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-if its in the left ventricle its going to cause sob , crackle alter level of
contcitions
-anytime valvues are involve echo and card cath
hypertrophic cardiomyopathy
-its a genetic disorder parents are asytomatic , happens to atletes a
lot
dysryhtymia are usually vtac and v fib
no vasodilartor blood is not going back up and elevate the fee
restrictive cardiomyopathy
start with left sided failure end up with right sided
preload its atrium down to ventricle
preoload after load affect stroke volume
know formula for mean atrial pressure 60 to sustain vital organs
know what plus pressure is
what can decrease preoload and high pulse pressure
ecg rhytym know what p wave send for , electrical pathway sa node to
peerkunji
know cardiac diet dash diet
know cardiac marker and normal valvue
stemi and no setmie
know what to do when pt comes in with chest pt( 12 lead ecg)
hyperkalemia peak t wave treated with insulin with d 5 if its due to
acidosis you would give bicarb, and then calclium gluconate to
increase the threasal
prep for cardiac cath and post care
trendmil tee and tte
holter monitors ( diabetic use it)
know onset for chronist stable angia( princmetal st is elevated and its
usually depressed)
ppl who have st elevation is an acute mi they dont have a lot of blood
in the coronary artery its total occulasion if tis nonsetmi its usually a
partial and not totally occuled
pqrsti pain , know what to do with onset what can you do 12 hurs
first 12 hours pci within the first 90 minutes when you enter the
emergency , only time not to do is 3 or more occuluded of coronary
artery or left asending artery, if they cant do that they will do the
fibrinolytic ( 6 hr of onset). If they cant do it anymore they will do a
caggabe
which one you can take nitro and it will work
systolic and dilastoic failure
right sided vs left sided