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How to Stop or Slow Down Stroke Damage
I can remember like yesterday, a dear friend in the prime of his life suffered a massive
stroke. My wife and I went to the hospital every day for a few weeks and watched the
medical team work to keep him alive. I will say that there is a time and place for
appropriate medical intervention.
My friend did survive only to be left blind and paralyzed on the right side of his body.
Life has not been same since his stroke.
I pray you never ignore any of the early warning signs of stroke, such as
The sad fact is these people are in the minority simply because traditional
medicine does not look at human biochemistry as it relates to stroke recovery.
As it now stands in standard medical care thrombolytic therapy is the only treatment for
ischemic stroke.
The most commonly used drug for thrombolytic therapy is tissue plasminogen
activator (T-PA). T-PA works best to help people with strokes caused by clots (ischemic
strokes) when it is given right away after stroke symptoms begin. Ideally, one should
receive thrombolytic medications within the first 90 minutes after arriving at the hospital
for treatment.
Unfortunately a 2008 paper published in the New England Journal of Medicine stated
that due to its narrow therapeutic window and complexity of administration, only 3-5% of
patients benefit from plasminogen activator (T-PA) therapy. The paper further
commented that the current treatment for stroke patients is simply not enough to give
good results.
A viable and medically documented answer that sadly is not addressed for the stroke
victim is the recommendation of DHA (docosahexaenoic acid, an omega-3 fatty acid
that is in cod liver oil).
Yes, you read that right.
Giving DHA cut the size of the infarct (the area of brain
--
Now there is something you MUST know to make this effective and that is DHA needs to
This study emphasized the significance of doctors giving as little as 500 mg of DHA
them DHA within five hours, which may cut the size of brain damage 77%!
References:
Belayev, et al, Docosahexaenoic acid therapy of experimental ischemic stroke, Transl