Ben Lynch Blood Sugar Genes

Blood Sugar Balancing Techniques
Dr. Jody Stanislaw has lived with Type 1 diabetes since age 7. Her experiences have taught her how to
manage it effectively. She is healthier than most non-diabetic individuals. What she shares can
tremendously benefit anyone who has blood sugar problems.
Dr. Jody Stanislaw

drjodynd.com
www.DirtyGenesSummit.com
© 2017 DR. BEN LYNCH. All Rights Reserved.
The information, comments, and opinions expressed in this presentation are those of the
speaker(s) and are based on their own clinical experience and interpretation of the
literature. Published content is intended for educational purposes only. Seeking Health
LLC, Dr Ben Lynch LLC, Dr Ben Lynch, and other featured speakers will not be held liable
for any direct, indirect, consequential, exemplary, or other damages and/or injuries
arising from the use or misuse of any materials or information published.
Always seek medical advice from your qualified health professional. This information is
not intended as a substitute for seeking care from a qualified health professional.
*These statements within have not been evaluated by the US Food and Drug
Administration. These products are not intended to diagnose, treat, cure, or
prevent any disease.
3
Type 1 Diabetes
• Rare for type 1 diabetes to be genetic
• Likely to be environmental factors.
• Things that weaken the immune system:

• Mercury
• Lack of breastfeeding
• Lack of Vitamin D
• Excessive sugar
• Stress - both physical and mental
• Introduction to cow’s milk too early
4
Constant Balancing Act
• Stress causes the body to make adrenalin
• Tells the liver to secrete glucose into the bloodstream
• Blood sugar peaks in the morning 
• Target to stay between 80 - 120 blood sugar level
5
Type 1 vs Type 2 Diabetes

• Type 1
• An autoimmune condition
• Beta cells in pancreas have been killed off
• Body doesn’t make insulin 
• Type 2
• Your body is ignoring insulin

• Insulin production is becoming weaker
• System is breaking down
• Can reverse: Stop overtaxing system, eat healthier, can revitalize the system 
• Carbs - There is no essential carbohydrate

• None of us need any source of carbohydrates other than nature-made sugars: Fruits,
vegetables, legumes, whole grains
• Eating less carbs makes getting healthy blood sugar levels easier
Lab Tests & Resources
1. Fasting blood glucose should be higher than 75, below 95 first thing in the morning 
2. Hemoglobin A1C should be below 5.5

a.) A1C hemoglobin is a type of protein in red blood cell
b.) Gold standard for monitoring blood glucose 
3. Oral Glucose Tolerance test 
4. Fasting C-Peptide test (fasting should be greater than 1) 
Recommended Reading:
The Case Against Sugar by Gary Taubes
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Dr. Stanislaw
Dirty Genes Summit:

Blood Sugar Balancing Techniques
Dr. Jody Stanislaw
The information, comments, and opinions expressed in this presentation

are those of the speaker(s) and are based on their own clinical experience
and interpretation of the literature. Published content is intended for
educational purposes only. Seeking Health LLC, Dr Ben Lynch LLC, Dr
Ben Lynch, and other featured speakers will not be held liable for any
direct, indirect, consequential, exemplary, or other damages and/or injuries
arising from the use or misuse of any materials or information published.
Always seek medical advice from your qualified health professional. This
information is not intended as a substitute for seeking care from a qualified
health professional.
*These statements within have not been evaluated by the US Food

and Drug Administration. These products are not intended to
diagnose, treat, cure, or prevent any disease.
© 2018 DR. BEN LYNCH. All Rights Reserved. 1
Dr. Stanislaw
Ben: Type 1 diabetes -- this is something that not many people have on their radar but
if you are alive on this planet, which you obviously are, this is something that you
need to be aware of because it’s increasing at an alarming rate, which Dr.
Stanislaw will be sharing the statistics with you.
So, Jody Stanislaw is a practicing naturopathic physician in gorgeous Sun Valley,

Idaho and she specializes in type 1 diabetes. Not only does she specialize in it,
she personally has it herself and she’s had it longer than I’ve known her, and Jody
and I have known each other, man, since 2002 I believe.
Jody: Fifteen years, Ben.
Ben: Yeah, we’re getting old. We’re getting old.
Jody: No, I'm not.
Ben: Yeah. Older, wiser.
Jody: That's right.
Ben: But we both graduated together in 2007 from our amazing school of Bastyr
University and with naturopathic medicine, we are keeping ourselves vibrant and
healthy. We want to pass this information on to you and your loved ones and your
family and even your enemies to make them sweeter so they’re no longer enemies.
So, Jody.
Jody: Yes?
Ben: Hello.
Jody: Hello, Ben.
Ben: And one thing you should know about Jody, folks, is she’s awesome. She’s just
awesome. Always happy. She doesn’t let type 1 get her down. And Jody, how old
were you when you got type 1?
Jody: Seven.
Ben: Seven. What happened?
Jody: Classic symptoms. Painfully thirsty, like I cried. My mouth was so dry. Lost a bunch
of weight. Wet the bed every night. I’d cry because I want more water and I had no
energy. Super hungry but losing weight. It just hit me pretty intensely and after
about a week of not getting better, I went to the doctor, I peed in a cup and my
urine was full of sugar and I got sent to a Children’s Hospital in Seattle for a week
of training, to learn how to live with my new life.
Dr. Stanislaw
Ben: Wow. And how did that affect you?
Jody: Well, as you said, I was just like, “Ooh! What are we doing now? Where are we
going? This is cool.” Like Children’s Hospital was so fun, that's why I became a
doctor. That week I had so much fun with all the doctors and there was like arts
and crafts room and there were elevators I could play in and there was a pool.
Ben: Awesome. That's cool.
Jody: I didn’t quite figure out. At 7, you’re like, “Oh! This is a fun adventure.” The only
thing that I understood is that I had to eat less sugar and I had to take one insulin
injection a day. So I was like, “Oh, I can deal with one insulin injection a day.” To
me, you can’t really grasp the intensity of it as a 7-year-old.
Ben: Good point. Looking back, what do you think caused you to get type 1?
Jody: Well, that's a great question. There’s been many studies and some say yes and
some say no about introducing cow’s milk at too young of an age. I was only
breastfed six weeks so that's going to weaken the immune system. I was probably
given cow’s milk at an early age, that's going to weaken the immune system. I lived
in Seattle so my vitamin D was low, that could weaken the immune system. I had
tubes put in my ears when I was 5 or 6 which to me is likely food allergy stuff going
on that wasn’t addressed properly. My mom had a full mouth of mercury amalgams
in her mouth when she carried me. So any or all of those things are going to
weaken the immune system. Mercury, lack of breastfeeding, low vitamin D,
introducing cow’s milk too early. So I'm kind of a poster child. But nobody in my
family has it and generally it’s very rare for type 1 to be in the family, so it’s definitely
more likely an environmental combination of factors.
Ben: Were you put on any antibiotics before you were 7 that you remember?
Jody: I don’t know. I really don’t know.
Ben: Okay. Because getting tubes in your ear, I mean, you and I are both screaming in
our heads “milk”, right?
Jody: Exactly.
Ben: So explain to folks the connection between why milk because we just think of milk
as milk. Why would milk cause you to have problems in your ears to get tubes and
then a couple years later to get type 1. To me, that's pretty damning evidence.
Jody: Yes. So it’s foreign animal proteins and these are the things that we ingest and
then the body has to assess “is this safe or not?” Milk has been known to be an
inflammatory food so there is inflammation going on. There’s also the theory of a
cross reaction that if a weak immune system in a child that wasn’t breastfed is now
Dr. Stanislaw
introduced to a foreign protein, then the immune system could potentially start
seeing the milk foreign, the foreign animal protein as something that needs to be
attacked and then there’s a theory that there’s a cross reaction between the
immune system attacking the milk and there’s an allele on the milk protein that’s
similar to that of a beta cell. But the studies are unfortunately not really clear. Some
of the milk studies say yes, some of the milk studies say no. But another thing is
kids that reduce their gluten intake also reduce their type 1 diabetes risk. So diet
is a huge part.
Ben: You’re not going to have any issue really with the milk proteins unless you have
leaky gut, right?
Jody: Exactly, yeah.
Ben: And you not being breastfed or having adequate vitamin D, that's going to kind of
set you up.
Jody: Exactly, yeah. Leaky gut for sure.
Ben: So 7, you got this one shot a day, you’re going through life. You had a great
experience in the hospital which is fantastic, so kudos to Children’s for that. That's
incredible. Now you specialize in type 1. Was there a period in your life where a
shot a day and watching your sugar intake was just not working? Did you get
sicker?
Jody: I have pictures of my logs that you can add to your slides of what my numbers
were because I wrote down all my numbers for years. I would go down to 40 and
go up to 300 on a regular basis. And of course, a healthy number is between 80
and 120. But we were just doing what we were told. We were told you give this
one shot a day and this is exactly what you have to eat. Now the 1 shot moved to
2 a day very quickly and then moved to 4 a day within three years. Now, I do
multiple shots all day long. I mean, I don’t even know how many shots a day I do
to more mimic a natural pancreas because every time a non-diabetic eats, the
pancreas gives out a little bit of insulin. So that is happening at multiple times all
day long. So the 1 shot a day was literally a shot in the dark to try to balance the
food being eaten with the blood sugar level going up and then insulin, of course,
insulin’s job is to take the sugar out and feed it to the muscles, the liver and the fat.
I'm walking on a tight rope every day in terms of is my sugar level going up or down
and is there something I need to do to balance it. So back then, when you only
have this one sledgehammer of a shot and then you’re eating multiple times all
day long, I wasn’t ever between 80 and 120 for very long. So now, I'm so sensitive.
Any time I go even above 120 or 130 now, I don’t feel like myself. I don’t feel well,
I can’t concentrate as well. I get kind of depressed and tired. I don’t know if I was
just so young and vibrant and resilient that I didn’t realize how crappy I felt or if
Dr. Stanislaw
literally my body just was more resilient. But if I go to 40 and 300 every day now, I
would just not be able to function.
Ben: When you’re saying these numbers, you mean your glucose level, I assume, right?
Jody: Yes.
Ben: So before, when you did the insulin shot once a day, your insulin was swinging all
over the place all the way up to you said 200, 400?
Jody: My blood sugar level was swinging all over the place.
Ben: Sorry, your blood sugar. Right. So your blood sugar is swinging all over the place.
Now you inject multiple times a day and now you’re keeping it between 80 and?
Jody: 80 to 120 is the goal. That's what non-diabetics stay between. But to be honest,
keeping the blood sugar level normal at all times is a full-time job.
Ben: I can imagine.
Jody: I work, I'm very diligent and I happen to be 150 right now. There’s so many factors
that raise the blood sugar level. I was a little stressed this morning and then we
have technical difficulties. Then, the slightest bit of stress causes your body to
make adrenaline and any time you have adrenaline or cortisol, a stress hormone,
it basically tells the liver to secrete glucose into your bloodstream because the
body thinks that it’s doing you a favor and any time you’re under stress, you need
extra fuel.
The natural rhythm of cortisol is to peak in the morning. This is for anybody. So in
a diabetic, that means my blood sugar level peaks in the morning without me even
eating. So what I put in my mouth is certainly not the only thing that affects my
blood sugar level. My hormones do, my stress level, adrenaline. So it’s a constant
balancing act every day to figure out how much insulin I need, what my blood sugar
level is doing, can I stay between 80 and 120. I'm on top of it as I can be and like
I said, I'm 150 right now. I gave myself a shot and it should come down soon.
Ben: Excellent. So just in case people don’t understand the difference between type 1
and type 2 diabetes, can you define that?
Jody: Absolutely, yes. So very, very different of course. Type 1 diabetes is an

