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http://www.who.int/mediacentre/news/releases/2006/pr32/es/
PASIVOS AMBIENTALES
MINEROS
LOS CONTAMINANTES
NO SON ESTTICOS
PROXIMAL / DISTAL
LOS
CONTAMINANTES
MIGRAN
FUENTE DE
CONTAMINACI
N
PUNTOS DE
EXPOSICIN
TOXICIDAD
DISRUPCIN
APERTURA
MATRICES
VAS DE
EXPOSICIN
EXPOSICIN
AGUDA
CRNICA
2.66mol/L=55g/dL(Ao1950-1970)
2.17mol/L=45g/dL(Ao1970-1974)
1,45mol/L=30g/dL(Ao1975)
0.48mol/L=10g/dL(Ao1991/Umbralalao2010)
0.24mol/L=5g/dL(CDC)
Noexisteningnnivelseguroparaelplomo
https://www.aap.org/en-us/aap-voices/Pages/Lead-Poisoning-Before-and-Beyond-Flint.aspx
Juberg, D., Kleiman,C, Kwon, S.,.(1997): Lead and Human HealthAmerican Council on Science and Health. 21-23
CDC. Biomonitoring Summary. Lead CAS No. 7439-92-1. National Biomonitoring Program. Page last updated: Wednesday December 04 2013.
https://www.cdc.gov/biomonitoring/Lead_BiomonitoringSummary.html
CDC. Lead: What Do Parents Need to Know to Protect Their Children? /Update on Blood Lead Levels in Children. USA-CDC
update 2016. https://www.cdc.gov/nceh/lead/acclpp/blood_lead_levels.htm
DipeshNavsaria. Childhood Lead Toxicity Journal Scan/ReviewJune 30, 2016.
http://www.practiceupdate.com/content/childhood-lead-toxicity/40867/37/6/1?trendmd-shared=1
Kent Bennett , Jennifer Lowry, Nicholas NewmanLead poisoning: Whats new about an old problem?. Contemporary
Pediatrics. Agency for Toxic Substances and Disease Registry. 2015.
http://contemporarypediatrics.modernmedicine.com/contemporary-pediatrics/news/lead-poisoning-what-s-new-about-oldproblem-1
American Academy of Pediatrics. AAP Commends CDC for Recognizing That for Children, There is No Safe Level of
Lead Exposure. 06-05-12.
https://www.aap.org/en-us/about-the-aap/aap-press-room/pages/AAP-Statement-CDC-Revised-Lead-ExposureGuidelines.aspx
DHHS. 1995. Report to Congress on workers' home contamination study conducted under the workers.family protection act (29 U.S.C. 671a). Cincinnati, OH: U.S. Department of Health and Human Services, Public Health Service, Centers for Disease Control and Prevention, National
Institute for Occupational Safety and Health. September 1995. Pub no. 95-123. PB96192000.
NTP. 2005. Report on carcinogens. 11 ed. Research Triangle Park, NC: U.S. Department of Health and Human Services, Public Health Service, National Toxicology Program. http://ntpserver.niehs.nih.gov/ntp/roc/toc11.html. February 15, 2004.
IRIS. 2007. Arsenic. Integrated Risk Information System. Washington, DC: U.S. Environmental Protection Agency. http://www.epa.gov/iris/subst/index.html. August 28, 2007
IARC. 2004. Overall evaluations of carcinogenicity to humans: As evaluated in IARC Monographs volumes 1-82 (at total of 900 agents, mixtures and exposures). Lyon, France: International Agency for Research on Cancer. http://www-cie.iarc.fr/monoeval/crthall.html. March 08, 2006.
WHO. World Health Organization. IPCS International Programme on Chemical Safety. Environmental Health Criteria 18: Arsenic. Geneva, 1981. 74p.
WHO. World Health Organization. IPCS International Programme on Chemical Safety. Environmental Health Criteria 224: Arsenic and arsenic compounds. 2.ed. Geneva, 2001. 521p.
WHO. World Health Organization. IPCS International Programme on Chemical Safety. Environmental Health Criteria 155: Biomarkers and risk assessment: concepts and principles. Geneva, 1993. 82p.
WHO. World Health Organization. United Nations Synthesis Report on Arsenic in Drinking Water: Exposure and health effects. Geneva, 2001. 84p.
WHO. World Health Organization. Environmental hazards kill 3 million children aged under 5 every year.
Agency for Toxic Substances and Disease Registry. Toxicological profile for arsenic, update. Atlanta: US Department of Health and Human Services; 2000 September.
