counter negative thoughts 3. 4. Worry only at certain times and keep a diary of the outcomes of worry Focus thoughts on the present moment
B.
Medications
1. 2. 3. 4. Benzodiazepenes Tricyclic antidepressants Buspirone SSRI
B.
C.
1. 2.
Attempt to suppress thoughts about these obsessions Over-occupation with suppressing their obsessive thoughts
IX.
Treatment of OCD
A. B. Behavioural approach ERP (Exposure and Ritual Prevention) Medication
X.
2. Hormones increase sensitivity of receptors to stress hormone cortisol, leading to high levels of norepinephrine 3. People with PTSD have high levels of norepinephrine
C. D.
E.
XI.
B.
Treatment
1. More exposure to fear-provoking stimuli to confront the trauma and gain mastery and anguish anxiety 2. 3. Intervention for acute cases Psychoactive drugs a) 4. Benzodiazepenes and antidepressants
Eye Movement Desensitization and Reprocessing (EMDR) a) Person imagines an object or situation related to the traumatic event b) Eye is made to move across the image back and forth, following the therapists finger c) The person is made to imagine a positive object or situation while following the therapists finger