autoimmune condition. The immune system attacked and killed my insulin-
producing cells. The beta cells are in the pancreas and my beta cells had been
killed off. So I don’t make insulin because you need beta cells to make insulin and
you need insulin to be alive because you need insulin to manage your blood sugar
levels and keep the amount of glucose in your blood normal. So type 1, we don’t
make insulin.
Dr. Stanislaw
Type 2, the body is ignoring the insulin they make and their insulin production is
becoming weaker. There’s no autoimmune condition going on in a classic type 2.
It’s basically the whole system is breaking down. You eat, your sugar level goes
up and the body is like, “I can’t deal with the sugar anymore and I'm going to ignore
it.” The cells don’t want to respond to the sugar, the cells aren’t going to open their
gates up and let the sugar in. The insulin is getting tired because the beta cells
have to keep working to make insulin and make insulin to take all the sugar out of
the blood. And on both ends of that game, the insulin production and the cells
opening up to get the sugar in, they’re both tired out and they’re just tired.
So, those people, if they stop overtaxing their system and got a lot healthier and
lost weight and started exercising and eating more vegetables, they can literally
revitalize their system. The beta cells will start making up insulin again. The cells
will start opening up and taking in the sugar. Unless you’re a late, late, late stage
type 2 who’s literally killed off their beta cells from overwork, type 2 can be
reversed. I don’t even say that type 2 is a “disease”. I mean, sure, there’s certain
people that have a predisposition that are unlucky and I do know some healthy
people that have higher blood sugar levels. But that could even be type 1 ½ which
is a whole another discussion.
Ben: Right. With type 1 diabetes, you’re getting the blood sugar because, I mean,
because you just tested it, right? You got 150. But having your blood sugar at 150
isn’t useful for you because it’s not in your cell. Right?
Jody: Right. Well, high blood sugar level is damaging to the body. Having too much
glucose in your bloodstream is what causes heart disease, kidney failure, strokes,
gangrene, blindness, Alzheimer’s. The blood obviously is a miracle fluid and it
needs to have the proper balance of things. And too much glucose in the blood
literally can rip apart the inside of arteries causing cardiovascular damage, strokes,
heart disease. It also damages nerves because nerves don’t have a filter, if you
will, for how much glucose should come in the cell. If there’s too much glucose in
the blood, there’s too much glucose in the nerve cell, too much glucose in the brain.
It just floods in and it builds up and it causes cellular damage. So the danger in
diabetes and why diabetes is such an expensive complication-rich disease is
because too much glucose in the blood causes damage throughout the entire
body.
Ben: That's definitely an issue. So you take the insulin in order to get the glucose out of
your blood and into your cells, right?
Jody: Exactly.
Ben: Right now, you have glucose in your blood. Yet glucose cannot even get into your
cell without insulin.
Dr. Stanislaw
Jody: Yes. Through exercise. So there’s these things called GLUT4 receptors on your
muscle and fat cells, glucose transporter 4 cells. If I wasn’t sitting here talking to
you, I would literally just go outside and go for a 5-minute run and I’d come down
way faster. But yeah, you become more insulin sensitive if you are a fit and active
person because the concentration of GLUT4 receptors on the cells increases and
in the middle of exercise, GLUT4 can bring glucose in without even the presence
of insulin. So as a health and fit person, I have more GLUT4’s on my cells 24 hours
a day but after an intense workout, I have an even higher GLUT4 concentration
that can last up to 24 to 48 hours.
So when I talk to patients, I say ideally, if you can exercise at least every other day,
you will have a constant increase in GLUT4 concentration that lasts every day. But
if they go two days without working out, their sugar levels, well, they’ll need more
insulin that third day. Because our sensitivity, just because insulin resistance is
thought of as a type 2 condition, which it is, basically their cells are so tired of
answering to insulin, presence of brain sugar into the cell that the cells are like,
“Look, I'm so tired of bringing sugar in. I'm not going to anymore. I'm going to
ignore.” Because basically the GLUT4 receptors get tired out. The GLUT4
receptors are like, “I can’t come up to the surface anymore. I'm too tired.” That's
classic insulin resistance in type 2.
But we all have various levels of insulin resistance or insulin sensitivity regardless
if you’re diabetic or not. I mean, if you sit on your butt all day versus if you are
walking around all day, obviously you’re more sensitive to insulin when you’re
walking around. If you gain weight, you’re more insulin resistant. If you ate a bunch
of fat, like if I eat a huge thing of avocadoes and nuts and olive oil, all of that fat
digests, breaks down into fatty acids. The fatty acids are now circulating in my
bloodstream for at least the next 5, 6, 7 hours and I will be more insulin resistant
in that time period afterwards. So this is what I mean that for a type 1 to manage
the blood sugar levels, it’s way more complicated than just how many grams of
carb do you eat at your meal today.
Ben: That's really cool because from what I'm hearing you, and correct me if I'm wrong,
is anyone regardless if they have type 1 or 2 diabetes, just anyone in general,
exercise will increase the genetic expression of producing this receptor on the cell
to take more glucose into the cell, and that's the GLUT4 receptor.
Jody: Yes.
Ben: So anyone living, anyone listening now whether they have type 1 or 2 diabetes or
not, if they exercise, they will be able to they will be able to remove that harmful
sugar in their blood to get it into the cell where it does its job.
Jody: Yeah. Your body becomes more efficient.
Dr. Stanislaw
Ben: That's cool.
Jody: It becomes more efficient. Insulin is inflammatory in excess. You need a certain
level of insulin. But in excess, insulin is inflammatory. So yes, the body is more
efficient and you don’t need as much insulin when you are exercising.
Ben: Right. That's brilliant. You talk about how smart and brilliant the body is to be able
to produce more receptors on a cell, to be able to adapt to a high exercise
environment by pulling in more sugar. And if you sit on your butt and you’re lazy
and you’re watching TV, those receptors go away because now the cell doesn’t
need as much energy anymore. It’s going to say, “Hey, I don’t need that glucose”
in your cell right now because you’re sitting on your butt. So the density of the
GLUT4 receptor is going to go down.
Jody: Exactly.
Ben: That's so cool.
Jody: Isn’t that cool? Yes.
Ben: So cool.
Jody: And I always like to say glucose is in the blood and not sugar because then people
get confused about is sugar good, is sugar bad. Wait, we need a bunch of sugar?
I mean people are confused that an apple raises my blood sugar level and so does
a Twinkie. So we have to look at the bigger picture of the food that we’re eating
and its associated nutrients. We can’t just vilify all glucose or all sugar. Sugar is
loaded word.
Ben: Got it. That's kind of with folate and folic acid, right?
Jody: Yeah.
Ben: So I go off on that. So talk to us a little bit more about the differentiation there.
Explain more why sugar and glucose you can’t really put in the same category.
Jody: Exactly. So the only forms of carbohydrates that are made by the earth are fruits,
vegetables, legumes and whole grains. That's where carbohydrates come from
from the earth. Fruits, vegetables, whole grains, legumes and beans. So also,
there is no such thing as an essential carbohydrate. There’s essential fatty acids
and there’s essential amino acids, but there is not essential carbohydrates. I ate a
piece of halibut the other day and I didn’t take any insulin for it just to prove my
point that protein can be turned into glucose. The piece of halibut raised my blood
sugar level by 75 points. So the body literally for people that don’t eat carbs, it’s
fine. The body takes protein and can convert it into glucose. So my eating a piece
Dr. Stanislaw
of halibut which officially has no carbohydrates in it, literally raised my blood sugar
level by 75 points.
Personally, my philosophy when I work with patients, I have this virtual practice, I
work with type 1 patients anywhere around the world, and a lot of parents are being
told their children need to eat 50-75 grams of carbohydrate per meal. That's kind
of the standard American Diabetes Association recommendation which really has
no data to support it. Their biggest concern is that your child needs these calories
for growth. And I’ll think, well, actually high blood sugar levels can stunt growth,
they stunt development. So I tell my parents to say eating less carbs is obviously
going to make getting healthy blood sugar levels easier and then you can monitor
growth through the growth chart, energy levels, and function in school. But
anyways, it really bothers me because when they give these recommendations,
they just say eat 100 carbs. So the kids can have a soda, they can eat chips and
have a Twinkie and they got their 100 carbs. I'm thinking, hello? Does anybody
see the problem with that?
So I really like to remind people like if you want to eat carbs, they should be fruit,
vegetables, beans, or whole grains. Those are the only carbs you need. And
frankly, you don’t even need them because your body can make carb from other
foods. It’s obviously much more complex. You want to make sure you get your B
vitamins and all that stuff if you’re going to reduce your carbohydrate. But the reality
is, none of us need any source of carbohydrate outside of those four. Everything
else is a processed product made from a greed focused company.
Ben: Right. So did I hear you right? Their recommendation is 50 to 75 grams per meal?
Jody: Yes.
Ben: Wow!
Jody: Yes. And so the kids are eating honey and that Cheerios, they’re having bread,
they’re having chips because they have to fill their carb. So I actually expressed
my disapproval for an event for children with diabetes that was serving a pancake
breakfast. I had parents unfriend me, tell me how uneducated I was because their
doctor said the children can just eat anything they want and give themselves
insulin, and “How dare you be so judgmental? You need to go back to medical
school and be properly trained.” I understand why the mama bear comes out
because they’re already sad that their child has this thing and now they just want
their child to be like all the other kids. But then I have to remind them like if you
want your kid to be like all the other kids, all the other kids are getting type 2
diabetes and becoming obese. So we’ve got to make type 1 diabetes a whole
family event of like, “Hey, you guys, we really should eat healthier.”
Dr. Stanislaw
Ben: So that immediately put some huge warning bells in my head. So type 1 diabetics
are recommended to eat 50 to 75 grams per meal as a kid.
Jody: Yeah.
Ben: That's a huge whopping load. People and their keto type diets, I'm not going to
classify them as extreme because it’s this huge movement now and it’s doing a lot
of good for people and we’ll talk about that in a second. But I mean, if they eat 50
grams of carbs a day, that's a lot.
Jody: A day, yeah. I would say 75, I only heard from this one physician and I couldn’t
believe it, but I think the standard recommendation is more like 45 to 50 per meal.
That's what a lot of pediatric endocrinology nutrition office or certified diabetes
educators are giving.
Ben: And of course they’re not classifying what a good carbohydrate is.
Jody: No, no, no. Sure, like you just give a shot. I mean, that's what we’re taught. The
more carbs you eat, the more insulin you need. And that's true. If I have quinoa, if
I have a fourth of a cup, I give myself a unit. If I have a half a cup, I give myself 2
units. If I eat a three-fourths of a cup, I give myself 3 units. So yes, it’s true. The
more carbs I eat, the more insulin I need.
But here’s the point that they’re missing. Insulin is not a precise tool. It’s not a
precise tool at all. So how do I know if my food is going to digest first and throw my
blood sugar level way up to the sky and then the insulin is going to hit eventually?
Insulin is low. It’s called rapid insulin; it’s not rapid. It takes like an hour to work. So
I would never eat pineapple for example. If I ate pineapple for breakfast, I can go
from 80 to 280 within, I don’t know, 30 minutes. But if I do my shot the second I eat
the pineapple, that shot is like a slug and the pineapple is like a sprinter. So there’s
all these horrible blood sugar fluctuations that occur.
So in a perfect world, the insulin would match the rise of the blood sugar level
perfectly, but it doesn’t. It doesn’t. And so when you eat anything you want and
