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Dr.Suhaemi,SpPD,Finasim
PEMBULUH DARAH KORONER
RCA
LM
LAD LCx
The Normal ECG
Normal ECG
• Standardization – 10 mm (2 boxes) = 1 mV
• Double and half standardization if required
• Sinus Rhythm – Each P followed by QRS, R-R constant
• P waves – always examine for in L2, V1, L1
• QRS positive in L1, L2, L3, aVF and aVL. – Neg in aVR
• QRS is < 0.08 narrow, Q in V5, V6 < 0.04, < 3 mm
• R wave progression from V1 to V6, QT interval < 0.4
• Axis normal – L1, L3, and aVF all will be positive
• ST Isoelectric, T waves ↑, Normal T↓ in aVR,V1, V2
GELOMBANG R DAN S DI LEAD PERIKORDIAL
V1 V2 V3 V4 V5 V6
Nomenklatur Kompleks QRS
NORMAL
Tunika media
Tunika Intima
Barrier
Minor
Plaque Non- Occlusive
Disruption Occlusive Thrombus
Thrombus
Non-Vulnerable Vulnerable
Atherosclerotic Atherosclerotic
Plaque Plaque Myocardial
Infarction or
Asymptomatic Sudden
Unstable Cardiac
Angina or Non- Death
Q-MI
Major Occlusive
Plaque Thrombus
Disruption
11
ATHEROGENESIS
LCX
LMS
LAD
pangkal
Platelet
rupture
Platelet
Adhesion
Platelet
Sequence of events Activation
• Plaque Rupture Platelet
• Platelet Adhesion Aggregation
• Platelet Activation
• Platelet Aggregation Thrombotic
• Thrombotic Occlusion Anti-platelet drugs Occlusion
19
Patofisiologi SKA
Erosi atau ruptur plak
Angina Pektoris tak Stabil (APTS) Infark Miokard dgnNon ST Infark Miokard
Elevasi dgn ST elevasi
Circulation 1998;98:2219-22
ER patient care
• Initial general treatment (memory aid:
“MONA” greets all patients
– Morphine, 2-4 mg repeated q 5-10 min
– Oxygen, 4 L/min; continue if SaO2 < 90%
– NTG, SL or spray, followed by IV for persistent or
recurrent discomfort
– Aspirin, 160 to 325 mg (chew and swallow)
Myocardial Ischemia
• Myocardial ischemia results when the heart’s demand for oxygen exceeds its
supply from the coronary circulation. Ischemia can resolve by reducing the
oxygen needs of the heart or increasing blood flow by dilating the coronary
arteries with medication such as nitroglycerin.
• An inverted T wave will be present in the leads facing the affected area of the
ventricle if ischemia is present through the full thickness of the myocardium
28
Myocardial Injury
• Myocardial injured cells do not function normally, affecting both
muscle contraction and the conduction of electrical impulses
33
Identify the ECG Complex
4
5
1
8
2
34
Putting it All Together
Segmen ST
Diukur dari akhir QRS s/d awal gel T
Normal : Isoelektris
T-waves
TRANSMURAL Injury ST
Elevation
49
Ischemia, Injury & Infarction
50
ST Segment Changes: Identifying MI Mimics
Lateral
Anterior
Lateral
Inferior
Blood Supply of Heart
RCA
LCX
LAD
RCA
LCA
53
Blood Supply of Heart
71
NSTEMI
73
Very Striking
74
What are these ECGs
75
STEMI and QWMI
77
What is
striking ?
78
Guess How Old is this MI !
79
Question 1
• In Conclusion
– is the patient having a MI?
– a variety of conditions can
mimic infarction
ST segment changes
ST Segment Changes: Identifying MI Mimics
87
ST depression
Lateral Wall Ischemia
90
Holter & TMT in CAD
91
Evolution of Acute MI
92
Serial ECG changes of MI
93
Normal Q waves
101
Muscle Tremor
SIRKULASI SISTEMIK
AFTERLOAD
INOTROPIK NEGATIF
B-BLOCKER Ca ANTAGONIST
VERAPAMIL, DILTIAZEM
B-BLOCKER
NITRAT
NITRAT
DILATASI Ca ANTAGONIST ARTERIOL REST VESSEL
VENOUS RETURN
H.Opie 2001;34-35
Which BP Drug to Choose ?
1. HT + DM ACEi, ARB
2. HT + IHD ACEi, Perindopril + BB (Meto, Carva)
3. HT + MRD ACEi + / or Methyl dopa (MD)
4. HT + CHF ARB, ACEi, Diuretics, No CCB
5. HT + Pregnancy MD or CCB (Amlo) No ACEi
6. HT + Asthma, COPD No beta blockers, Alpha blockers OK
7. HT + Tachycardia No CCBs, Give BB
8. HT + DyslipidemiaNo Diuretics- give ACEi, ARB, CCB
9. HT in elderly, ISH Indapamide, Diuretics, CCB
109
THIS IS NOT THE END
Thank YOU
110