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Dr Alex Harper
Temporal arteritis
Retinal detachment
Retinal artery occlusion
Retinal vein occlusion
Vitreous haemorrhage
Macular degeneration
Note: the eye is "white" with a normal anterior segment (clear cornea and lens)
History
One eye or both eyes?
Onset and progression
- very sudden onset suggests a vascular cause
- gradual deterioration suggests degenerative disease( eg cataract)
Associated visual symptoms
- flashes suggest retinal traction (but can be cortical eg CVA, migraine)
- floaters suggest vitreous debris
Past ocular history
- trauma and myopia are risk factors for retinal detachment
Systems review
- in elderly patients, ask about headache and polmyalgia(?temporal arteritis)
- any history of diabetes, including a "touch of sugar"
- cardiovascular disease, TIA symptoms suggest emboli
Examination
1. Visual acuity
2. Pupil reaction to light (? relative afferent pupil defect= RAPD)
3. Visual Field
4. Fundus examination
Temporal Arteritis
History
Elderly patients (age >65)
Sudden and severe loss of vision in one eye initially
Systemic symptoms are headaches, scalp tendemess,malaise, jaw claudication
Examination
Vision 6/60 or worse RAPD
Extensive visual field loss
Pale swollen optic disc (anterior ischaemic optic neuropathy), rarely CRAO.
Management
Aim to prevent loss of the other eye!
Urgent ESR (expect >60)
Prednisolone l00mg stat.
Urgent referral to ophthalmologist
Temporal artery biopsy will confirm the diagnosis
Retinal detachment
History
Patients may notice an enlarging shadow in peripheral vision(not just a floater)
Sudden loss of central vision occurs when the macula detaches
Flashes and floaters are common associated symptoms
Ocular history of trauma, surgery and myopia.
Examination
Acuity normal = macula "on"
Acuity poor = macula "off'
RAPD
Absolute field defect corresponding to area of detached retina
Fundus examination is diagnostic (but may be difficult to pick with direct ophthalmoscope)
Management
Urgent referral to ophthalmologist
Retinal reattachment surgery successful in 90%
Visual results better if macula "on",
Emboli entering the retinal circulation may cause any of the following:
Transient loss of vision (lasting minutes) known as amaurosis fugax (retinal emboli may be
visible on fimdoscopy)
Sudden partial loss of vision due to branch retinal artery occlusion (BRAO)
Sudden total loss of vision due to central retinal artery occlusion (CRAO)
Central retinal artery occlusion
History
Examination
Management
Ocular massage( digital)
Diamox 500mg stat (to lower intraocular pressure)
Urgent referral to ophthalmologist
Anterior chamber paracentesis ( if < 24 hours, preferably within 6 hours)
ESR to exclude temporal arteritis as a possible cause
Systemic assessment for source of embolus (carotid doppler)
Long term aspirin or warfarin
History
Patients usually>50 yo
Strong association with hypertension and cardiovascular disease
Sudden painless bIur of vision
Examination
Vision varies with severity (from 6/6 to hand movements)
Afferent pupil defect if severe CRVO (HM vision)
Fundus : extensive retinal haemorrhages in all quadrants
retinal venous distension
optic disc swelling
Management
No immediate treatment of proven benefit
Assess cardiovascular risk factors
Retinal laser treatment for late complications (neovascular glaucoma)
Vitreous Haemorrhage
Aetiology
Proliferative diabetic retinopathy (new vessels present)
BRVO with new vessels
Retinal tears (tear through a retinal vessel)
History
Blurred vision with floaters
?diabetes(may be undiagnosed)
Examination
Vision: varies with severity of haemorrhage (6/6 to PL)
Pupils: NO RAPD (unless retina detached as well)
Fundus: reduced red reflex and difficult to see retinal detail
Management
Urgent referral to ophthalmologist
B scan ultrasound if no view of fundus
Vitrectomy surgery may be necessary
Atrophic ("dry") AMD causes gradual loss of vision due to loss of retinal photoreceptor cell
function in the macular region
Exudative ("wet") AMD is the major cause of blindness and may present with sudden loss of
vision due to haemorrhage from abnormal blood vessels beneath the macula
History
Recent onset blur with distortion (straight lines appear bent)
Examination
Vision: better than 6/60 in early stage, less than 6/60 in late stage
Pupils: NO RAPD (area of retina involved by the disease is not large enough to result in an
afferent pupil defect)
Field: central scotoma only
Fundi: Haemorrhage, drusen (pale age related deposits) and pigment changes in the macula
Management
Refer urgently to ophthalmologist if any recent change in vision
Fluorescein angiogram
Argon laser ablation of abnormal vessels in early stages
Low vision aids to assist with reading
Optic neuritis
Typically affects one eye of young women
Vision progressively dims over 48 hours (not truly "sudden")
Ache around eye at onset (worse with eye movement)
Reduced acuity and colour vision
A relative afferent pupil defect (RAPD) is present
Fundus may be normal (retrobulbar neuritis)
Recovery over 6 weeks, more rapid if IV methylprednisolone.
Strong association with MS (MRI Brain will help predict risk)
CVA
Unilateral CV A will cause homoymous field defects, but usually not loss of acuity
Bilateral occipital CV A may cause loss of acuity (plus visual field loss)
Migraine
The visual aura of classical migraine is a common cause of transient (usually lasting 5 to 20
minutes) visual disturbance in young healthy patients. Patients describe shimmering, flashing
lights and jagged lines and often have a history of previous similar episodes. The visual
disturbance may affect one or both eyes. The visual aura is often (but not always) followed by
headache.