shoot up anything you want, you are going to go super high at times because your
food is going to hit before your insulin does. But the other really huge like life-
threatening problem, is if you’re going to eat 100 carbs, you need like 10 units of
insulin. Well, what happens if 10 units of insulin hits before the food does? Your
blood sugar level drops down and you die. You go into diabetic coma, you have a
seizure and you die. Because the second that all that glucose is taken out of your
blood, you go into brain damage. You die. So I don’t give myself any more than 1
or 2 or maybe 3 units of insulin at any one time. But 10 units? I mean, that's like a
lethal dose and that's what these kids are giving themselves when they eat their
100-carb pop, pizza, fry lunch. But who knows what’s going to hit first? Is it the
food or the insulin? Both of them are dangerous.
Dr. Stanislaw
Ben: Right. And then they’re going to get themselves overgrowth of candida and start
getting their palate gravitating towards more sugar and sweets, so now they’re on
a vicious rollercoaster where they can’t get off even if they try.
Jody: Right. Yeah.
Ben: This might be a stupid question but I'm going to ask it anyway. Can you become
insulin resistant as a type 1 diabetic?
Jody: No, it’s not a stupid question. So the insulin resistance is the GLUT4’s get tired of
coming to the surface. The GLUT4’s are like, “I am so tired.” Or you get so
overweight that the GLUT4’s don’t work efficiently. So, yeah. So every 5 pounds
somebody gains, they’re going to need more baseline insulin. My patient just the
other day, she’s like, “I don’t know why, like I'm high all the time now.” And I’m like,
“Didn’t you just tell me you gained 10 pounds last month?” And she’s like, “Oh
yeah.” Well, sorry, you’re going to need more insulin now. Because the more
inefficient the health of the body is, the more insulin resistant they become. The
less the GLUT4’s are going to be responsive.
Ben: That's dangerous. So they gain weight and they go on vacation, they just go on a
spell like we all do, a bingeing spell or what-have-you or we gravitate towards the
TV more, so we’re just becoming less exercised. We perform less exercise, we eat
more, we kind of get in this funk and we gain 10 pounds but yet we don’t really
monitor too much. We’re taking our usual insulin and we see our blood levels are,
say, 180 to 200, we’re like, “Ugh, I'm still taking the same insulin”, and they just
kind of keep going, I assume. Right?
Jody: Exactly, yeah. So they’ll have to increase. So a type 1, so I did a fast, I literally
didn’t eat for 7 days and a lot of people have no idea how I did that. You need
insulin 24 hours a day no matter what because the liver is putting glucose into the
bloodstream 24 hours a day. So even if I don’t eat all day long, I still need a tiny
bit of insulin to counteract the glucose that’s made by my liver. As I said, cortisol
for example, it has a 24-hour cycle and its peak is in the morning, so the body
thinks it’s doing us a favor, it gives us a little bit of extra energy in the morning by
having the liver put even more glucose in the body in the morning. So I have to
take a teeny little bit of insulin to counteract what the liver does all day long. So
that's called slow-acting insulin or long-acting insulin. It’s a slow insulin, it’s called
long-acting. And so I do one shot in the morning, right now 8 units, and 2 units at
night. That insulin has like a 24-hour timeline of use. So it’s supposed to work on
a baseline level. The only purpose of that insulin is to match my liver. However,
when I didn’t eat at all, I actually reduced that dose down to 2 units I literally was
on 2 units of insulin a day and didn’t eat for a week.
So my point is not to tell people they should do that but my point is that there’s a
baseline level of insulin that we need regardless of if we’re eating or not or how
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active we are or not. And that baseline level changes dramatically all the time. So
if I do an 8-hour car ride, I increase my dose that morning from 8 to 10. If I'm going
to be going on a 6-hour hike, I decrease that dose from 8 to 5. So there’s this
baseline internal sugar production that you also have to monitor. All the other shots
I do are directly in correlation with just managing the glucose that I'm eating. But
yeah, there’s that background insulin.
Like right before my period, I get insulin resistant, so there’s something about the
hormones that block the insulin from working. I have to increase my dose for about
3 days before I get my period. The day I get my period, it drops and I have to
reduce that background insulin down. So that’s why I mean like having healthy
blood sugar level was a lot more complex than just “Oh, what did you eat today?”
Ben: For sure. There’s so much hormone control, like you said, with cortisol and
estrogen as well, obviously with cycles. Hearing all this, this tracking is becoming
more and more popular among people. I myself wear an Oura Ring and I love it. A
lot of people will wear 24-hour glucose monitors even though they’re not diabetic.
And it seems to increase their awareness and if you have increased awareness
and you measure things and then you can take action on it. So if you’re a type 1
diabetic and you’re monitoring these things and you know what you’re doing, you
could actually be healthier than a person who’s just kind of winging it, I would
imagine.
Jody: I completely agree. I will often say that having type 1 diabetes has made me
healthier than most people my age because I'm constantly managing what do I
eat, I got to exercise. I'm constantly taking action that is focused on keeping my
blood sugar level in a healthy range, and generally, those actions that are focused
on keeping my blood sugar a healthy range are healthy actions. “I'm not going to
eat that cookie because my number is perfect right now and I don’t want to eat that
cookie because I don’t want to mess up my number.”
Ben: Because you take pride in having a log of great numbers there and that’s a
reflection of you kicking ass and performing at a good level and great level. And
as soon as you eat that cookie, you’re like “Dang it, I just ruined my streak.”
Jody: Well, I do have a continuous glucose monitor and I think it’s more important than
insulin pumps, frankly. Having this kind of 24-hour data has been life-changing.
I’ve had diabetes for 37 years and I’ve had this monitor now for 3 years and it’s
been the most technologically advanced tool for achieving better glucose than
anything I’ve seen in these 37 years. It's just a little teeny wire. It’s like a centime-
long wire that I insert in my subcutaneous tissue and right now it’s on the back of
my arm. I can also wear it on my stomach or anywhere you can just get a little
pinch of skin. I just tape it down, I wear it. It’s waterproof. And then there’s a
transmitter that is in the little device that’s stuck to my skin and that transmitter
sends a signal to a receiver that as long as the receiver is within 20 feet of my
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body, all I have to do to see my blood sugar level is push a button. So right now,
I’m now 130. The insulin has brought me down 20 points now in these past 20
minutes or so.
Ben: And the technical difficulties have gone away.
Jody: Yeah, yeah. My stress is less. Yes. I can put a high alarm and a low alarm level on
it so if I go low it will beep at me, if I go high it will beep at me. And all these years
that I would go to bed and maybe I ate a late dinner or I wasn’t really sure what my
number was going to do in the next hour or two after going to bed, I would have to
set my alarm and wake up and test my blood sugar level. I’ve done that for
decades. And now I just put this thing, I kind of cradle it in my hand as I go to sleep
and if I go high or low, it just wakes me up. Same with like if I'm exercising. I just
keep it in my sports bra and if it’s not vibrating, I know I'm in range. I don’t have to
get off my bike or get off the chair lift and pull out my supplies and test my blood
sugar. If it’s not beeping, I'm in range. Awesome.
Ben: Nice. What’s the name of this device?
Jody: Dexcom is the company. Continuous glucose monitor or CGM is the generic name.
But the company that has the most accurate one in the market right now is called
Dexcom.
Ben: D-E-X-C-O-M?
Jody: Yeah.
Ben: How is it signaling? Is it Wi-Fi, Bluetooth?
Jody: I'm not all techy like that, Ben. I don’t know. it just tells me stuff.
Ben: It’s not connected to your phone, is it?
Jody: So the latest addition, people don’t even need the receiver. The latest addition you
can just see the data on your phone. But I don’t have that version because I don’t
want to carry my phone with me all the time.
Ben: Yeah, that will be Bluetooth.
Jody: Okay.
Ben: Okay, good to know. Because I wanted to start tracking my glucose as well
because I know as you said earlier, high glucose levels are going to be
cardiovascular disease, Alzheimer’s, dementia, all these things.
Jody: Blindness.
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Ben: Yeah. Blindness, that's no good. Erectile dysfunction, that's no good. So there’s a
lot of issues that I want to prevent and maintain. I also want to see okay, if I'm
presenting or doing these summits like I am now, interviews, how much is it
increasing my stress?
Jody: Cool. I have often made my friends wear this so I can watch their blood sugar and
I’ll make them wear and I’ll say “Okay, I want you to eat a whole pineapple now.” I
had friends spike up to 150, for sure. It was awesome, yeah. Next time we’re
together, Ben, you can just wear mine for a couple of days.
Ben: Awesome. Well, I think it’s great for people to hear this too because they think type
1 or type 2, that's not me. Well, it might not be you but your blood sugar levels
could still be all over the place. Your fasting insulin levels or your serum insulin
levels can be super high and if your insulin levels are super high, you’re going to
be super sick. And if your glucose levels are super high, you also might not be sick
but you’re going to be having symptoms and underperforming. And we’re all about
optimizing our life years.
Jody: I just did a TEDx Talk last month and it was called Sugar is Not a Treat. I talked
about how frankly there was a study done last month that showed most primary
care physicians aren’t even picking up on people’s elevated blood sugar levels.
They’re not even picking up on pre-diabetes and maybe even type 2 diabetes. So
if we’re eating a standard American diet and we’re eating even healthy granola
and yogurt that's full of sugar or healthy bread and all this, I mean, there’s so many
carbs out there that are promoted as healthy but they’re still processed carbs. And
you are tiring you’re your beta cells. Type 2 diabetes or pre-diabetes doesn’t just
happen overnight like type 1 did in me. I was healthy one day and the next week I
was sick as a dog.
Type 2 is a slow, slow onset. Over years. Maybe your average blood sugar level
in your 30’s is 85 and then your average in your 40’s is 95 and then your average
in your 50’s is 110 and then it's 130. So there’s a whole different section of tests
that look at different areas of analyzing the blood glucose level that need to be. If
you do your fasting blood glucose, that's not enough. That's one minute in one day
at one time. What were you when you woke up this morning? You were 95. Okay,
great. That doesn’t tell me about what you were in the past 3 months or the past
24 hours.
Ben: Have you read the book “The Case Against Sugar”?
Jody: No.
Ben: Oh my God, you got to read that book. You have to read that book.
Jody: Okay. I'm writing it down.
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Ben: I almost threw it across the room a few times. Because it made me so pissed off
about the sugar industry. It’s a full history of sugar and how it became popularized
and how it became industrialized, how it became lobbied and control of even the
cigarette industry, it’s connected.
Jody: I believe it.
Ben: So it’s really amazing. I got so mad that I had to stop probably the way through. I
got to go back and finish it but is a phenomenal book. I really want everyone to
read this book. It’s very interesting too. I just know my blood sugar was going up
as I was reading it because I was getting so pissed off.
Getting type 2 is like you said a slow, insidious problem. A lot of people aren’t even
aware of it because the doctors aren’t looking at it. They’re just taking a snapshot
of glucose and they just say, “Oh yeah, it’s kind of high” and leave it at that. They’re
not even checking their insulin levels. So if people are looking to optimize their
health, say for me, for example, I want to go to the doctor, what lab tests that are
easy, inexpensive, and quick and reliable would I use?
Jody: So the three main ones really are the fasting blood glucose. It should definitely be
below 95 which means first thing in the morning, below 95. Your hemoglobin A1C
