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Agroya
M.S.(Ortho) D.Ortho (Bom) Consulting Orthopaedic Surgeon Apex Hospital,Latur. Professor Department of Orthopaedics MIMSR Medical College,Latur.
LBP
Evolutionary Basis
The Low Back Pain is the penalty mankind is paying for his Erect Posture.
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Anatomy
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Abdominal Muscle
Back Extensors
Spine Spine
Figure 1
Figure 2
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Composition :
+
Mucopolysaccharides
Functions
:-
Holds the Vertebrae together and allows Movement between the bones
Seperates the vertebrae as part of a functional segmental unit acting in consert with facet joint By seperating the vertebrae helps in free passage of nerve roots through 18 intervertebral foramen
Clinical Approach
What is Backache for the patient is the headache for the Doctor ?
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Traumatic Causes
Inflammatory Causes
Metabolic Causes
- Osteoporosis - Osteomalacia
Miscellaneous Causes
- Functional Back Pain - Postural Back Pain - Protuberant Abdomen - Occupational bad posture
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History
Uncommon Congenital deformity Fall from height Infection ( Psoas abcess or tuberculosis) Carrying school bags
Elderly
Osteoporosis Osteomalacia Spinal Canal Stenosis Degenerative Spondylosis Malignancies Primary 22 Secondary
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PAIN :
History ( Contd.)
It is the gift of nature.
Definition : Pain is the subjective Emotional reaction. Which is felt at the site of injury but is recognised in Thalamus. The pattern of pain in the low back is an important clue to make perfect diagnosis. PAIN
Pain is an unpleasant sensory and emotional experience associated with potential or actual tissue damage
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History ( Contd.)
1. Localised 2. Diffuse Acute myofascitis Sprain Early disc degeneration Lumbar spondylosis Malignancies Infection Ankylosing Spondylosis 3. Referred pain Pain felt upto the knee joint never below it.
Types :
4. Radicular Pain - Pain felt upto the toes Disc prolapse SOL
PAIN Onset :
History ( Contd.)
1. Acute Trauma Significant or insignificant Disc prolapse Fractures Degenerative spondylosis Recurrent attack of disc prolapse
2. Chronic
Progression :
Max.at onset gradually subsides over the days. Disc prolapse Traumatic injury Remissions and exacerbations Disc prolapse Ankylosing spondylosis Constant pain aggrevated by activity Degenerative spondylosis Progressively increasing Infection SOL 27 Malignancies
PAIN Sleep
History ( Contd.)
Four types of pattern of pain
LBP till patient sleeps Sleep is very peaceful and 1. patient fresh in morning. Causes of this pattern are non cognizable e.g.
Malingaring Psychological Fatigue pain 2. No LBP before or during sleep but pain begins at getting up in the morning and wincess off as the day advances. e.g. Degenerative spondylosis Ankylosing spondylosis Rheumatoid affection
3. No pain before sleep - Patient sleeps well but awakened by pain in the middle of the sleep. This is a very geuinine pain caused by SOL and T.B. of spine (Night Cries) 4. Pain which does not allow to sleep at all. e.g. Malignancy 28 Infection
History ( Contd.)
myofascitis.
3. Sleeping prone if painful may be due to facetal arthropathy. 4. LBP caused while getting up from sitting position on the floor is very classical of spodylolysis or spondylolisthesis. Interestingly the same pain becomes less while patient gets up from the chair. 5. LBP only on standing further increased by walking is diagnostic of spinal canal stenosis. 6. Painful forward bending may be a feature of trauma,infection,acute disc prolapse,malignancy. 7. Coughing,sneezing and straining causing LBP is a diagnostic feature of acute disc prolapse again.
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History ( Contd.)
Neurological Symptoms
Common symptoms are.. Parasthesia Tingling Numbness Weakness Bowel and Bladder involvement
Anterior view
Posterior view
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History ( Contd.)
Appearance of LBP associated with Neurological symptoms in lower extremites after standing and walking for certain distance is called Neurogenic Claudication. It is the diagnostic feature of spinal canal stenosis.
Neurogenic Claudication
Psychological Complaints
Malingaring Hysteria
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Mechanical Posture Acute Disc Prolapse Spinal canal stenosis Spondylolysis Spondylolisthesis Inflammatory
Causes of LBP
Metabolic Neoplastic Meningeoma Visceral Abdominal Genito Urinary Gyanecological Functional Malingaring Psychological Ankylosing Spondylosis Osteoporosis Tuberculosis Rheumatoid Arthritis Pyogenic Arthritis
Degenerative spondy.
