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A.Rodin
Etiology.
Exogenous factors:
1. Destruction of water-lipid
coverage under the influence of
skin hygiene insufficiency
(cement pollution, gas-oil
materials, coal, etc.).
2. Skin traumas (as well as
microtraumas).
3.
4.
Endogenous factors:
1. Endocrine system pathology:
diabetes , thyroid gland insufficiency,
hypophysis and epinephros system
insufficiency, and sex gland
insufficiency.
2. Vegetative neuroses accompanied by
vascular tonicity malfunction as well
as sweat gland malfunction.
3.
4.
Classification.
Staphylodermia
Vesiculopustulosis.
Pseudofurunculosis.
(multiple abscesses).
Disintegrity of infiltrate is
accompanied with skin node
thinning than abscesses eruption
takes place discharging yellowgreen pus. Commonly, weak
children suffer this disease
(artificial feeding, delivery
trauma etc.).
Epidemic pemphigus in
infants (pyococcic
pemphigoid).
Exfoliate Ritters
dermatitis.
Ostiofolliculitis.
Folliculitis.
Furuncle.
Carbuncle.
Hydradenitis.
Staphyllogenic purulent
inflammation of apocrine sweat
glands and as a result the
localization is in the armpit, in the
area of genitalia, perianus area.
Clinical manifistation are in
hyperemic abscesses (the size of a
pea) and central fluctuation. The
abscess extracts with a significant
pus amount. There are no necrotic
cores. The disease may prolong
turning into a chronic-recurrent
form.
Staphylodermia treatment.
Osteofolliculitis, superficial
folliculitis can be treated
topically pustule opening,
painting the focuses with anilin
paintings, prophylaxis around
the area of focuses 2%
salicylic, boric, camphor acid.
Carbuncle.
Streptodermia.
Impetigo or Streptodermia
characterized by the surface
involvement of the skin, folders.
Follicles and sweat glands are
not involved. Females and
children suffer this disease
commonly.
2. Cleft-like impetigo.
This form is characterized with a
quickly erupted form of
phlyctena localized in the
peripheries of the mouth, at the
base of the nose wings, eye
fissures. Due to the maceration
clef-like fissures form, the crusts
reject very quickly the disease is
accompanied with painful cracks.
3. Intertrigo
streptodermia.
This form is usually developed in
overweighed people, diabetic
persons and pastosis children. The
phlyctena occur on the surface of
large folders, become fused, after
the eruptions there are huge
erosive moist surfaces of pink
bright colour. This kind of
dermatosis has the form of pruritis.
Chronic pyoderma
treatment
immunization.
1. Absorbed staphylococcic
anatoxin
2. Native staphylococcic anatoxine
3. Staphylococcic antiphagin
Passive immunization
Autohemotherapy; blood
transfusion
Pyrotherapy (pyrogenal)
Ultraviolet and laser radiation of
the blood, ozonetherapy
Immune modulators (T-activin,
pentoxil, amixine, thymolin etc).
Scabies.
is a parasitary disease,
relating to dermatosoonoses (the
penetration of a live parasite into
a living organism). The causative
agent is a scabby tick, the tick
drills an entrance of 5-10 mm at
length under the horny layer of
epidermis and saves there its
eggs. Contamination usually takes
from 3-14 days to 1.5 months by
means of hand-shaking, coitus ,
articles of the dress, toys, etc as
well as cat.
Clinical characteristics
Scabies tick drills entrances in
the thin layers of the skin
(interfinger spaces, wrists,
genitalia., etc. in distinction
from the adults the infants may
have the involvements of face,
palms, soles).
Treatment.
PEDICULOSIS.
Head pediculosis.
Pubic pediculosis
Underwear pediculosis
Treatment of the
pediculosis