should definitely be below 5.5. So glucose is sticky. Glucose sticks to the
hemoglobin proteins in red blood cells. Red blood cells last for about three months
before they die. They’re constantly making them and they’re constantly dying every
day. So if you look at a nice big volume of your blood, the oldest blood cells in that
blood draw will be about 3 months, maybe 4 months. So you get a picture of how
much glucose is stuck to these red blood cells. Because once it sticks, it stays
there. So an A1C takes a volume of your blood and looks at how much glucose is
stuck to your red blood cells and then you get a picture of what your average blood
sugar level has been for the past three months. So that's why like fasting is only
one second in time. You might be out of range 23 hours a day and 59 minutes and
in that one minute fasting you’re in range. You’re like, so that's going to be enough,
right? So A1C is the opposite end of like kind of giving you the average of what
you’ve been.
Ben: What does A1C mean? What does hemoglobin A1C mean?
Jody: It just refers to the A1C hemoglobin protein. There’s different proteins in the red
blood cell. There’s hemoglobin A1C and then there’s hemoglobin, I don’t know,
A2C maybe.
Ben: So it’s a type of hemoglobin.
Jody: It's a type of hemoglobin that’s in it, yeah.
Ben: In the red blood cell.
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Jody: Yeah, the A1C refers to the type, yeah.
Ben: Before you mention the third lab test. So glucose is sticking to your red blood cells
and once it does that, there’s no coming off. There’s no scrubbing that pan, right?
Jody: Well, the red blood cell eventually dies.
Ben: You have to throw it out basically.
Jody: Yeah. So if I’ve had super high blood sugar levels, my A1C will be high for the next
three months because whatever I'm doing this week, if I'm high every day, these
red blood cells will be in the test I do in three months. So the best way to get an
A1C is to have awesome blood sugar levels for three months and then get the A1C
done.
Ben: So if you have blood sugar binding to your red blood cells, is that going to affect
the oxygen binding?
Jody: Well, you know what, I need to look into that because I can’t breathe as well when
I'm high. If I'm trying to exercise and I'm above 150, I can’t breathe as well. So
that's always been my theory but I actually had never looked at the physiology. It
makes sense because the red blood cells carry oxygen and now the red blood
cells are having glucose stuck to them.
Ben: And it’s looking at hemoglobin.
Jody: And it’s looking at hemoglobin, yeah. So it kind of was like, I don’t know, that's just
own thinking out loud. I haven’t actually looked at the physiology. I’d love to figure
that out but I haven’t.
Ben: Because I'm wondering if that's part of the mechanism of action because why does
high blood glucose – I didn’t say sugar – why does high blood glucose cause
cardiovascular disease. I mean, there’s many reasons because you can get these
reactive pathways and depleting your glutathione and all that, but that's a different
story. I'm just trying to think of oxygen delivery. So if you have glucose sticking to
your hemoglobin and your hemoglobin must be carrying oxygen, okay, so we have
fasting blood glucose, we have HbA1C that we want. Well, we want fasting blood
glucose around, you said, 85?
Jody: Below 95.
Ben: Below 95, okay. And higher than?
Jody: Higher than 72. One thing to know about the hemoglobin A1C is there is some
conflicting data on this But the A1C number correlates with an average blood
glucose level. So if your A1C is 6%, your average blood glucose level is 126. If
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your A1C is 5.5, your average blood sugar level is 111. If your A1C is 5%, your
average blood sugar level is 97.
Now, there’s different charts that have different numbers. They do these kinds of
data collecting studies to get these kinds of charts. But what they’ve learned is that
there is a lot of variation to how A1C charts are actually designed and the reality
is, if you have any sort of blood variant disease, people have different metabolism
of the red blood cells, do the red blood cells only last two months, do they last four
months, there’s a lot of cases unfortunately with A1C is not an accurate predictor
of the average blood sugar level. That's why we’re loving these continuous glucose
monitors now because if you wear continuous glucose monitor 24 hours a day and
it's reading your blood sugar level 24 hours a day, its average is literally the
average of what your blood sugar levels are. It’s not doing this background like,
well, how much glucose is stuck to your hemoglobin. There are so many other
reactions going on with the glucose sticking to the red blood cell. They’ve even
shown that some people’s average blood sugar level on a glucose monitor is like
110 and somebody else’s average blood sugar level is 150, and yet, they have the
same hemoglobin A1C. So the most accurate average is now being realized to be
these continuous glucose monitors. Having said that, A1C is still the gold standard
for monitoring one’s diabetes. It’s absolutely still the gold standard. But if you by
chance have a continuous glucose monitor, that average would allow you to be –
the reality is only 20% or less of type 1’s have a continuous glucose monitor.
People are still poking their finger 10 times a day.
Ben: Say that statistic again?
Jody: I think it’s only 17% of patients with type 1 have a continuous glucose monitor.
Which is a tragedy, because it's the best tool I’ve ever seen in 40 years of diabetes.
Ben: So I'm assuming you’re recommending this to every patient.
Jody: Yes. They’re not cheap. I think I paid a couple grand and then the sensors I have
to change every week are 80 bucks, but you can make the sensor last longer than
a week, that's for sure. So I can make a sensor last 3 weeks and now it's 80 bucks
every 3 weeks And my insurance doesn’t cover it but a lot of people’s insurance
does.
Ben: Good. Okay. So fasting blood glucose, HbA1C, and the third one?
Jody: Oral glucose tolerance test. Now this one, I'm not a real big fan of this one. Now,
you don’t have to drink that disgusting orange... what it is, is they give you 75
grams of this orange sugary glucose and they look at your blood sugar level 1 hour
and 2 hours later. And basically, on an empty stomach you take a ridiculous
amount of straight sugar and you see how well your body can handle it. But you
know what, you could frankly do that, it doesn’t have to be this chemically colored.
Dr. Stanislaw
You could drink 75 grams of orange juice or something and two hours later your
blood sugar level should be below 140.
So these three different tests are really looking at three different angles of can you
stay in the normal range in a fasting state, what are your blood sugar levels on
average over the past three months, and how vital is your body’s ability to bring
your blood sugar level back to normal after ingesting a ridiculous amount of
glucose. So it’s three different angles that all really need to be looked at.
Ben: Makes sense. You have a baseline which is your glucose level, then you look at
your, well, actual, I shouldn’t say baseline, your actual reading currently right now
as you’re fasting blood glucose. Then you have your long-term snapshot look of
the last, say, three months which is HbA1C, and then you’re looking at kind of how
you respond to sugar or glucose in general.
Jody: Exactly.
Ben: See, I corrected myself again. I said sugar once again. Now, in terms of fasting
insulin, I'm a big proponent of checking fasting insulin. In a type 1 diabetic, should
that also be looked at?
Jody: No. I don’t make any insulin.
Ben: But for the rest of the typical population, that would be a good marker.
Jody: Yes, definitely. Because what if their A1C is normal and their fasting is normal, but
their fasting insulin is like through the roof. So that means that eventually, that
fasting insulin is going to die out because the only reason why the numbers are
normal is because their beta cells are working so darn hard to make enough insulin
to stay normal. But that's on its way to burnout.
Ben: Yeah. The beta cells genes are getting dirty because they’re working too damn
hard.
Jody: Exactly. There’s also a test called the C-peptide test. I will do this in newly
diagnosed type 1’s and I’ll also do this in these adults that are thin and active that
are having a high blood sugar level. They’re kind of the type 1 ½ or also what’s
called LADA (Latent Autoimmune Diabetes of Adulthood). So all ages now are
having this autoimmune destruction of beta cells, it's not just kids anymore. And
so when I'm trying to figure out, you know, do you still have beta cell production?
You don’t really seem like you’re a type 2 because your number is suddenly high
and yet you’re think. So I look at their C-peptide test because C-peptide is the
peptide that's attached to what I’ll say dormant insulin. So the body is making
insulin, it's ready to go and then it’s dormant state, it’s attached to a C-peptide. The
second the insulin becomes activated, the C-peptide is cleaved off and then the C-
peptide floats around in the bloodstream. So I personally have had my C-peptide
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tested and I had 0, which means I have no internal insulin production. But I get a
newly diagnosed diabetic, a child or one of these likely type 1 ½ adults, I will literally
monitor the health of their beta cells by looking at their C-peptide levels. If you
have C-peptide, you’re making insulin. Lucky you. They found me a six-year-old
with type 1 diabetes. They found me within two weeks of diagnosis. I immediately
got them on my beta cell preservation protocol and I just had a follow-up call with
them yesterday and they are not on any insulin.
Ben: That's a brilliant transition. So I got two questions for you before we elaborate on
this patient. So C-peptide is something that's secreted by the pancreas when it’s
working, is that correct?
Jody: So inside the pancreas are the beta cells. The beta cells make insulin. When insulin
goes from dormant to active, the C-peptide on the dormant insulin molecule is
cleaved off and now the C-peptide molecule is floating around in the blood. It's just
a by-product of insulin being activated. So if you have C-peptide in your blood, that
means your beta cells are working and you’re making insulin.
Ben: So if it’s low or absent, well, if it's low then you are in trouble.
Jody: Yeah. That means your beta cells are weak. Of course you have to do a fasting
because of course it’s going to be different numbers after you just finished eating.
So a fasting C-peptide shows you your baseline. It should be above 1. I don’t know.
I can’t remember what the units are but it should be above 1. Like a normal C-
peptide in the fasting state is like from 1 to 4.5. If a patient comes to me and their
C-peptide is between .5 and 1, then they might be able to get away with only having
some long-acting background insulin. Once it gets down below .5, they’re likely
going to need long-acting and short-acting. But I have seen C-peptide come back.
As soon as I get people on a beta cell preservation protocol, it can come back.
Ben: We’ll get into this in just a second here because I got another question. So if a
person does not have type 1 diabetes yet and they’re listening to this and they say,
“You know what, I heard in the beginning of this that type 1 diabetes is an
autoimmune destruction of the beta cell in the pancreas.” So they’re thinking like,
“You know what, I already have Hashimoto’s thyroiditis, I might have other
autoimmune conditions. Am I at risk for beta cell destruction?”
Jody: Well, 16% of type 1’s have celiac disease, 17% have Hashimoto’s. If you have any
autoimmune disease, your risk of getting another one is increased. So yes, I mean,
think of me, I’ve got these tubes in my ears and I wasn’t breastfed and I believe if
I knew what I knew now, back when I was two years old, I would have been like,
“Hey mom, we need to get me off wheat, dairy, sugar. We need to raise my vitamin
D. We maybe need to do a detox if I had mercury.” I would have done all that. So
yes, you can actually get a family member. So let’s say a four-year-old gets type1
diabetes. Well, there’s a study called TrialNet that wants as many siblings of others
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with type 1 as they can find because the antibodies to the insulin and the beta cells
appear in the blood before diabetes. So a lot of the studies are looking at once we
have the antibody reactions, how can we reverse it before it goes into full-blown
type.
So I have a couple families that one child has type 1 and so then I put the beta cell
preservation protocol on everybody in the family. The dad got tested for the
antibodies and he apparently has antibodies against his own insulin and then he
freaked out and the daughter does.
So there’s four steps to my beta cell preservation protocol. Anybody that’s

