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  • Pathophysiology of Pneumonia

    Загружено:

    Darlene Clarice V. Labaclado
    8K просмотров1 страница
    85 y.o, male Contributing / Precipitating Factors Bacteria Entry of microorganisms to respiratory system through inspiration / aspiration (staphylococcus and Gram-negative bacilli) Ineffective immune response resulting to overwhelming infection Penetrate the sterile lower respiratory tract Organism multiplies and releases damaging toxins Release of inflammatory mediators (IL-1, IL-6, TNF, GCSF)

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    85 y.o, male Contributing / Precipitating Factors Bacteria Entry of microorganisms to respiratory system through inspiration / aspiration (staphylococcus and Gram-negative bacilli) Ineffective immune response resulting to overwhelming infection Penetrate the sterile lower respiratory tract Organism multiplies and releases damaging toxins Release of inflammatory mediators (IL-1, IL-6, TNF, GCSF)

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    Attribution Non-Commercial (BY-NC)
    Скачайте в формате DOCX, PDF, TXT или читайте онлайн в Scribd
    Отметить как неприемлемый контент
    8K просмотров1 страница

    Pathophysiology of Pneumonia

    Загружено:

    Darlene Clarice V. Labaclado
    85 y.o, male Contributing / Precipitating Factors Bacteria Entry of microorganisms to respiratory system through inspiration / aspiration (staphylococcus and Gram-negative bacilli) Ineffective immune response resulting to overwhelming infection Penetrate the sterile lower respiratory tract Organism multiplies and releases damaging toxins Release of inflammatory mediators (IL-1, IL-6, TNF, GCSF)

    Авторское право:

    Attribution Non-Commercial (BY-NC)
    Скачайте в формате DOCX, PDF, TXT или читайте онлайн в Scribd
    Отметить как неприемлемый контент
    из 1

    PATHOPHYSIOLOGY OF PNEUMONIA PREDISPOSING Factors 85 y.

    o, male Contributing/Precipitating Factors Bacteria

    Entry of microorganisms to respiratory system through inspiration/aspiration (staphylococcus and Gram-negative bacilli) Activation of normal pulmonary defenses Ineffective immune response resulting to overwhelming infection

    Penetrate the sterile lower respiratory tract

    Organism multiplies and releases damaging toxins

    Release of inflammatory mediators (IL-1, IL-6, TNF, GCSF)

    Inflammation

    Airway constriction

    Increased mucus production

    Chills & fever

    Vasodilation & increase blood flow

    Hyperventilation Cough se RR

    crackles

    Alveolar capillary leakage

    Decreased lung compliance

    Results in: * se BS * tactile fremitus *dullness

    Inflamed & fluidfilled alveolar sac

    DYSPNEA

    Altered gas exchanged

    Hypoxemia

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