Mr.

Cruz, a lawyer, smokes three packs of cigarette every day for the past 20
years. Now on his late fifties, he started to notice that his cough has been going
on for more than three months. This has also occurred last year wherein his
cough lasted for almost three months. There is sputum production and he
experiences difficulty of breathing whenever he performs his daily activities.

Contents [hide]
1 Description
2 Classification
o 2.1 Chronic Bronchitis
o 2.2 Emphysema
3 Pathophysiology
4 Epidemiology
 5 Causes
6 Clinical Manifestations
7 Prevention
8 Complications
9 Assessment and Diagnostic Findings
10 Medical Management
o 10.1 Pharmacologic Therapy
o 10.2 Management of Exacerbations
11 Surgical Management
12 Nursing Management
o 12.1 Nursing Assessment
o 12.2 Diagnosis
o 12.3 Planning & Goals
o 12.4 Nursing Priorities
o 12.5 Nursing Interventions
o 12.6 Evaluation
o 12.7 Discharge and Home Care Guidelines
o 12.8 Documentation Guidelines
13 Practice Quiz: Chronic Obstructive Pulmonary Disease (COPD)
14 See Also
15 Further Reading

Description

Nurses care for patients with COPD across the spectrum of care, from outpatient
to home care to emergency department, critical care, and hospice settings.

Chronic Obstructive Pulmonary Disease (COPD) is a condition of

chronic dyspnea with expiratory airflow limitation that does not
significantly fluctuate.
 Chronic Obstructive Pulmonary Disease has been defined by The Global
Initiative for Chronic Obstructive Lung Disease as
a preventable and treatable disease with some significant
extrapulmonary effects that may contribute to the severity in individual
patients.

This updated definition is a broad description of COPD and its signs and
symptoms.

Classification

There are two classifications of COPD: chronic bronchitis and emphysema.

These two types of COPD can be sometimes confusing because there are
patients who have overlapping signs and symptoms of these two distinct
disease processes.
Image source: medcomic.com

Chronic Bronchitis

Chronic bronchitis is a disease of the airways and is defined as the

presence of cough and sputum production for at least 3 months in
each of 2 consecutive years.

Chronic bronchitis is also termed as blue bloaters.

Pollutants or allergens irritate the airways and leads to the production of

sputum by the mucus-secreting glands and goblet cells.
 A wide range of viral, bacterial, and mycoplasmal infections can produce
acute episodes of bronchitis.

Emphysema

Pulmonary Emphysema is a pathologic term that describes an abnormal

distention of airspaces beyond the terminal bronchioles and destruction
of the walls of the alveoli.

People with emphysema are also called pink puffers.

There is impaired carbon dioxide and oxygen exchange, and the exchange
results from the destruction of the walls of overdistended alveoli.

There are two main types of emphysema: panlobular and centrilobular.

o In panlobular, there is destruction of the respiratory bronchiole,

alveolar duct, and alveolus.

o All spaces in the lobule are enlarged.

o In centrilobular, pathologic changes occur mainly in the center

of the secondary lobule.

Pathophysiology

In COPD, the airflow limitation is both progressive and associated with an

abnormal inflammatory response of the lungs to noxious gases or particles.
Image source: pathophys.org

An inflammatory response occurs throughout the proximal and peripheral

airways, lung parenchyma, and pulmonary vasculature.

Due to the chronic inflammation, changes and narrowing occur in the

airways.

There is an increase in the number of goblet cells and enlarged

submucosal glands leading to hypersecretion of mucus.

Scar formation. This can cause scar formation in the long term and
narrowing of the airway lumen.

Wall destruction. Alveolar wall destruction leads to loss of alveolar

attachments and a decrease in elastic recoil.

The chronic inflammatory process affects the pulmonary vasculature and

causes thickening of the vessel lining and hypertrophy of smooth muscle.

Schematic Diagrams:

o COPD Emphysema Schematic Diagram

o COPD Schematic Diagram

Epidemiology

Mortality for COPD has been increasing ever since while other diseases have
decreasing mortalities.

COPD is the fourth leading cause of death in the United States.

COPD also account for the death of 125, 000 Americans every year.
 Mortality from COPD among women has increased, and in 2005, more
women than men died of COPD.

Approximately 12 million Americans live with a diagnosis of COPD.

An additional 2 million may have COPD but remain undiagnosed.

The annual cost of COPD is approximately $42.6 billion with overall

healthcare expenditures of $26.7 billion.

Causes

Causes of COPD includes environmental factors and host factors. These

includes:

Smoking depresses the activity of scavenger cells and affects the

respiratory tracts ciliary cleansing mechanism.