concerned they’re going to get type 1, you can find out if you have antibodies
against insulin islets and then you can go on a beta cell preservation protocol.
Ben: So I'm hearing another lab test because you can check for C-peptide. I'm just
thinking here, the folks are listening and they say, “Well, you know, I have an
autoimmune condition and I don’t want to get type 1. I don’t want...” what was it,
TADA?
Jody: LADA.
Ben: LADA. “I don’t want to get LADA.” Late autoimmune –
Jody: Disease of Adulthood, yeah.
Ben: Say it again?
Jody: LADA - Latent Autoimmune Diabetes of Adulthood.
Ben: Right. So they’ve got Hashimoto’s or celiac and they say, “Okay, I don’t want to
get that.” So how do I check to see if my beta cells are under attack? I can look at
my C-peptide and then you say they can look at their antibodies with insulin and
antibodies against their beta cells.
Jody: Yeah. So there’s four auto antibodies that are markers of beta cell autoimmunity
and type 1 or LADA or type 1 ½. I can just give you the letters first. One is ICA
which islet cell antibody. One is GAD65 which is an antibody to glutamic acid
decarboxylase. One is IAA which is just insulin auto antibodies. Those are the
three most common and then the fourth one is IA-2A which is against the protein
tyrosine phosphatase. So anybody can just Google autoimmune tests for type 1
diabetes. Literally if anybody is concerned they’re going to be on the road of type
1, you get these. The standard marker for LADA is the GAD antibody. If you have
GAD and you’re an adult, your diagnosis is LADA.
Ben: That's interesting. Do they have neurological problems in the brain at all? Because
GAD, will they have increased glutamate levels?
Dr. Stanislaw
Jody: That's an interesting question. What I learned is that we make auto antibodies
because things get presented to the immune system that shouldn't be, right? So
the glutamic acid is within the brain cell. So the antibodies in the bloodstream don’t
suddenly get inside the brain cell and attack it. Because I thought that as well.
Ben: But the antibodies bind to the receptor of the GAD gene which then could block
glutamate from...
Jody: You know, I don’t know. I should actually retract that last statement. I don’t know
the full. I just found out about this whole... because there’s something about GABA
too.
Ben: Right, exactly. Because if you have an antibody floating around against a certain
cell or a receptor and I'm assuming GAD65 is the receptor but I'm just assuming
that, I don’t know. But if the antibody binds to it, then glutamate can’t bind to it.
Jody: Well, I think it’s an enzyme, glutamic acid decarboxylase.
Ben: Yeah. And what that enzyme’s job to do is to convert glutamate to GABA.
Jody: Oh yes, you’re exactly correct.
Ben: So I wonder if irritability would be increased in these individuals and excitation,