Fractures
Multiple Myeloma
Lymphoma Metastatic
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Posture
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Etiology
Disc Prolapse
Degenerative changes with advancing age make the disc increasingly susceptible to trauma such as.. Lifting
Twisting movements 34
C/o.parasthesias often describes as pins and needles corresponding to the dermatome of the affected nerve root. Numbness in the leg or foot and weakness of muscles e.g. S1 root weak push off L5 root weak toe extension L4 weak dorseflexion of the foot
In a massive disc prolapse patient may report with CAUDAEQUINA syndrome having paraplegia and bladder and bowel involvement. The chronic disc prolapse patient may report the gross WASTING of muscles.
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1. Posture
Examination
2. Limitation of Movements
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3. Tenderness
Examination
Diffuse but purcussion on spinous process may evoke the pain. 4. Straight leg raising Test
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Neurological Examination
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Investigations
1. X-Rays
2. Myelography
3. C.T.Scan 4. MRI Scan
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1. X-Rays
Investigations
Not very much diagnostic in acute disc prolapse but must be done to rule out other affections. Standard projections are A P view, Lat.view,oblique view and PBH prone. But in chronic disc prolapse one may see.. 1. Reduction in disc degenerative osteophytes.
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2. Myelography
Investigations
Due to adventage of MRI myelography is outdated. Disadvantage - Invasive Extra vasation of dye Indirect assessement CT Scan Good for hard structures. Poor visualiations of soft parts like disc. But can be useful if combined with myelography. C.T. Myelo
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4. MRI Scan
Investigations
More versatile investigation. It gives exact anatomical picture of cord,roots and disc in all three planes. i.e. Saggital Coronal Axial
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30/12/2004
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Treatment
Every disc is not for removal and at the same time no culprit disc should be spared. This decision is based on.. 1. Clinical co-relation on MRI findings Perfect clinical co-relation 2. Response to conservative
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1. Rest
Conservative Treatment
- It reduces intradiscal pressure. Relaxes the nerve roots. Prevents the mechanical irritation of the nerve roots.
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Conservative Treatment
2. Lumbar Traction Controvertial ? Therotically it distracts the disc space so prolapse disc is reduced. 3. SWD and Massage 4. NSAIDS with Muscle Relaxtant 5. Sedatives 6. Curtailment of curtain activities
Activity And Percentage Increasing in Disc Pressure at L3 Coughing or Straining 05 to 35% Laughing 40 to 50% Walking 15% Side bending 25% Small jumps 40% Bending forward 150% Rotation 20% Lifting a 20 Kg weight with the back Straight and knees bent 73% Lifting a 20 Kg weight with the back Bent and knees straight 169%
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Conservative Treatment
7. Back Supports May help during mobilisation. Should be adviced strictly for short term use because of 1. Muscle atrophy further degeneration 2. Psychological dependency 8. Physiotherapy -The purpose of physiotherapy is to.. 1. Unload the disc pressure. 2. Even distribution of load of day today activity. 3. To stretch the nerve root to prevent adhesion. This can be achieved by strengthening Abdominal Muscles Back extensors Hip extensors Hip flexors
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Operative Treatment
The aim of the surgery is to remove the disc compressing the root and the theca. But the question is.
b) Without neurological deficit but LBP is refractory to fair trial of conservative treatment.
2. WHEN 3. HOW
? ?
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Extremely common condition It is caused by reduction in the space of spinal canal. The diagnosis is made only by history. Clinical examination of little value. Clinical Feature : Common in males Above the age of 40 No LBP at rest or sitting Neurogenic claudication As the patient stands and start walking LBP begins. LBP keeps on increasing as the distance increases. LBP may be associated with parasthesias and both LE. At critical point patient cannot walk and sit down. After few minutes patient is fresh to cover the same distance and the cycle repeats. Patient prefers to walk bending forward and flex knee and hip to get rid of symptoms. As the disease advances the claudication distance become less and less. This is the most diagnostic rather only diagnostic clinical information. 53
Clinical Examination :
No pathological findings in clinical examination. One must ruleout PVD.
Wall test
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Treatment :
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Spondylolisthesis
It is the condition caused by forward displacement of body of the vertebrae due to the defect in the posterior element. Fairly common cause of LBP many times a symptomatic accidently detected by on X-rays.
