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ON
NON-ST SEGMENT
ELEVATION
MYOCARDIAL
INFARCTION
(NSTEMI)
CARDIOVASCULAR NURSING
INTRODUCTION
Cardiovascular disease (CVD) generally refers to conditions that involve narrowed or blocked
blood vessels that can lead to a heart attack, chest pain (angina) or stroke. Other heart conditions, such
as those that affect your heart’s muscle, valve rhythm, also are considered forms of heart disease. It is
one of the leading cause of death worldwide, accounting for 17.3 million deaths in 2008 (or ~ 30% of all
deaths worldwide). ACS is now a leading cause of mortality in the Asia-Pacific region, accounting for
around half of the global burden. In fact, due to rapid industrialization, fewer than 50% of patients across
the Asia-Pacific region attain the National Cholesterol Education Program Adult Treatment Panel lII low-
Myocardial infarction (MI), commonly known as a heart attack, occurs when blood flow decreases
or stops to a part of the heart, causing damage to the heart muscle. The most common symptom is chest
pain or discomfort which may travel into the shoulder, arm, back, neck, or jaw. Often it occurs in the
center or left side of the chest and lasts for more than a few minutes. The discomfort may occasionally
feel like heartburn. Other symptoms may include shortness of breath, nausea, feeling faint, a cold sweat,
or feeling tired. About 30% of people have atypical symptoms. Women more often present without chest
pain and instead have neck pain, arm pain, or feel tired. Among those over 75 years old, about 5% have
Myocardial infarction (MI) refers to tissue death (infarction) of the heart muscle. The phrase
“heart attack” is often used specifically to refer to a myocardial infarction and sudden cardiac death. An
MI is different from-but can cause-cardiac arrest, where the heart is not contracting at all or so poorly
that all vital organs cease to function, thus causing death. It is also distinct from heart failure, in which the
pumping action of the heart is impaired. However, an MI may lead to heart failure.
Chest pain may be accompanied by sweating, nausea or vomiting, and fainting, and these
symptoms may also occur without any pain at all. Loss of consciousness due to inadequate blood flow to
the brain and cardiogenic shock, and sudden death, frequently due to the development of ventricular
fibrillation, can occur in myocardial infarctions. Cardiac arrest, and atypical symptoms such as palpitations,
occur more frequently in women, the elderly, those with diabetes, in people who have just had surgery,
Non-ST segment elevation myocardial infarction (NSTEMI) is a heart attack or MI that may not
cause changes on electrocardiogram. However, chemical markers in the blood indicate that damage has
occurred to the heart muscle. In NSTEMI, the blockage may be partial or temporary, and so the extent of
According to the latest WHO data published in 2017 Coronary Heart Disease Deaths in Philippines
reached 122,950 or 19.86% of total deaths. The age adjusted Death Rate is 191.79 per 100,000 of
as needed to enable the heart to receive a fixed amount of oxygen. Increases in myocardial oxygen
demand such as physical exertion or an increase in BP causes the arterioles to dilate to maintain oxygen
supply to the heart. In atherosclerosis, plaque narrows the larger conductance vessels, causing the
arterioles to dilate under normal or resting conditions to prevent ischemia. Stress, exercise, or any
increase in myocardial oxygen demand in the setting of limited oxygen supply results in ischemia and
angina.
Patients Profile
Name: R.B
Sex: Male
ECG that displays each heartbeat as a waveform is used to determine if an NSTEMI or a STEMI has occurred
in a person. When looking at the waveforms of a person who has had an NSTEMI, they appear very distinct
from those of someone who has had a STEMI. The damage to the heart from an NSTEMI is less serious
than that from a STEMI. However, any heart attack can be very frightening.
No progression to Q wave
HISTORY
HISTORY OF PRESENT ILLNESS:
Few hours prior to admission, the patient had chest pain radiating to the right shoulder and upper
extremities while he was working that why his officemates decided to bring him home. While at home,
the pain increases and he loss consciousness that’s why his wife decided to bring him to the hospital.
He was admitted at Divine Mercy Wellness Center (2010) due to hypertension. He was given
According to the patient, both his parents died due to heart disease.