headaches, insomnia.
Jody: You’re right. You’re reminding me now of what that enzyme does. I'm speaking
outside of my expertise right now but I did kind of dive into this a little bit and I
talked to some autoimmune experts in San Diego. I’d have to get back to you on
what their... because I thought that too. Because I know plenty of diabetics that
have a lot of anxiety and I thought, well, maybe their GAD is high and their GABA
is low.
Ben: Yeah, that's really interesting. Because people are saying here, “God, you know
what, I have anxiety. I have headaches. I have autoimmune diseases.” Because
there’s so much. We’re going to sidestep here a little bit kind of into my
wheelhouse. And because a lot of people will do genetic testing and they’ll get this
genetic report back that's like 70 pages and it’s got all these glutamate
decarboxylase SNPs and they’ll say that they’re homozygous for GAD variations.
And then I'm like, that explains everything. But then I look on the research and I
look and these SNPs are not clinically relevant. PubMed, National Library of
Medicine says there’s no SNPs that are saying that if you have a polymorphism or
a SNP in the GAD gene, that it’s going to be altered in function. And I just wrote
an article on this probably a month or two ago that you can get antibodies to the
GAD gene or the GAD enzyme, like receptor, and you just told me a cool
mechanism because I don’t know where they came from and that can definitely
increase your anxiety because it's blocking the body’s ability to bind glutamine to
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convert it to GABA. So that's really cool because I keep telling people, look, most
of the time it’s environmental. It’s not. It doesn’t have anything do with your SNPs.
It’s environmental so screw the GAD SNPs in your report which are nonsense.
Focus on do you actually have antibodies and you just told us a phenomenal tool
to evaluate that which is the antibody against the GAD65 which is really neat.
Jody: We’re like thinking out loud right here. Because I did, I started digging into this a
couple of months ago but then life just got away with me and I talked to a couple
of researchers. So I was like, “Wait a minute. Should type 1’s take GABA?” Like,
are we all completely stressed out and anxious because we’re low on GABA?
Because our antibody to the enzyme to make GABA, you know. So I would love to
dig further into this with you because I kind of got nowhere with my digging and
then just kind of gave up.
Ben: I’ll answer that really fast. If I gave patients or clients GABA and they have high
glutamate, the problem is, the glutamate is still high but they have this wired but
tired feeling and they just feel. Because the glutamate is still high and now they got
this GABA and they’re just all whacked. So you can load them with vitamin B6
because that’s the cofactor for GAD. You can try to saturate the body with vitamin
B6 so that will bind to the receptor over the antibody and you will compete with it
that way. So vitamin B6 and magnesium can be helpful for that but not to detract
from this. So I’ve got some great resources for these guys and gals. So we’ve got
fasting insulin for non type 1 diabetics people are going to be interested in. We
have fasting blood glucose; it should be higher than 72 and lower than 95. HbA1C
should be less than 5.5. Or glucose tolerance test where you just take a big dose
of orange juice. And then C-peptide fasting should be greater 1. Then the
autoimmune test for type 1 which is a bunch of different markers which is really
cool. Now, you said that you had a 7-year-old thereabout who was newly
diagnosed with type 1 diabetes and you caught him early enough and reversed it.
Jody: I won’t say reversed it because I'm assuming he still has a lower C-peptide. I
encouraged the family yesterday to get a C-peptide because I want to see where
it’s at and I said you need to follow the beta cell preservation protocol, likely forever.
But here’s my theory. So number one is you have to avoid glucose toxicity. High
glucose itself is toxic to the beta cells. So here are these newly diagnosed kids.
They still have probably 20% of the beta cells left. They go to the hospital, they’re
diagnosed, and they’re told “eat anything you want and just take insulin for it.” Well,
they’re going to kill off their beta cells really fast because they’re going to have
crazy blood sugar levels. And if you have a broken foot, you don’t go running, right?
You rest it. If you have weakened beta cells, don’t make them work. Don’t eat a
bunch of grains and carbs. So they immediately did a protein veggie-based diet
the day that they met me. So they went with all the protein in veggie. So number
one, they’re not demanding a lot from the beta cells and number two, they’re having
Dr. Stanislaw
better blood sugar levels so they’re not having glucose toxicity kill the beta cells.
So that's the most important part, really.
Number two is exercise. We already talked about the more you exercise, the more
the GLUT4 receptors come to the surface and take glucose in without the need for
beta cells functioning and making insulin. So your body is more efficiently using up
the glucose in your blood without the need for insulin when you exercise.
Especially weight training. The more muscle you have on the body the more you
eat up glucose 24 hours a day. The more muscle tissue eats up glucose. The more
muscle tissue you have, the more glucose you eat up in the absence of insulin.
Number three is there’s actually some supplements. Niacinamide has been shown,
it’s a form of niacin, it has been shown to protect, to elongate the honeymoon
period. The honeymoon period is you’re diagnosed with diabetes but you still have
beta cells working. BioGymnema, well, gymnema sylvestre is the herb. The
product I use is BioGymnema by Ayush. Gymnema has been shown to protect
beta cell mass and actually so has green tea. Of course, keeping your vitamin D
in a health range is important to keep that immune system strong. Even essential
fatty acids have been show to be protective. But the three most important ones I
always say are niacinamide, green tea and Gymnema because they literally work
directly on the beta cells. So that's step three.
So all three of those are important to keep in place but besides the vitamin D being
adequate, none of those really take care of the immune system. All three of those
protect the beta cells, exercise, low carb. Certain supplements, they protect the
beta cells. But if we can really reverse the autoimmune destruction, the fourth step
is all about gut healing. Run a food allergy test, remove the food allergens, remove
gluten, remove dairy, increase probiotics, increase L-glutamine to heal the GI tract.
Get that gut healed and see if we can get those auto antibody numbers to go down.
In that case, yeah, then I could potentially cure him. But right now if he just keeps
eating low carb and exercising, he’s just functioning well on a weakened system.
Can I say that I completely give him back to being able to eat two bananas and
three bananas and be fine? I don’t know. But I'm like, now is not the time to
experiment with testing how “cured” he is. He needs to eat low carb, he needs to
stay fit.
Ben: It’s brilliant. I'm so glad you brought that up because cure is not really... because I
remember I think it was first quarter at the past year when we’re in the room with
Brad Lichtenstein and he goes, “Define cure. Is cure possible?” He trapped us all.
He definitely trapped me. Because like, well, yeah, cure is possible. But it’s not
possible. Health is a four-letter word; it's work. It’s constant maintenance of
keeping yourselves happy and healthy and yourself happy and healthy. And if your
beta cells have gotten a beatdown for various reasons and you have 20 percent
left or 30 percent left of them, well then you need to be very careful with that. It’s
Dr. Stanislaw
just like having a dirty MTHFR or dirty COMT gene. You’re born with a slower
MTHFR gene like I am, I can’t handle a bunch of alcohol like some other people
can. One drink, I'm done. And my MTHFR, I’ve got 20 to 30 percent capacity of my
MTHFR gene just like this kid has got like a 20 to 30 percent capacity of his beta
cells. So I have adjusted my lifestyle around my decreased performing MTHFR
and this kid better do the same thing or he’s going to get type 1 diabetes and be in
a world of hurt. But he won’t be in a world of hurt if he’s like you who are monitoring
your glucose all the time and making lifestyle changes which optimize your
performance and your health and you looking fantastic. You look super healthy,
you’re vibrant and you’re doing awesome despite having type 1 because you’ve
altered your lifestyle. You haven’t cured it and you can’t cure it anymore, your beta
cells are dead.
Jody: Well, it's interesting actually. I just actually watched a TEDx Talk and they stained
and analyzed the pancreases of patients that have passed but had diabetes for
decades. They’re finding that these type 1 diabetics who have had been on insulin
injections for decades actually still have beta cells in their pancreas. So the theory
from that talk is that I have beta cells, these cute little teeny buds just keep every
day trying to come up and my immune system just keeps killing them.
Ben: What are systemic enzymes that would help get rid of those antibodies or what-
have-you?
Jody: Well, there is a lot of studies going on right now changing like T cells because
there’s the T cells that are attacking it and they are looking at studies to try to stop
that to alter the T cells that are acting inappropriately and then curing us that way.
That would be awesome if they could do that.
Ben: Yeah, it would be curious too to see if the VDR SNPs that are out there, research
is really conflicting. I'm wondering if the vitamin D ratio, the 25 hydroxy to 125 is
off in type 1 because if the 125 levels of vitamin D are a lot higher than a 25, say,
1 ½ to 2 times higher, then there’s some type of infection or autoimmune thing
going on so you could look at the 25 hydroxy and 125 vitamin D ratio. We got an
article on DrBenLynch.com about that, it’s very brief but spotting infections is really
important. Any autoimmune condition, you’re going to have a pathogenic
component too. Well, possibly a pathogenic component. Not necessarily. But you
could have an infection of some sort – Lyme, mold, yeast overgrowth, parasites,
viruses, bacteria – that you’re exciting the immune system to a point. We know
that dairy was an allergen for you because you have your ear tubes. I wonder if
you also had because like you said, low vitamin D so that would make you
susceptible to an infection so do you have any hidden viruses or bacteria that are
keeping your antibodies up because your body is constantly trying to fight this
thing.
Dr. Stanislaw
Jody: So 30 percent of patients with type 1 have had a recent intense viral infection, so
that's been definitely been correlated. There are so many potential environmental
triggers that's not clear but what we do know, it’s not just a single one or we would
have identified it by now.
Ben: You did mention before we got on air here that you said that – well, not air but
whatever, interview. That there’s one gene that's associated with 40 percent of
type 1 diabetes. What is that? What class of genes?
Jody: The HLA, so it’s the human leukocyte antigen complex. But it’s very confusing
because these certain alleles, some may increase risk, some seem to have no
effect, some even seem to be protective. So nobody in my family has type 1 which
is common but then you’ll meet a family where all four kids have type 1. So the
research that I was reading earlier, it says “These haplotypes seemed to increase
the risk of an inappropriate immune system response to beta cells. However, these
variants are also found in the general population and only about 5 percent of
individuals with the variants developed type 1. HLA variations account for
approximately 40% of the genetic risk of the condition. However, other HLA
variations appear to be protective. So clearly, other contributors such as
environmental factors and variations in other genes are also thought to influence
the development of this complex disorder.”
Ben: Fantastic. Again, the environment. Again, it’s a dirty gene. It’s not a deadly gene
or a disease-causing gene. It’s just dirty. You can clean it up. That's the whole
point, the whole concept.
Jody: In the past 20 years worldwide, incidence of type 1 has been increasing by 2 to 5
percent each year.
Ben: Wow.
Jody: Type 1 diabetes accounts for only 5 to 10 percent of cases of diabetes worldwide
because in most people, 90 to 95 have type 2 which of course the body continues
to produce insulin but becomes less able to use it is how they say. Now, here’s an
interesting statistic: The disorder occurs with similar frequencies in Europe, the
UK, Canada, and New Zealand. Type 1 diabetes occurs much less frequently in
Asia and South America.
So it’s interesting, I find that very fascinating. So type 1 diabetes occurs in 10 to

