Type of pathological
The characteristic features of the disease
condition
Fatty degeneration
(fatty liver)
Amyloid degeneration
(Kidney amyloidosis)
Lung pulmonary
tuberculosis (caseous
necrosis)
The etiology of the disease
Disturbance of fat metabolism may lead
The liver is enlarged, yellow,
to:
soft, and greasy. Accumulation of fat in the hepatocytes
Antigen-antibody reaction may lead to:
The renal parenchyma is
Deposition of amyloid.
thickened and has a starchy-
( Pathologically deposition of
appearance. protein+charbohydrates+mucopolysaccharides
to form Amyloid )
Caseous denotes the whitish,
cheese-like appearance of this
form of necrosis.
Cavitation often occurs when T.B
phagocytes cause digestion of
areas of caseous necrosis in the
lung.
 Destruction of pancreas leads to release
Fatty necrosis of Appearance of the soft chalky, of lipase enzymes which act on lipid
pancreas white areas. content so, if with Ca++
Will give soap.

The cells are granular ("cloudy");

Cloudy swelling of The cells have enlarged ("swollen") Disruption of mitochondrial enzymes in
kidney tubules to the point that the lumen is kidney cells.
obliterated

Protein droplets appear within the

cytoplasm of sick cells. These
droplets appear homogeneous,
Hyaline degeneration glassy, bead-like structures giving Hyaline protein deposition
an appearance known as "hyaline."

Waxy degeneration The striated muscle loses its

Typhoid fever.
(Zinkers degeneration) striation.