Occupational exposure. Prolonged and intense exposure to

occupational dust and chemicals, indoor air pollution, and outdoor air
pollution all contribute to the development of COPD.

Genetic abnormalities. The well-documented genetic risk factor is a

deficiency of alpha1- antitrypsin, an enzyme inhibitor that protects the
lung parenchyma from injury.

Clinical Manifestations

The natural history of COPD is variable but is a generally progressive disease.

Chronic cough. Chronic cough is one of the primary symptoms of

COPD.
 Sputum production. There is a hyperstimulation of the goblet cells and
the mucus-secreting gland leading to overproduction of sputum.

Dyspnea on exertion. Dyspnea is usually progressive, persistent, and

worsens with exercise.

Dyspnea at rest. As COPD progress, dyspnea at rest may occur.

Weight loss. Dyspnea interferes with eating and the work of breathing is
energy depleting.

Barrel chest. In patients with emphysema, barrel chest thorax

configuration results from a more fixed position of the ribs in the
inspiratory position and from loss of elasticity.

Prevention

Prevention of COPD is never impossible. Discipline and consistency are the keys
to achieving freedom from chronic pulmonary diseases.

Smoking cessation. This is the single most cost-effective intervention

to reduce the risk of developing COPD and to stop its progression.

Healthcare providers should promote cessation by explaining the risks of

smoking and personalizing the at-risk message to the patient.

Complications

There are two major life-threatening complications of COPD: respiratory

insufficiency and failure.
 Respiratory failure. The acuity and the onset of respiratory failure
depend on baseline pulmonary function, pulse oximetry or arterial blood
gas values, comorbid conditions, and the severity of other complications
of COPD.

Respiratory insufficiency. This can be acute or chronic, and may

necessitate ventilator support until other acute complications can be
treated.

Assessment and Diagnostic Findings

Diagnosis and assessment of COPD must be done carefully since the three main
symptoms are common among chronic pulmonary disorders.

Health history. The nurse should obtain a thorough health history from
patients with known or potential COPD.

Pulmonary function studies. Pulmonary function studies are used to

help confirm the diagnosis of COPD, determine disease severity, and
monitor disease progression.

Spirometry. Spirometry is used to evaluate airway obstruction, which is

determined by the ratio of FEV1 to forced vital capacity.

ABG. Arterial blood gas measurement is used to assess baseline

oxygenation and gas exchange and is especially important in advanced
COPD.

Chest x-ray. A chest x-ray may be obtained to exclude alternative

diagnoses.

CT scan. Computed tomography chest scan may help in the differential

diagnosis.
 Screening for alpha1-antitrypsin deficiency. Screening can be
performed for patients younger than 45 years old and for those with a
strong family history of COPD.

Chest x-ray: May reveal hyperinflation of lungs, flattened diaphragm,

increased retrosternal air space, decreased vascular markings/bullae
(emphysema), increased bronchovascular markings (bronchitis), normal
findings during periods of remission (asthma).

Pulmonary function tests: Done to determine cause of dyspnea,

whether functional abnormality is obstructive or restrictive, to estimate
degree of dysfunction and to evaluate effects of therapy, e.g.,
bronchodilators. Exercise pulmonary function studies may also be done to
evaluate activity tolerance in those with known pulmonary
impairment/progression of disease.

The forced expiratory volume over 1 second (FEV1): Reduced FEV1 not
only is the standard way of assessing the clinical course and degree of
reversibility in response to therapy, but also is an important predictor of
prognosis.

Total lung capacity (TLC), functional residual capacity (FRC), and residual
volume (RV): May be increased, indicating air-trapping. In obstructive
lung disease, the RV will make up the greater portion of the TLC.

Arterial blood gases (ABGs): Determines degree and severity of

disease process, e.g., most often Pao2is decreased, and Paco2 is normal or
increased in chronic bronchitis and emphysema, but is often decreased
in asthma; pH normal or acidotic, mild respiratory alkalosis secondary to
hyperventilation (moderate emphysema or asthma).

DL CO test: Assesses diffusion in lungs. Carbon monoxide is used to

measure gas diffusion across the alveocapillary membrane. Because
carbon monoxide combines with hemoglobin 200 times more easily than
 oxygen, it easily affects the alveoli and small airways where gas exchange
occurs. Emphysema is the only obstructive disease that
causes diffusion dysfunction.

Bronchogram: Can show cylindrical dilation of bronchi on inspiration;

bronchial collapse on forced expiration (emphysema); enlarged mucous
ducts (bronchitis).

