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P-CMV mode:
pressure trigger, Psupport =
20 cmH2O, PEEP =
5 cmH2O
• The yellow triangles
indicate the pressure
triggered breaths.
• The blue arrows indicate
the abnormal baseline
drop.
• The auto-triggered
breaths have a much
shorter Te.
• The inspiratory time of
normal and abnormal
breaths is the same,
because both are time
cycled.
Mode P-CMV:
pemicu tekanan, Psupport =
20 cmH2O, PEEP =
5 cmH2O
• Segitiga kuning
tunjukkan tekanannya
memicu nafas.
• Tanda panah biru menunjukkan
garis dasar yang tidak normal
penurunan.
• Pemicu otomatis
nafas memiliki banyak
pendek Te.
• Waktu inspirasinya
normal dan abnormal
nafas adalah sama,
karena keduanya adalah waktu
bersepeda
• SPONT/PSV mode:
flow trigger, Psupport =
20 cmH2O, PEEP =
5 cmH2O, flow cycling =
25%
• The pink triangles indicate
the flow-triggered
breaths.
• The blue arrows indicate
abnormal inspiratory flow
(hardly visible) at the end
of expiration.
• The auto-triggered
breaths have a much
shorter Te.
• A sizeable gas leak
causes the peak pressure
to be lower than expected.
Fig. 13.10 N ormal and abnormal volume waveform in pressure breaths. Abnormalities
include: (a) the volume waveform fails to return to zero, (b) the volume peak increases (this is not so
in volume breaths), and (c) the monitored VTE is considerably smaller than the VTI.
VTE: expiratory tidal volume; VTI: inspiratory tidal volume.
Fig. 13.11 Waveforms of pressure breaths with gas leakage. Both graphs have three
waveforms: from top down, pressure (white), flow (pink), and volume (green). The breaths on
the left of each graph are normal (leak free), and the breaths on the right are abnormal due to
a moderate or sizeable gas leak. The yellow arrows indicate the reset of volume monitoring.
Abnormalities include: (a) auto- triggering, (b) a difference in size of flow area, (c) a higher- thannormal
peak of the volume waveform, and (d) a sharp drop in volume at end expiration.
Fig. 13.12 Waveforms of volume breaths with gas leakage. Abnormalities include: (a) autotriggering,
(b) a difference in size of flow areas, (c) a lower- than- normal peak pressure, and (d) a
sharp drop in volume at end expiration.
Thus, during the expiratory phase, the height of the volume waveform has two parts. The first
part is monitored expiratory tidal volume (VTE). The second part is the height of the sharp
drop, which is a good indicator of the leak volume.
Pressure breaths have an inherent ability to compensate leaks: the ventilator increases the
inspiratory flow, causing a greater inspiratory tidal volume (VTI). This explains why gas leakage
causes the peak of the volume waveform to be higher than normal, a phenomenon that does not
occur in volume breaths.
D. Reduction in monitored expiratory tidal volume:
Ventilators calculate monitored volume from flow measurements taken over time. As we saw
above, a gas leak causes the expiratory flow area to decrease, resulting in a significantly reduced
expiratory tidal volume (VTE).
This reduced VTE does not necessarily mean that ventilator monitoring is malfunctioning. It
means that most of the expired tidal volume exited the system through the abnormal opening
(i.e. without being ‘seen’ by the flow sensor).
The VTE represents the minimum tidal volume that the patient actually received. In other
words, the patient may have received more, but not less, than the displayed VTE. This is why a
patient can still survive with an unbelievably small VTE.
When the displayed VTE is abnormally low, you need to consider the possibility of a gas leak.
E. Flow cycling failure:
In pressure support and adaptive support modes, a sizeable gas leak can cause a failure of the
flow cycling mechanism.
As we saw in Chapter 7, flow cycling is based on inspiratory flow measurements in its descending
part. The ventilator switches from inspiration to expiration when the inspiratory flow drops
to a preset threshold. An excessive leak may cause the inspiratory flow to stay higher than the
cycling threshold so that inspiration does not end. Such an endless inspiration is clinically
unacceptable.
To prevent this possible abnormality, some ventilators implement an additional time cycling
mechanism as a backup. For instance, Hamilton Medical ventilators have a Timax control.
When the operator- set maximum inspiratory time ends, the ventilator terminates inspiration
and starts expiration regardless of the inspiratory flow rate (Fig. 13.13).
Fig. 13.13 N ormal and abnormal pressure support breaths. In the two breaths on the right, flow
cycling fails due to a sizeable gas leak. The ventilator does not switch from inspiration to expiration
as expected, causing the inspiration to be much longer than expected. Finally, the inspiration is
terminated by the backup time cycling mechanism.
Jadi, selama fase ekspirasi, tinggi gelombang volume memiliki dua bagian.
Pertama
Bagian tersebut dipantau volume arus ekspirasi (VTE). Bagian kedua adalah
tinggi yang tajam
drop, yang merupakan indikator bagus dari volume kebocoran.
