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INSIDENCY INCREASE
PROBLEMS OF MULTIPLE PREGNANCIES:
FETAL COMPLICATION
-Prematurity
-Deceased survival (fetal death rate 4x greater,
the likelihood not surviving the first year 7xgreater)
-IUGR (14-25%) and 25% require NICU admission
-Risk of Cerebral palsy 4x greater for twins, 17x
greater for triplet
MATERNAL COMPLICATION
-Preeclampsia and diabetes are 2-3 times more
common.
Factors That Influence Twinning
Race
Maternal Age
Parity
Nutritional Factors
Herediter
Pituitary Gonadotropin
MONOZYGOTIK (IDENTICAL)
1/3 of MULTIPLE PREGNANCIES
One egg
Based on classic report by BERNIRSHKE AND KIM,
there are 3 types of placentation:
1. DICHORIONIC-DIAMNIOTIC(1/3,on day 0-3)
2. MONOCHORIONIC-DIAMNIOTIC(2/3,on day 4-8)
3. MONOCHORIONIC-MONOAMNIOTIC(<1%,
occuring on day 9-12)
4. CONJOINED TWINS
DIZYGOTIK/POLIZYGOTIK
2/3 of MULTIPEL PREGNANCY
FERTILISAZION 2 OR MORE OOSIT BY 2 SPERM IN
THE SAME CYCLE
THE MOST DRAMATIC INCREASES: ART
THE TYPE OF PLACENTATION IS THE MOST
IMPORTANT PREDICTOR
1/90 PREGNANCIES
ANOTHER TYPE: SUPERFETACY
.
DIAGNOSIS OF TWINS
Ultrasound is crucial for the diagnosis of twins.
US scanning should begin with a complete imaging sweep of
the uterus.
Always start in the suprapubic area and scan cephalad in a
transverse axial plane until reach the top of the uterine
fundus.
Important sonographic details to note in the first trimester
include:
The number of gestational sacs
The location of the placenta or placentas,
Cont.
The presence and characteristics of the dividing membrane or
membranes,
Amniotic fluid status
The number of yolk sacs and
The fetal hearts.
This informations helps to determine chorionicity
DICHORIONIC
EASY TO DIAGNOSE IN FIRST TRIMESTER
THICK MEMBRANETWIN PEAK SIGN
> 16-20 MG TWIN PEAK DIFFICULT TO FIND
IF IT IS FOUND DICHORIONIC
IF IT IS NOT FOUND STILL NOT EXCLUDED
DICHORIONICITY
CAROLL et al performed study at 10-14 weeks: Sensitivity
97%, specificity 100%
TWIN PEAK SIGN
TRIPLET
MONOCHORIONIC
MONOZIGOTIK TWINS
1. MONOCHORIONIC MONOAMNIOTIC
2. MONOCHORIONIC DIAMNIOTIC
λ --- LAMBDA SIGN
T APPEARANCE
>9 MINGGU : LAMBDA SIGN
MONOKORIONIK
MONOAMNIOTIK
DUPLICATA INCOMPLETA
DICEPHALUS
TYPES OF CONJOINED TWIN
DISCORDANT TWINS
STRUCTURAL CARDIAC ABNORMALITIES IN
MONOZYGOTIC TWINS WITHOUT TTTS ARE 4 X
ESTIMATED FETAL WEIGHT < 20%
AC > 20%
BPD > 6MM
HEAD PERIMETER DIFFERENT >5%
S/D RASIO >15%
DISCORDANT TWIN
FETUS PROBLEM
CONGENITAL ANOMALY (15-20%)
SINGLETON 1,2%, MULTIPEL 2,1%
CHROMOSOM ABNORMALITY
SPONTAN REDUCTION OF THE
FETUS
7 WEEKS : 71 % > 20 WEEKS
7-9 WWKS > 84%
VANISHING TWIN
TTTS (TWIN TO TWIN TRANSFUSION
SYNDROME)
PATOLOGIS
ABNORMAL ANASTOMOSIS OF PLACENTA
VASCULAR
DONOR: ANEMIC, HIPOVOLEMIC , IUGR, URINE
PRODUCTION DECREASED
RESIPIEN: HIPERVOLEMIC, HIPERSYSTEMIC,
HIDROPS
DIAGNOSIS
MONOCHORIONIC
SAME SEX
SECOND TRIMESTER, UNBALANCED AMNIOTIC
FLUID
DISCORDANT GROWTH,RESIPIEN >DONOR
DONOR BLADDER FOUND, RESIPIEN BLADDER
DILATED
HIDROPS
ACARDIAC TWIN/TWIN REVERSE ARTERIAL
PERFUSION (TRAP)
1% OF MONOCHORIONIC TWIN
ANASTOMOSIS ARTERI-ARTERI, VENA-VENA.
DELAYED HEART FUNCTION
CHROMOSOM ABNORMALITY
DOPPLER: REVERSE ARTERIAL PERFUSION
ACARDIAC TWIN
FETUS IN FETU
0,02:10.000, 5% FROM CONJOINED TWIN
DEFFECT PRIMORDIAL GROWTH
ABNORMAL GERM CELL GROWTH.
STRUCTURAL ABNORMALITY FOUND.