20 people per 100,000 per year in the US. So that's also in Europe, UK, Canada
and New Zealand but not Asia and South America.
Ben: I wonder how the frequency of type 1 diabetes is too along kind of the vitamin D
winters that we get.
Dr. Stanislaw
Jody: Yes. Those cities are always higher. Vitamin D cities is higher, yeah. Ben, I
absolutely love chatting with you but I actually need to wrap up.
Ben: So do I, so do I. So real quick question before we go. Ketogenic diet - yay or nay?
Jody: I have patients that love it. I actually haven’t looked into the science of what will
they look like in 5, 10, 20 years. Is it nutritionally sustainable? Are they getting all
the nutrients they need? Obviously, they do get great blood sugar levels because
they’re really eating very few carbs but yet I know people that are advocates of
eating a plant-based, no fat diet for diabetes. There’s these two guys in San Diego
that have thousands of followers and they tell people they should go raw vegan
and eat fruit all along. So frankly, I don’t get involved in exactly the science of the
diet. I know that I want them to have good blood sugar levels and that's my main
focus. So if you can achieve good blood sugar levels, that's my focus. But what is
the diet actually doing to the whole body over time, I don’t know.
Ben: Got it. So Jody, phenomenal session with you. Thank you. Appreciate your insights
and knowledge and what you’re doing. If you are interested in learning more about
Jody, you can go to DrJodyND.com and the website is there on the slides for you.
She lives in Sun Valley, Idaho. She works with patients worldwide with type 1
diabetes and if you’re concerned about it, we’ll also consider evaluating the slides
that we talked about.
Jody: I also have a link that people can sign up for a free call with me. That will go on the
slide. It’s consultwithdrjody.com/type1. So you immediately get taken to my
calendar, you can sign up for a free intro call. You fill out a little questionnaire and
if you want to work with me privately, I do that. Also, I know, Ben, you were going
to talk about this but I’ll just plug it here so we can wrap up – is my passion is
helping people with type 1 diabetes live vibrant lives and I’ve seen millions of
people – well, millions. But I know there’s millions of people that really do not have
access to good care of type 1 diabetes and so I'm putting together an online
course, The 5 Essentials of Thriving With Type 1. And my goal is that this course
reaches millions of people with type 1 around the world because the reality is most
doctors do not know how to treat type 1’s. It’s too complex. And the few that do,
it’s really hard to get enough time with them. So my goal is to empower patients at
home with an online course through learning all of my information that I’ve used to
change people’s lives with type 1 for the past 5 years and put it into a very easily
accessible course. So, stay tuned. There’s a free gift on my homepage of my
website and if you sign up for the free gift, 3 Essentials of Lowering Your
Hemoglobin A1C, you’re immediately on my newsletter list which then you’ll be
updated with as soon as that course goes live. Which will probably be somewhere
in 2018.
Ben: Fantastic. Great. Because I don’t know how to help people with type 1 diabetes.
That's for sure.
Dr. Stanislaw
Jody: You send them to me.
Ben: Yeah, I will do that. I just did. So fantastic. Thank you again and appreciate your
helping the Dirty Genes Summit.
Jody: Awesome. Thanks so much, Ben.
Ben: Yup. Bye-bye!
Jody: Bye.