Tekanan napas memiliki kemampuan inheren untuk mengkompensasi kebocoran:
ventilator meningkatkan
aliran inspirasi, menyebabkan volume arus inspirasi lebih besar (VTI). Ini
menjelaskan mengapa kebocoran gas
menyebabkan puncak gelombang volume menjadi lebih tinggi dari biasanya,
sebuah fenomena yang tidak
terjadi dalam volume napas.
D. Pengurangan volume tidal ekspirasi terpantau:
Ventilator menghitung volume yang dipantau dari pengukuran aliran yang
diambil dari waktu ke waktu. Seperti yang kita lihat
Di atas, kebocoran gas menyebabkan daerah aliran ekspirasi berkurang,
sehingga terjadi penurunan yang signifikan
volume arus ekspirasi (VTE).
VTE yang berkurang ini tidak berarti bahwa pemantauan ventilator tidak
berfungsi. Saya t
berarti sebagian besar volume tidal kadaluarsa keluar dari sistem melalui
lubang yang tidak normal
(yaitu tanpa 'dilihat' oleh sensor aliran).
VTE mewakili volume tidal minimum yang benar-benar diterima pasien. Lainnya
Kata-kata, pasien mungkin sudah menerima lebih banyak, tapi tidak kalah,
dari pada VTE yang ditampilkan. Inilah sebabnya mengapa a
Pasien masih bisa bertahan dengan VTE yang luar biasa kecil.
Bila VTE yang ditampilkan tidak normal, Anda perlu mempertimbangkan
kemungkinan kebocoran gas.
E. Kegagalan bersepeda:
Dalam dukungan tekanan dan mode pendukung adaptif, kebocoran gas yang cukup
besar dapat menyebabkan kegagalan
mekanisme arus bersepeda.
Seperti yang kita lihat di Bab 7, siklus arus didasarkan pada pengukuran
arus inspirasi dalam penurunannya
bagian. Ventilator beralih dari inspirasi ke kadaluarsa saat aliran
inspirasi turun
ke ambang batas yang telah ditentukan. Kebocoran yang berlebihan dapat
menyebabkan aliran inspirasi tetap tinggi dari pada
ambang bersepeda agar inspirasi tidak berakhir. Inspirasi tanpa henti
seperti itu secara klinis
tidak dapat diterima
Untuk mencegah kemungkinan kelainan ini, beberapa ventilator menerapkan
waktu bersepeda tambahan
mekanisme sebagai cadangan Misalnya, ventilator Hamilton Medical memiliki
kontrol Timax.
Bila waktu jeda maksimum yang ditentukan oleh operator berakhir, ventilator
mengakhiri inspirasi
dan mulai kadaluarsa terlepas dari tingkat aliran inspirasi (Gambar 13.13).
Gambar 13.13 Tekanan napas tidak normal dan tidak normal. Dalam dua napas
di sebelah kanan, mengalir
bersepeda gagal karena kebocoran gas yang cukup besar. Ventilator tidak
beralih dari inspirasi ke kadaluarsa
seperti yang diharapkan, menyebabkan inspirasi menjadi lebih lama dari yang
diperkirakan. Akhirnya, inspirasinya adalah
Diakhiri dengan mekanisme bersepeda waktu cadangan.
Therefore, for a given pressure gradient, increased resistance causes decreased gas flow. In a
pressure mode, a significant occlusion results in a sharp decrease in the generated tidal volume.
In a volume breath where the tidal volume is predefined and controlled, a significant occlusion
causes a sharp increase in peak pressure.
Consequences of an occlusion
In theory, the ventilated patient cannot:
◆ Inhale if the inspiratory limb is completely occluded;
◆ Exhale if the expiratory limb is completely occluded;
◆ Inhale and exhale if the airway is completely occluded.
Obviously, none of these situations is clinically tolerable. The clinical consequences of gas passageway
occlusion are determined by the size of occlusion and the clinician’s response.
Hypoventilation is the inevitable consequence of all types of occlusion. An active patient
will fight frantically for life. Multiple alarms will be annunciated. If the occlusion is close to
complete, clinical signs of sudden and serious respiratory distress syndrome appear, requiring
immediate correction.
How to recognize an occlusion
An occlusion can happen to any ventilated patient at any time. Compared to a gas leak, a partial
occlusion is typically more difficult to recognize, even for experienced clinicians. In an emergency,
therefore, the general rules of emergency management apply; refer to section 13.3.
Most manifestations of occlusion are non-a•‰specific. Ventilator alarms are indeed a help,
but only when they are set sensitively. If hypoventilation and patient-a•‰ventilator asynchrony
do not respond to corrective measures as expected, the clinician in charge should consider
the possibility of occlusion and conduct the investigation as explained in Tables 13.4, 13.5
and 13.6.
As you apply the information above, note the following:
◆ It assumes a passive patient and use of a proximal flow sensor at the Y-a•‰piece;
◆ Typical changes in waveforms and monitoring parameters appear only when the occlusion is
severe but incomplete;
◆ The alarms mentioned may be activated only when alarm limits are set properly;
◆ Trending these parameters, if available, may help to identify small and progressive changes.
You can relieve all three types of occlusion, with the exception of ETT kinking, by disconnecting
the patient from the ventilator. If you suspect ETT kinking, pass a suction catheter through
the ETT for verification. If the tube is occluded, the catheter cannot pass through.