ANATOMY AND PHYSIOLOGY
Our hearts beats 100, 000 times a day, pushing 5,000 gallons of blood through our body every 24
hours. It delivers oxygen-and nutrient-rich blood to our tissues and carries away waste.
The heart contracts at different rates depending on many factors. At rest, it might beat around 60 times
a minute, but it can increase to 100 beats a minute or more. Exercise, emotions, fever, disease, and some
medications can influence heart rate.
The left and right side of the heart work in unison. The right side of the heart receives deoxygenated blood
and sends it to the lungs; the left side of the heart receives blood from the lungs and pumps it to the
system. Newly oxygenated blood returns to the left atrium via pulmonary artery to the lungs, it travels
through tiny capillaries on the surface of the lung’s alveoli (air sacs). Oxygen travels into the capillaries,
and carbon dioxide travels from the capillaries into the air sacs, where it is breathed out into the
atmosphere.
The muscles of the heart need to receive oxygenated blood, too. They are fed by the coronary arteries on
the surface of the heart.
Where blood passes near to the surface of the body, such as at the wrist or neck, it is possible to feel your
pulse; this is the rush ventricle and the pulmonary artery.
Most people are familiar with the sound of a human heartbeat. It is often described as a “lub-DUB” sound.
The “lub” sound is produced by the tricuspid and mitral valves closing, and the “DUB” sound is caused by
the closing of the pulmonary and aortic valves.
The heart’s electrical system
To pump blood throughout the body, the muscles of the heart must be coordinated perfectly- squeezing
the blood in the right direction, at the right time, at the right pressure. The heart’s activity is coordinated
by electrical impulses.
The electrical signal begins at the sino-atrial strong and stretchy, which helps push blood through the
circulatory system. Their elastic walls help keep blood pressure consistent. Arteries branch into smaller
arterioles.
Veins: these carry deoxygenated blood back to the heart and increase in size as they get closer to the
heart. Veins have thinner walls than arteries.
Capillaries: they connect the smallest arteries to the smallest veins. They have very thin walls, which allow
them to exchange compounds with surrounding tissues, such as carbon dioxide and water.
Health Perception- Health According to the patient, he The patient perceives himself
health.
of water a day.
Elimination Pattern Patient has a regular bowel Patient has a regular bowel
Sleep-Rest Pattern Patient sleeps 8-10 hours a day During confinement, patient
during his off but during their sleeps 6-8 hours. Patient naps 2-
not busy.
hypertension (Amlodipine 10
mg once a day).
Coping- Stress Tolerance Pattern According to the patient, his Patient considers his illness a big
children are his best stress change in his life and during
guide him to surpass all this the patient feels hopeless and
Values- Belief Pattern Patient considers the almighty Patient considers the almighty
and his faith with the Lord is his and his faith with the Lord is his
process.
Laboratory exams/ diagnostic exams
1. Electrocardiogram
It record the electrical activity of the heart over a period of time using electrodes
placed on the skin.
Result: Non-ST segment elevation
2. CPKMB
Is a cardiac marker used to assist diagnoses of an acute myocardial infarction
Result: 20.95 U/L Normal Value: 0-24 U/L
3. Troponin T
Is useful in the laboratory diagnosis of heart attack because it is released into the
blood-stream when damage to heart muscle occurs.
Result: POSITIVE
4. Troponin I
Is a cardiac and skeletal muscle protein useful in the laboratory diagnosis of heart
attack
Result: 0.52 ng/mL Normal Value: <0.3 ng/mL
NURSING CARE PLANS
vasoconstrictor causes
because of sympathetic
cardiac output is
compromised. Tachycardia
may be sustained as a
compensatory response.
environment. Restrict
visitors as necessary.
Provide supplemental
oxygen as indicated.
Increases oxygen available
reversal of ischemia.
ASSESSMENT DIAGNOSIS PLANNING NURSING RATIONALE EVALUATION
INTERVENTIONS
Subjective: Activity At the end Document heart rate Trends determine Goal met. The
“hinihingal ako kapag Tolerance of the shift, and rhythm and patient’s response to patient reports
pumupumta ako sa CR saka R/t the patient changes in BP before, activity and may decreased
sumasakit ang dibdib ko”,as Imbalance reports during, and after indicate myocardial chest pain and
verbalized by the patient. between absence or activity. Correlate oxygen deprivation shortness of
myocardial decrease with reports of chest that may require breath when
Objective: oxygen chest pain pain or shortness of decrease in activity doing ADL’s
BP=150/100 supply and and breath. level and/or return to with support.
