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INTRODUCTION
SD chronic superficial
inflammatory
predilection for the
nasolabial creases,
axillae, umbilicus,
EPIDEMIOLOGI
ETIOLOGY
E/ of seborrheic dermatitis unknown.
Potensial etiologies of seborrheic dermatitis :
stress, weather extremes, oily skin, infrequent
shampoos, obesity, strokes, HIV
E/ Pityrosporum ovale
PATHOGENESIS
Over secretion in sebaceous glands
predisposition factor.
P ovale in the seborrheic area can
sensitize an individual resulting in spesific IgG
response, can activated complement by both
pathways.
CLINICAL MANIFESTATION
The lesions to be dull or yellowish red in
colour had covered with greasy scales.
They commonly marginate in hairy skin and
involve the scalp, face, sternal and
interscapular regions and the flexures.
Adult
Scalp
Dandruff
Inflammatory may extend onto non hairy
areas (e.g. postauricular)
Face (may include blepharitis and
conjunctivitis)
Trunk
Petaloid
Pityriasiform
Flexural
Eczematous plaques
Follicular
Generalized (may be erythroderma)
HISTOPATHOLOGY
Histological
examination
subacute
dermatitis reaction with spongiosis and even
vesicle formation.
There is also considerable acanthosis, with
elongation and the clubbing of the rete ridges,
and there may be some thinning of the supra
papillary part of the dermis.
DIAGNOSIS
The diagnosis of SD based on assessment
of symptoms, accompanied by consideration
of medical history, discovery clinical
manifestation and histopathology.
DIFFERENTIAL DIAGNOSIS
The differential diagnosis of seborrheic
dermatitis includes:
* psoriasis
* pityriasis rosea
* tinea
* atopic dermatitis
THERAPY
Infants
Scalp : - salicylic acid 3-5%
- hydrocortison 1%
- imidazoles
-warm olive oil compress
- mild baby shampoos
- proper skin care with emolients,
creams and soft pasts.
Intertriginous areas:
- Corticosteroid mild potency
- imidazoles preparations (e.g.ketokonazole
2% in soft pastes, creams or lotion)
Adults:
Scalp:
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