Clinical Feature
Common in female with 30 to 40 age Posture Hyperlordotic back Pain Low grade Episodic Worse when standing and walking relieved by sitting and lying down. Pain is restricted to low back, buttocks and thigh never radiates down. Hyperextension of Lower back will the pain. Pain is more while getting up sitting position on the floor. Same pain is very less while getting up from the chair. It is because of hamstring tightness and spasm. Neurological symptoms are usually absent in the initial phase.
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Investigation
X-Rays
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Treatment
Bed rest Lumbar Traction NSAIDS with MR
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Trauma
Trauma can cause two types of backache.. a) Acute Backache It is due to injury to bony elements of vertebrae, ligaments or the muscles.
Clinical Features :
H/o.injury Pain moderate to severe acute onset relived by rest and passage of time,well localised,severe tenderness at the sight of injury,haematoma in the paraspinal region +. Movements are grossly restricted. Neurological deficit may or may not be present.
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Trauma(Contd.)
a) Acute Backache
Investigations : X-rays may show various degrees of fractures of the vertebrae. CT Scan is indicated only if fracture is not detected on x-rays. MRI is indicated only if neurological deficit is there.
Treatment : Enforced bed rest till pain subsides. NSAIDS with MR Mobilisation with back support. In absence of neurological deficit surgery is indicated only to stabilise the spine if the injury is unstable.
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Trauma
b) Chronic Backache : The cause of LBP is due to ultered biomechanics of spine due to malalignment of vertebral column. It accelarates the degenerative process. Clinical Feature : H/o.old injury Pain intermitant mild to moderate diffuse more on activity and more after activity(Fatigue pain),Subsides after rest. Investigations : X-Rays may show kyphotic deformative old,healed compression fracture of the body,degenerative changes away from the site of injury. MRI is indicated if associated with neurological complaints. Treatment : Curtailment of activities like forward bending,lifting heavy weight and travelling . NSAIDS with MR SOS Back supports SOS 61 Back extension excercises
Degenerative Spondylosis
It is a condition caused by degeneration of cartilage with advancing age. Clinical Feature : Elderly person If young some anticedant problem of the spine like Osteopania,postural deformity etc. Pain mild to moderate intermitant diffuse relieved by rest increased by activities. Some times acute severe due to unaccustumed activity. Stiffness May or may not be associated with neurological symptoms of disc prolapse or spinal canal stenosis. Investigations : X-Rays may show deformity( reduction in disc space, affection of facet) MRI is indicated if neurological symptoms are present. Treatment : Bed rest Hot foamentation Massage SOS NSAIDS PT to improve back extensors,abdomen and gluteous 62 muscles.
Ankylosing Spondylosis
Its a condition is characterised by progressive inflammation of joints of the vertebral column leading to ankylosis(Fusion).
Clinical Feature :
Fairly common hereditary Young adult LBP localised to SIJ Morning stiffness Severe Spasm ( Bamboo Spine ) Lordosis reduced Tenderness at SIJ Limitation of movements Chest expansion reduced X-rays may show patchy osteoporosis and increased distance with subchondral Erosion at SIJ. Late cases may show fusion. Lumbar spine fusion of the vertebral bodies may be seen. Symptomatic 63 Physiotherapy
Investigations :
Treatment :
Tuberculosis
It is a condition caused by acid fast bacilia( Mycobactarium Tuberculosis). Common but steady decline in the prevalance.
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Tuberculosis(Contd.)
Clinical Features : Varies with the stage of the disease i.e. 1) Inflammatory stage 2) Destructive stage Symptoms : No age bar Constitutional symptoms present LBP, Dull ache,Diffuse,Referred or Redicular associated with stiffness due to spasm,Night cries(Children),Sleep disturbances. Movements very painful and restricted. Swelling in paraspinal gluteal,groin region(Cold abcess) Weakness in lower limb with parasthesias(Impending cord compression) Kyphotic deformity(Vertebral collapse) Signs : Short steps gait to avoid jerkin the spine takes time to lie down. Loss of lumbar lordosis. Soft fluctuant in paraspinal gluteal region. Localised tenderness. Forward flexion restricted. Neurological examination may vary from normal paratic plagic
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Tuberculosis(Contd.)
