Republic of the Philippines

Naval State University

College of Arts and Sciences
NURSING AND HEALTH SCIENCES DEPARTMENT
Naval, Biliran

DRUG THERAPEUTIC RECORD

NAME: Altar, Lucio Jr. Decangchon SEX: Male AGE: 67 years old WARD: Neurological (stroke Station) DATE: 4 24- 2017 SHIFT: 7:00am 3:00pm
DIAGNOSIS: CHIEF COMPLAINTS: PHYSICIAN:

DRUG CLASSIFICATION INDICATIONS SIDE EFFECTS NURSING RESPONSIBILITIES

THERAPEUTIC CLASS: Immediate release only: CNS: dizziness, light BEFORE:
Antianginal chronic stable (effort headedness, headache, Observe 12 rights of drug
BRAND NAME Antihypertensive related) angina. Use alone or athenia, fatigue administration.
Calcium channel blocker Assess for hypersensitivity
with beta blockers.
Nicardipin hydrochloride GI: nausea, hepatic injury Check patients weight,
Immediate release and SR:
blood pressure and
Management of essential CV: peripheral edema, angina, electrolyte imbalance.
GENERIC NAME hypertension alone or with hypotension, arrhythmias, Teach the patient and the
other antihypertensive. bradycardia, AV block, SO about the signs and
2 mg IV: Short term treatment asystole symptoms of dry mouth and
DOSE of hypertension when oral feel thirsty.
Dermatologic: Flushing, rash
use is not feasible.
DURING:
FREQUENCY Instruct the patient to take
the drug with food.
Advise the patient to give a
TIME frequent mouth care or
fluids if he may feel thirsty or
IVTT have a dry mouth.
ROUTE
 REFERENCE
NURSING 2013 DRUG
HANDBOOK; Lippincott,
Williams & Wilkins, page 857-
859
MECHANISM OF ACTION
Inhibits the movement of
calcium ions across the
membranes of cardiac and
arterial muscle cells; calcium is
involved in the generation of
the action of potential in
specialized automatic and
conducting cells in the heart,
in arterial smooth muscle ,
and in excitation contraction
coupling in cardiac muscle
cells.
CONTRAINDICATIONS
Contraindicated with allergy
to nicardipine, pregnancy,
lactation, advanced aortic
stenosis.
Use cautiously with impaired
hepatic or renal function, sick
sinus syndrome, heart block.
SIDE EFFECTS NURSING RESPONSIBILITIES
AFTER:
Monitor patients weight
gain, blood pressure and
electrolyte levels.
Instruct patient to
promptly report adverse
reactions immediately.
 Republic of the Philippines
Naval State University
College of Arts and Sciences
NURSING AND HEALTH SCIENCES DEPARTMENT
Naval, Biliran

DRUG THERAPEUTIC RECORD

NAME: Altar, Lucio Jr. Decangchon SEX: Male AGE: 67 years old WARD: Neurological (stroke Station) DATE: 4 24- 2017 SHIFT: 7:00am 3:00pm
DIAGNOSIS: CHIEF COMPLAINTS: PHYSICIAN:

DRUG CLASSIFICATION INDICATIONS SIDE EFFECTS NURSING RESPONSIBILITIES

THERAPEUTIC CLASS: Reduction of risk of stroke and - Headache Stress that atorvastatin is
Dyslipidaemic Agent heart attack in type 2 diabetes - Flatulence an adjunct to not a
BRAND NAME patients without evidence of - Diarrhoea substitute for low-
heart disease but with other - Nausea cholesterol diet
Atorvastatin Calcium CV risk factors, and - Vomiting Tell patient to take drug at
revascularization procedures - Anorexia the same time each day to
in patients without evidence - Xerostomia maintain its effects
GENERIC NAME of coronary heart disease - angioedema Instruct patient to take a
(CHD) but with multiple risk - myalgia missed dose as soon as
40 mg factors other than diabetes - rash/pruritus possible. If its almost time
DOSE (eg, smoking, HTN, low HDL-C, - alopecia for his next dose, he should
family history of early CHD) - allergy skip the missed dose.
Once a day (HS) Patients with CHD, to reduce - Infection Advise patient to notify
FREQUENCY risks of MI, revascularization - chest pain prescriber immediately if
procedures, hospitalization he develops unexplained
for CHF, and angina Potentially muscle pain, tenderness, or
TIME Fatal: Thrombocytopenia. weakness, especially if
Rhabdomyolysis with acute accompanied by fatigue or
IVTT renal failure. fever.
ROUTE
 REFERENCE MECHANISM OF ACTION CONTRAINDICATIONS SIDE EFFECTS NURSING RESPONSIBILITIES

NURSING 2013 DRUG Atorvastatin competitively Hypersensitivity, active liver

HANDBOOK; Lippincott, inhibits HMG-CoA reductase, disease or unexplained
Williams & Wilkins, page 857- the enzyme that catalyses the
persistent elevations of serum
859 conversion of HMG-CoA to
mevalonic acid. This results in transaminase, porphyria,
the induction of the LDL pregnancy, lactation.
receptors, leading to lowered
LDL-cholesterol
concentration.
Absorption: Rapid from the GI
tract (oral).
Distribution: Protein-binding:
98%.
Metabolism: Extensively
hepatic; converted to active
inhibitors of HMG-CoA
reductase.
Excretion: Faeces (as
metabolites); 14 hr
(elimination half-life).