Integrated Risk Information System (IRIS) U.S. Environmental Protection Agency National Center for Environmental Assessment EPA Integrated Risk Information System (IRIS).Chemical Assessment Summary IRIS: Arsenic, inorganic CASRN 7440-38-2: Cancer Assessment
https://cfpub.epa.gov/ncea/iris2/chemicalLanding.cfm?substance_nmbr=278
Enterline, P.E. and G.M. Marsh. 1982. Cancer among workers exposed to arsenic and other substances in a copper smelter. Am. J. Epidemiol. 116(6): 895-911.
Lee, Te-Chang, J. Ko and K.Y. Jan. 1989. Differential cytotoxicity of sodium arsenite in human fibroblasts and chinese hamster ovary cells. Toxicology. 56(3): 289-300.
Lee-Feldstein, A. 1983. Arsenic and respiratory cancer in man: Follow-up of an occupational study. In: Arsenic: Industrial, Biomedical, and Environmental Perspectives, W. Lederer and R. Fensterheim, Ed. Van Nostrand Reinhold, New York.
Axelson, O., E. Dahlgren, C.D. Jansson and S.O. Rehnlund. 1978. Arsenic exposure and mortality: A case referent study from a Swedish copper smelter. Br. J. Ind. Med. 35: 8-15.
Tokudome, S. and M. Kuratsune. 1976. A cohort study on mortality from cancer and other causes among workers at a metal refinery. Int. J. Cancer. 17: 310-317
Chen, C-J., Y-C. Chuang, T-M. Lin and H-Y. Wu. 1985. Malignant neoplasms among residents of a Blackfoot disease-endemic area in Taiwan: High-arsenic artesian well water and cancers. Cancer Res. 45: 5895-5899
Chen, C-J., Y-C. Chuang, S-L. You, T-M. Lin and H-Y. Wu. 1986. A retrospective study on malignant neoplasms of bladder, lung, and liver in blackfoot disease endemic area in Taiwan. Br. J. Cancer. 53: 399-405.
Chen, C-J. and C-J. Wang. 1990. Ecological correlation between arsenic level in well water and age-adjusted mortality from malignant neoplasms. Cancer Res. 50(17): 5470-5474.
Chen, C-J., CW. Chen, M-M. Wu and T-L. Kuo. 1992. Cancer potential in liver, lung bladder and kidney due to ingested inorganic arsenic in drinking water. Br. J. Cancer. 66(5): 888-892.
Chen, C-J., M-M. Wu, S-S. Lee, J-D. Wang, S-H. Cheng and H-Y. Wu. 1988. Atherogenicity and carcinogenicity of high-arsenic artesian well water. Multiple risk factors and related malignant neoplasms of Blackfoot disease. Arteriosclerosis. 8(5): 452-460.
YANACANCHA 2006
MINSA -INS
NIOS AFECTADOS
CON PLOMO
QUIULACOCHA 2005
MINSA-INS
NIOS AFECTADOS CON
PLOMO
CERRO DE PASCO
2006
MINSA -DIGESA
NIOS AFECTADOS
CON PLOMO
Sexo
FACTORES ASOCIADOS
EN NIOS Y MUJERES GESTANTES DE LAS
POBLACIONES
QUIULACOCHA Y CHAMPAMARCA CERRO DE
PASCO
Nivel de Plomo
Edad
SETIEMBRE
2005
TOTAL
< 10
10 a 14.99
15 a 19.99
20 a 44.99
1-2
3-4
0.0
1.8
3.6
0.0
0.0
0.9
3.6
2.7
5-6
0.9
7.2
2.7
6.3
7-8
2.7
6.3
3.6
1.8
9-10
2.7
3.6
1.8
0.9
59
88.12/100%
SubTotal
6.3
22
19.8
17
15.3
13
11.7
1-2
0.0
1.8
3.6
2.7
0.9
1.8
7.2
2.7
3-4
5-6
0.9
0.9
4.5
2.7
7-8
0.9
3.6
3.6
2.7
9-10
1.8
4.5
0.0
0.0
4.5
14
12.6
21
18.9
12
10.8
52
79.66/100%
12
10.8
36
32.4
38
34.2
25
22.5
111 /109
100.0% /
98.2%
SubTotal
TOTAL
Mas del 50% de nios evaluados presentan valores > a g/dL plomo por cada decilitro de sangre
LA DESINFORMACIN Y LA
POCA TRANSPARENCIA NUNCA
HAN SIDO AMIGOS DEAutoridades
LA PROTECCION Y ocultaron qu
e poblacin p
PREVENCIN EN FAVOR resenta
DE LAplomo
VIDA Y LA SALUD en la sangre
http://www.radiomaranon.org.pe/index.php/actualidad-regional/cajamarca/793-autoridades-ocultaron-que-poblacion-presenta-plomoen-la-sangre-denuncia-alcalde-hualgayoc
, denuncia Al
calde Hualgay
http://larepublica.pe/26-12-2014/ocultaron-a-comuneros-de-espinar-que-tienen-11-metales-
MUCHAS GRACIAS