HR=110 demand shortness bedrest, changes in
Generalized of breath medication regimen,
weakness when or use of
doing supplemental
ADL’s oxygen.
Accurate
information about
Answer all the situation
questions reduces fear,
factually. Provide strengthens nurse-
consistent patient
information; relationship, and
repeat as assist patient and
indicated. SO to deal
realistically with
situation.
Attention span
may be short, and
repetition of
information helps
with retention.
Sharing
information elicits
Encourage patient support and
and SO to comfort and can
communicate with relieve tension of
one another, unexpressed
sharing questions worries.
and concerns.
Conserves energy
and enhance
Provide rest coping abilities.
provides and/or
uninterrupted
sleep time, quiet
surroundings, with
patient controlling
type, amount of
external stimuli. Helps patient
and/or SO identify
Encourage realistic goals,
discussion about thereby reducing
postdischarge risk of
expectations. discouragement in
face of the reality
of limitations of
condition and/or
pace of
recuperation.
DRUG STUDY
Anti-
inflammatory
effects are are
mediated by a
decrease in
prostaglandin
synthesis. It also
decreases
platelet
aggregation.
NAME CLASSIFICATION MECHANISM OF INDICATION CONTRAINDICATIONS ADVERSE NURSING
ACTION AND CAUTIONS EFFECTS CONSIDERATIONS
CAPTOPRIL Ace Inhibitor Blocks ACE from Treatment of Hypersensitivity Patient’s Administer 1hr or
converting exhibit no
angiotensin I to hypertension Hx of angiodema adverse 2hrs after meals
angiotensin II, a effects
potent alone or with
vasoconstrictor,
leading to combination Used cautiously with: Monitor patient
decrease blood
pressure, with thiazide Impaired renal closely for fall of
decreased function, CHF, salt or
aldosterone type diuretics. volume depletion BP secondary to
secretion, a small
increase in reduction in fluid
potassium level,
and sodium and Treatment of volume (excessive
fluid loss;
increased CHF in perspiration and
prostaglandin
synthesis also patients dehydration,
involve in the
antihypertensive unresponsive vomiting,
action.
to diarrhea);
conventional excessive
Respiratory
System: Cough,
dyspnea,
bronchitis,
sinusitis,
pharyngeal
discomfort,
epistaxis, rhinitis,
respiratory
congestion.
NAME CLASSIFICATION MECHANISM INDICATION CONTRAINDICATIONS ADVERSE NURSING
OF ACTION AND CAUTIONS EFFECTS CONSIDERATIONS
ALDACTONE Potassium- Mild diuretic Primary Acute renal Dizziness, Take as directed
sparing diuretic that acts on hyperaldosteronism, insufficiency, headache, with a snack or
the distal adjunctive therapy progressive renal drowsiness, meals to
tubule to in the treatment of failure, hyperkalemia, rash, minimize GI
inhibit edema associated and anuria. Clients cramping, upset. Report if
sodium with CHF, nephrotic receiving potassium diarrhea, nausea, bloating,
exchange for syndrome, hepatic supplements, hyperkalemia, anorexia,
potassium, cirrhosis, treatment amiloride or hirsutism, vomiting or
resulting in of hypokalemia or triamterene. gynecomastia, diarrhea persist.
increased prevention of deepening of
secretion of hypokalemia in the voice, Instruct client not
sodium and patients at high risk irregular to drive/operate
water if hypokalemia menses. machinery until
conservation occurs; essential drug effects are
of hypertensiom. realized; may
potassium. cause drowsiness
An or uneasy galt.
aldosterone
antagonist. Report if deep,
rapid
respirations,
headaches or
mental slowing
occurs; may
indicate
hyperchloremic
metabolic
acidosis.
Remind client
that intake of
spironolactone
may cause breast
swelling and
diminished sex
drive due to
reduction of
testosterone
levels.
Since the drug is
metabolized in
liver, report
jaundice, tremors
or mental
confusion’ may
develop hepatic
encephalopathy
with liver disease.