Conduction Disorder

Conduction disorder
Conduction
disorders:
Obstruction or
slowing the
electrical
conduction
pathways normal
 Blok Atrioventikular
(blok av)

Blok AV Blok AV Blok AV

degree 1 degree 2 degree 3
(blok total /
komplit)

Blok Mobitz Blok Mobitz

tipe 1 tipe 2
(Wenckebac
h)
 AV BLOCK DEGREE 1
Characterized by prolonged slowing
conduction in the AV node or bundle of His
PR interval extends> 0.2 seconds
(keywords!)
Atrial impulse is still able to pass through
the AV node to activate the ventricles, AV
block degree one is not really a block, only
slowing conduction.
Can still be found in the normal heart, does
not need to be treated.
AV BLOCK DEGREE 1
AV BLOCK DEGREE 2 type 1
lock occurs in the AV node
PR interval becomes progressively longer until a P
wave is not followed by a QRS complex. (QRS
missing)
After missing pulse (QRS), a series of regularly
repeated continuously.
This disorder usually causes no symptoms, if the
ratio is very low koduksi can cause bradycardia and
decreased cardiac output.
The commonest cause is coronary heart disease,
inferior myocardial infarction, aortic valve disease.
AV BLOCK DEGREE 2 type 1
(mobitz 1 / wenckebach)
 Second-degree av block type 2
(mobits type 2)
Conduction phenomena in the form of all-or-
none. Two or more normal pulse having a
normal PR interval, then the P wave is not
followed by QRS (QRS missing).
Their pulse disappeared without a
progressive lengthening of the PR interval
The cycle then repeats itself, with a ratio of 3:
2, 4: 3 and so on
Irregular ventricular rhythm
Occurs in acute anterior myocardial infarction
Second-degree av block type
2 (Mobitz tipe II)
 AV BLOCK TOTAL
There is no atrial impulse that got through to
activate the ventricles.
Ventricular responded to this situation by giving
rhythm passes (entricular escape rhythm)
inadequate (atrial and ventricular depolarization
separately from one another, unrelated). Atria and
ventricles remain contracted yet with its own
intrinsic frequency, 60-100 x / min for the atrium
and 30-45 x / min ntuk ventricles.
P wave is normal, regular rhythm.
Slower frequency than the frequency of ventricular
or atrial sinus.
AV block Total ( degree 3)
Bundle branch block

Blok hantaran
Right bundle Left bundle fasikulus
branch block branch
(RBBB) block(LBBB)

Left anterior Left psoterior

fasicular block fasicular block
(LAFB/LAHB) (LPFB/LPHB)
 RBBB
Conduction pass right bundle diobstruksi. As a result,
late right ventricular depolarization occurs, and does
not begin until the left ventricle is almost completely
depolarized.
Widened QRS complex exceeds 0.12 seconds
RSR 'in V1 and V2 (such as rabbit ears) that
accompanied the ST segment depression and T-wave
inversion
S wave width> = 0.04 sec and blunt (slurred) in V5,
V6, I and aVL.
RBBB pattern is common in patients with mitral
stenosis, atrial septal defect, IMA.
RBBB
 LBBB
Widened QRS complex exceeds 0.12
seconds
Notched R wave width or slightly
sloping
initially in V5, V6, leads I and aVL,
accompanied by ST segment
depression and T wave inversion.
There were no Q waves in V5, V6,
and lead I
Can be found to the left axis
LBBB
CASE STEMI WITH AV BLOCK