Lung scan: Perfusion/ventilation studies may be done to differentiate

between the various pulmonary diseases. COPD is characterized by a
mismatch of perfusion and ventilation (i.e., areas of abnormal ventilation
in area of perfusion defect).

Complete blood count (CBC) and differential: Increased hemoglobin

(advanced emphysema), increased eosinophils (asthma).

Blood chemistry: alpha1-antitrypsin is measured to verify deficiency and

diagnosis of primary emphysema.

Sputum culture: Determines presence of infection, identifies pathogen.

Cytologic examination: Rules out underlying malignancy or allergic

disorder.

Electrocardiogram (ECG): Right axis deviation, peaked P waves

(severe asthma); atrial dysrhythmias (bronchitis), tall, peaked P waves in
leads II, III, AVF (bronchitis, emphysema); vertical QRS axis
(emphysema).

Exercise ECG, stress test: Helps in assessing degree of pulmonary

dysfunction, evaluating effectiveness of bronchodilator therapy,
planning/evaluating exercise program.

Medical Management
Healthcare providers perform medical management by considering the
assessment data first and matching the appropriate intervention to the existing
manifestation.

Pharmacologic Therapy

Bronchodilators. Bronchodilators relieve bronchospasm by altering

the smooth muscle tone and reduce airway obstruction by allowing
increased oxygen distribution throughout the lungs and improving alveolar
ventilation.

Corticosteroids. A short trial course of oral corticosteroids may be

prescribed for patients to determine whether pulmonary function improves
and symptoms decrease.

Other medications. Other pharmacologic treatments that may be used

in COPD include alpha1-antitrypsin augmentation
therapy, antibiotic agents, mucolytic agents, antitussive
agents, vasodilators, and narcotics.

Management of Exacerbations

Optimization of bronchodilator medications is first-line therapy and

involves identifying the best medications or combinations of medications
taken on a regular schedule for a specific patient.

Hospitalization. Indications for hospitalization for acute exacerbation of

COPD include severe dyspnea that does not respond to initial
therapy, confusion or lethargy, respiratory muscle fatigue, paradoxical
chest wall movement, and peripheral edema.
 Oxygen therapy. Upon arrival of the patient in the emergency room,
supplemental oxygen therapy is administered and rapid assessment is
performed to determine if the exacerbation is life-threatening.

Antibiotics. Antibiotics have been shown to be of some benefit to

patients with increased dyspnea, increased sputum production, and
increased sputum purulence.

Surgical Management

Patients with COPD also have options for surgery to improve their condition.

Bullectomy. Bullectomy is a surgical option for select patients

with bullous emphysema and can help reduce dyspnea and improve
lung function.

Lung Volume Reduction Surgery. Lung volume reduction surgery is

a palliative surgery in patients with homogenous disease or disease that
is focused in one area and not widespread throughout the lungs.

Lung Transplantation. Lung transplantation is a viable option for

definitive surgical treatment of end-stage emphysema.

Nursing Management

Management of patients with COPD should be incorporated with teaching and

improving the respiratory status of the patient.

Nursing Assessment

Assessment of the respiratory system should be done rapidly yet accurately.

 Assess patients exposure to risk factors.

Assess the patients past and present medical history.

Assess the signs and symptoms of COPD and their severity.

Assess the patients knowledge of the disease.

Assess the patients vital signs.

Assess breath sounds and pattern.

Diagnosis

Diagnosis of COPD would mainly depend on the assessment data gathered by

the healthcare team members.

Impaired gas exchange due to chronic inhalation of toxins.

Ineffective airway clearance related to bronchoconstriction, increased

mucus production, ineffective cough, and other complications.

Ineffective breathing pattern related to shortness of breath, mucus,

bronchoconstriction, and airway irritants.

Self-care deficit related to fatigue.

Activity intolerance related to hypoxemia and ineffective breathing

patterns.

Planning & Goals

Main article: 5 Chronic Obstructive Pulmonary Disease (COPD) Nursing

Care Plans

Goals to achieve in patients with COPD include:

 Improvement in gas exchange.

Achievement of airway clearance.

Improvement in breathing pattern.

Independence in self-care activities.

Improvement in activity intolerance.

Ventilation/oxygenation adequate to meet self-care needs.

Nutritional intake meeting caloric needs.

Infection treated/prevented.

Disease process/prognosis and therapeutic regimen understood.

Plan in place to meet needs after discharge.

Nursing Priorities

1. Maintain airway patency.

2. Assist with measures to facilitate gas exchange.

3. Enhance nutritional intake.

4. Prevent complications, slow progression of condition.

5. Provide information about disease process/prognosis and treatment

regimen.

