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14 (2013)
Histological slides
48/48a. Glomerulonephritis diffusa extracapillare proliferativa (H+E). Find
inflammatory exudate and cells proliferation (crescents) in Bowman capsule
cavity; proteinous casts and erythrocytes in the lumen of tubules, cytoplasmatic
proteinosis in epithelium of convoluted tubules (if it present).
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Self-learning questions
Remember the histology and physiology of kidney (glomerular filter).
What clinical syndromes of kidney damage do you know and what is characteristic
for them?
What are mostly frequent morphological changes of glomeruli and its results?
16 (2013)
18 (2013)
(2013) 19
Program
1. Damage of the glomeruli, its pathogenesis, structural changes, mechanisms of
progression and clinical syndromes.
2. Pathogenesis, morphology and clinical course of the hematuria/nephritic
syndrome causing (most frequently) by acute proliferative (poststreptococcal,
postinfectious) glomerulonephritis.
3. Pathogenesis, morphology and clinical course of the rapidly progressive
(crescentic) glomerulonephritis.
4. Pathogenesis, morphology and clinical course of the proteinuria/nephrotic
syndrome causing (most frequently) by membranous nephropathy, minimal-change
disease, focal segmental glomerulosclerosis and membranoproliferative
(mesangiocapillary) glomerulonephritis (causing mixed symptoms syndrome).
5. Morphology and clinical course of chronic glomerulonephritis.
6. Pyelonephritis, its pathogenesis, classification, morphology, complications and
causes of death.
7. Urolithiasis, its pathogenesis, morphology, complications and causes of death.
8. Kidney cysts and polycystic kidney disease, its pathogenesis, morphology and
complications.
9. Morphological manifestations of the kidney failure (uremic syndrome).
20 (2013)