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Etiology

Usually functional

Organic in
- RHD ( with TS)
- IE ( drug addicts)
- congenital
- Ebsteins anamoly
- carcinoid syndrome
- trauma
In presence of TS , TR is organic
TR alone is mostly functional
Dilatation of RV / tricuspid valve ring
seen in CCF/RHF/PH/ cor pulmonale
Infarction of papillary muscles
Tricuspid valve prolapse
H/o weakness, fatigue (decrease CO)

Throbbing pulsations neck (increase


JVP)

Increased JVP- prominent v waves

Features of RHF & pulsatile liver


RV apex beat (hyperdynamic) & RV S3

PSM high pitched at TA, increase on


inspiration

Usually has atrial fibrillation


Chest X ray: cardiomegaly (RA & RV type)

ECG : RVH, RAD, RBBB

ECHO : RV dilatation
tricuspid valve abnormalities
tricuspid valve prolapse
cardiac function / EF

Doppler ECHO : regurgitation vol/ severity


Isolated TR without PH well tolerated

TR secondary to PH & MS, MR


treat the cause MS, MR, etc

Treat CCF efficiently

Surgery (rare) tricuspid annuloplasty


- valve replacement

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