Ben Lynch Blood Sugar Genes

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Blood Sugar Balancing Techniques

Dr. Jody Stanislaw

• Rare for type 1 diabetes to be genetic

• Likely to be environmental factors.

• Things that weaken the immune system:

Constant Balancing Act

• Stress causes the body to make adrenalin

• Tells the liver to secrete glucose into the bloodstream

• Blood sugar peaks in the morning

• Target to stay between 80 - 120 blood sugar level

Type 1 vs Type 2 Diabetes

• Your body is ignoring insulin

• Carbs - There is no essential carbohydrate

2. Hemoglobin A1C should be below 5.5

3. Oral Glucose Tolerance test

4. Fasting C-Peptide test (fasting should be greater than 1)

The Case Against Sugar by Gary Taubes

Don't wait to see where your genes take you...

Q We respect your privacy

Dirty Genes removes the overwhelm of genetics.

• Quizzes. How are your genes working?

Order your copy today from:

Dirty Genes Summit:

The information, comments, and opinions expressed in this presentation

*These statements within have not been evaluated by the US Food

So, Jody Stanislaw is a practicing naturopathic physician in gorgeous Sun Valley,

Jody: Fifteen years, Ben.

Ben: Yeah, we’re getting old. We’re getting old.

Jody: No, I'm not.

Ben: Yeah. Older, wiser.

Jody: That's right.

Jody: Hello, Ben.

Ben: Seven. What happened?

Ben: Wow. And how did that affect you?

Ben: Awesome. That's cool.

Jody: I don’t know. I really don’t know.

Jody: Exactly, yeah.

Jody: Exactly, yeah. Leaky gut for sure.

Ben: I can imagine.

Jody: Absolutely, yes. So very, very different of course. Type 1 diabetes is an

Jody: Yeah. Your body becomes more efficient.

Ben: That's cool.

Ben: That's so cool.

Jody: Isn’t that cool? Yes.

Jody: Right. Yeah.

Ben: And the technical difficulties have gone away.

Ben: Nice. What’s the name of this device?

Ben: How is it signaling? Is it Wi-Fi, Bluetooth?

Ben: It’s not connected to your phone, is it?

Ben: Yeah, that will be Bluetooth.

Jody: Okay. I'm writing it down.

Jody: I believe it.

Ben: So it’s a type of hemoglobin.

Jody: It's a type of hemoglobin that’s in it, yeah.

Ben: In the red blood cell.

Jody: Yeah, the A1C refers to the type, yeah.

Jody: Well, the red blood cell eventually dies.

Ben: You have to throw it out basically.

Ben: And it’s looking at hemoglobin.

Jody: Below 95.

Ben: Below 95, okay. And higher than?

Ben: Say that statistic again?

Ben: So I'm assuming you’re recommending this to every patient.

Jody: No. I don’t make any insulin.

• Blood sugar peaks in the morning 

3. Oral Glucose Tolerance test 

4. Fasting C-Peptide test (fasting should be greater than 1) 

Dirty Genes removes the overwhelm of genetics. 