Nursing Interventions

Patient and family teaching is an important nursing intervention to enhance

self-management in patients with any chronic pulmonary disorder.
To achieve airway clearance:

The nurse must appropriately administer bronchodilators and

corticosteroids and become alert for potential side effects.

Direct or controlled coughing. The nurse instructs the patient in direct

or controlled coughing, which is more effective and
reduces fatigue associated with undirected forceful coughing.

To improve breathing pattern:

Inspiratory muscle training. This may help improve the breathing

pattern.

Diaphragmatic breathing. Diaphragmatic breathing reduces respiratory

rate, increases alveolar ventilation, and sometimes helps expel as much
air as possible during expiration.

Pursed lip breathing. Pursed lip breathing helps slow expiration,

prevents collapse of small airways, and control the rate and depth of
respiration.

To improve activity intolerance:

Manage daily activities. Daily activities must be paced throughout the

day and support devices can be also used to decrease energy expenditure.

Exercise training. Exercise training can help strengthen muscles of the

upper and lower extremities and improve exercise tolerance and
endurance.

Walking aids. Use of walking aids may be recommended to improve

activity levels and ambulation.

To monitor and manage potential complications:

 Monitor cognitive changes. The nurse should monitor for cognitive
changes such as personality and behavior changes and memory
impairment.

Monitor pulse oximetry values. Pulse oximetry values are used to

assess the patients need for oxygen and administer supplemental oxygen
as prescribed.

Prevent infection. The nurse should encourage the patient to be

immunized against influenza and S. pneumonia because the patient
is prone to respiratory infection.

Evaluation

During evaluation, the effectiveness of the care plan would be measured if goals
were achieved in the end and the patient:

Identifies the hazards of cigarette smoking.

Identifies resources for smoking cessation.

Enrolls in smoking cessation program.

Minimizes or eliminates exposures.

Verbalizes the need for fluids.

Is free of infection.

Practices breathing techniques.

Performs activities with less shortness of breath.

Discharge and Home Care Guidelines

It is important for the nurse to assess the knowledge of patient and family
members about self-care and the therapeutic regimen.

Setting goals. If the COPD is mild, the objectives of the treatment are to
increase exercise tolerance and prevent further loss of pulmonary
function, while if COPD is severe, these objectives are to preserve current
pulmonary function and relieve symptoms as much as possible.

Temperature control. The nurse should instruct the patient to avoid

extremes of heat and cold because heat increases the temperature and
thereby raising oxygen requirements and high altitudes increase
hypoxemia.

Activity moderation. The patient should adapt a lifestyle of moderate

activity and should avoid emotional disturbances and stressful situations
that might trigger a coughing episode.

Breathing retraining. The home care nurse must provide the education
and breathing retraining necessary to optimize the patients functional
status.

Documentation Guidelines

Documentation is an essential part of the patients chart because the

interventions and medications given and done are reflected on this part.

Document assessment findings including respiratory rate, character of

breath sounds; frequency, amount and appearance of secretions
laboratory findings and mentation level.

Document conditions that interfere with oxygen supply.

Document plan of care and specific interventions.

Document liters of supplemental oxygen.

 Document clients responses to treatment, teaching, and actions
performed.

Document teaching plan.

Document modifications to plan of care.

Document attainment or progress towardsgoals.

Practice Quiz: Chronic Obstructive Pulmonary Disease

(COPD)

EXAM MODE
PRACTICE MODE
TEXT MODE
In Exam Mode: All questions are shown but the results, answers, and
rationales (if any) will only be given after youve finished the quiz.

Practice Quiz: Chronic Obstructive Pulmonary

Disease (COPD)

Start
See Also

Posts related to Chronic Obstructive Pulmonary Disease (COPD):

10 COPD: Bronchitis Nursing Care Plans

5 Chronic Obstructive Pulmonary Disease (COPD) Nursing Care Plans

 8 Pneumonia Nursing Care Plans

Further Reading

Select books and recommended resources:

1. Medical-Surgical Nursing: Assessment and Management of Clinical

Problems

2. Medical-Surgical Nursing: Patient-Centered Collaborative Care

3. Saunders Comprehensive Review for the NCLEX-RN Examination

4. Brunner & Suddarths Textbook of Medical-Surgical Nursing

5. COPD National Library of Medicine

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Marianne Belleza, RN
I am always seen in bookstores trying to find peculiar books that tickle my senses and tear-jerker
biographies. I prefer Maternal and Child Health Nursing and Medical-Surgical Nursing more than any
other fields. I am always lusting after banana-infused food and beverages. I am currently in my
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