Investigations : Blood Hb X-Rays : ,Lymphocytosis ESR Positive PCR, HIV Test Initially may appear normal. Severe focal osteoporosis of lumbar vertebrae is the diagnostic early feature. Loss of disc space Variable amount of erosion and destruction of vertebral body. Complete anterior collapse. In AP view PSOAS shadow may be prominent due to PSOAS abcess. Indicated when x-rays normal despite strong clinical suspecion. Shows very acurately destruction of the body,disc degeneration,cold abcess and cord compression in an anatomical fashion.
CT Scan
MRI
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24/11/2004
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Tuberculosis(Contd.)
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Pts Name :- Shri. Karpude Mohammad 26/01/2001
Tuberculosis(Contd.)
69 11/12/2000
Tuberculosis(Contd.)
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22/12/2000
Tuberculosis(Contd.)
Treatment : Urgent measures are required to prevent neurological insult. With the adventage of superb chemotherapy agents 90% cases can be treated without surgery. Conservative Treatment : Hospitaliasation Absolute bed rest Lumbar traction Chemotherapy Streptomycin INH Rifampicin Ethambutol Pyrazinamide Mobilisation after 4 to 6 weeks with back support. Surgical Treatment : Indicated when progression continues despite AKT. Surgical objectives are.. Decompression Excision of infected tissue Reduce the spinal deformities 71 Provide stability by spinal fusion
Osteoporosis
It is a commonest metabolic disorder. Definitions :
Reduction in bone mass per unit of its volume. It occures when the rate of bone resorbtion exceeds the rate of bone formation. Causes : A) B) C) D) E) Senility Post Menopausal Post Immobilisation Endocrinal Cushing Syndrome & Hyperparathyroidism Drug Induced Steroid,Anti epileptic,Antacid
Clinical Features : Usually asymptomatic Kyphotic posture Some times generalised bony pain. Acute pain may occur after a trivial trauma leading to fractures.Commonest sites is dorsal lumbar region,neck femur,colles fracture etc.
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Osteoporosis(Contd.)
Investigations : X-Rays Ground glass apperance,Wedge vertebrae,Cod fish vertebrae etc.
BMD
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Osteoporosis(Contd.)
Treatment :Since Etiology is multifactoral treatment is difficult. High protein Diet Calcium Supplementation Androgens Estrogens Vitamin D3 Exercises Caution (Osteoporosis is not a disease rather a disorder. There may be an underlying major or minor condition to cause it.)
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Neoplastic Causes
LBP is a very common presentation of tumours of vertebral column and spinal cord. Multiple Myeloma :It is a fairly common malignant tumor of vertebral column. Clinical Features : Age : 40 to 60 years Acute severe LBP may be localised to low back may rediate down to extremetes. Pain is unremitting progressive and awakens the patient in the night. Investigations : X-Rays Disproportionately severe osteoporosis. Destruction and the collapse of the vertebrae. Lytic cavities Punch out areas in the skull and other bones MRI May show cord compression Blood - Anaemia,ESR very high Low serum proteins with reversal of A-G ratio,Hypercalcimia,Positive serum electro phoresis,Positive bone marrow aspiration for abnormal plasma cells. Urine : BJ proteins positive 75 Treatment : Chemotherapy
Neoplastic Causes(Contd.)
Metastasis :Vertebral column is a very preferred site for secondary deposits. The common primaries are Ca thyroid,Ca bronchus,Ca prostate,Ca breast etc. These tumours often present as LBP or with neurological deficit. These cannot be diagnose on clinical ground but are detected after investigations.
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Chame Maruti 5/2/2002 Mrs.Surve Suman 1/7/2000
Neoplastic Causes(Contd.)
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Pts Name :Bhalekar Satyabhama 25/11/1999
Neoplastic Causes(Contd.)
Tumours of the Spinal Canal :-
Alongwith LBP these tumours often cause neurological symptoms in the lower extremity. Again it is very difficult to diagnose this clinically and are revealed only after investigations. The common tumours are Meningioma, Neurofibroma ,Epindiomoma ,Schwanoma etc..
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Visceral Causes
These are not the true causes of LBP but one is confused because the pain is pointed towards the back. a) Abdominal Causes Pancreatitis Cholaecystitis Renal colic b)Ca Cervix Very common cause of LBP in females Unremitting LBP Leucorrhoea Abnormal PV bleeding Foul smelling discharge etc gives enough information to suspect
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Functional Causes
Malingaring Psychological
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