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LIGAMENT
Presented
by:
Ishu
Kansal
CEMENTU
M
CONTENTS
•Introduction
•Physical characteristics
•Composition
•Classification of cementum
•Cementogenesis
•Mineralization
•Cementum associated cells
•Cementoenamel junction
•Cementodentinal junction
•Functions of cementum
•Developmental anomalies of
cementum
•Abnormalities of cementum
INTRODUCTION
• Calcified, Avascular mesenchymal tissue that forms the
outer covering of the anatomic root.
Carranza 11th edition.
• First demonstrated in 1835 by FRANKE & RASCHKOV, two
pupils of purkinje.
• Begins at the cervical portion of the tooth at the
cementoenamel junction and continues to the apex.
• Two main types-
- Acellular ( primary)
- Cellular ( secondary)
• Both consist of interfibrillar matrix and collagen
fibrils.
PHYSICAL
C H A R A C T E R I S T IC S
• Hardness < Dentin.
• Light yellow in color and lacks luster.
• Lighter in color than dentin, however it may not
be distinguished on basis of color alone.
• Permeability of cellular cementum is greater than that of
acellular cementum. With age, the permeability of
cementum decreases.
• Thinnest at CEMENTOENAMEL JUNCTION (20-50
um)
• Thickest towards the APEX (150-200 um)
COMPOSITION
• Dry weight basis:
– 45-50% inorganic substances which consists of calcium and
phosphate in the form of hydroxyapetite crystals.
– 50-55% organic material and water.
• Organic matrix of cementum consists of :
– Type I collagen ( 90%)
– Type III collagen ( 5% )
– Non collagenous proteins.
• By volume:
45% inorganic
35% organic
20% water
• Two main sources of collagen fibers
1. Sharpeys fibers ( Extrinsic) are the embedded portion
of the principal fibers of periodontal ligament and
formed by fibroblasts.
2. Fibers that belong to the cementum matrix ( intrinsic)
and produced by cementoblast.
• Due to its lower crystallinity of mineral component :
– has the highest Flouride content
– Readily decalcifies in the presence of acidic conditions.
NON COLLAGENOUS
• Non- collagenous proteins- play important
role in matrix deposition, initiation and control of
mineralization and matrix remodelling.
Include: Bone sialoprotein,
osteopontin,tenascin, fibronectin, osteocalcin .
• Proteoglycans- Chondroitin sulphate,hyaluronate,
heparan sulfate, biglycan and osteoadherin.
Growth factors- TGFß, bone morphogenetic
proteins (BMP’s),Platelet derived growth
factors, Osteoprotegerin (OPG).
• CELLULAR CEMENTUM
ACELLULAR CEMENTUM
• Term acellular is UNFORTUNATE because as a
living tissue Cells are an integral part.
• However some layers do not incorporate cells
while other layers do not contain such cells
in their lacunae.
• First to be formed.
• Sharpeys fibers make most of the structure.
• Forms during root formation before tooth
reaches occlusal plane.
• Covers approx. cervical 1/3rd (coronal portion)of
the root.
• Does not contain any cells.
• More calcified.
• Formation is slow
• Arrangement of collagen fibers are more
organized
CELLULAR CEMENTUM
• Forms after the eruption of tooth once it
reaches occlusal plane.
• Its formation is also in response to the
functional demands.
• Sharpeys fibers occupy a smaller
portion.
• Contains cementocytes in lacunae that
communicate with each other by canaliculi.
• Covers apical 2/3rd of the root
• Contains cementocytes
• Its deposition is more rapid
• Collagen fibers are irregularly arranged.
SH R O ED E R & PA G E
1986
C L A S S I F I C A T I O N
• Classified CEMENTUM on the basis of :
– LOCATION
– MORPHOLOGY
– HISTOLOGICAL APPEARANCE
1. Acellular Afibrillar Cementum (AAC)
2. Acellular Exrinsic Fiber Cementum (AEFC)
3. Cellular Intrinsic Fiber Cementum (CIFC)
4. Cellular Mixed Stratified Cementum (CMSC)
5. Intermediate Cementum
A C EL L U L A R A F I B R I L L A R
C E M E N T U M (AAC)
• FIBERS -ABSENT
• CELLS- ABSENT
• FORMED BY-CEMENTOBLASTS
• LOCATION- CORONAL CEMENTUM
• THICKNESS- 1-15μm
ACELLULAR
E X T R I N S I C F IB ER
• C EM EDENSELY
FIBERS- N T UPACKED
M ( A EF C )
BUNDLES OF SHARPEY’S FIBRES
• CELLS-ABSENT
• FORMED BY – FIBROBLASTS
& CEMENTOBLASTS
• LOCATION -CERVICAL THIRD O
F
ROOT
• THICKNESS - 30-230μm
C E L L U L A R
I N T R I N S I C F I B E R
C E M E N T U M ( C I F C )
• FIBERS - INTRINSIC FIBRES
• CELLS - PRESENT
• FORMED BY - CEMENTOBLASTS
• LOCATION - RESORPTION LACUNAE
C E L L U L A R MIXED
ST R A T I F IE D C E M E N T U M
(C M SC
)
• FIBERS- EXTRINSIC
SHARPEY’S &
INTRINSIC FIBRES
&
• CELLS - PRESENT
rd
•• FORMED
LOCATIONBY- APICAL
- FIBROBLASTS
1/3
CEMENTOBLASTS
OF ROOT & FURCATION
• THICKNESS - 100 -1000μm
INTERMEDIATE CEMENTUM
Cementocytes
CEMENTOBLAST S
Derived from dental
follicle.
Transformation of
mesenchymal cells of
dental follicle.
Cemento-progenitor cells
synthesize collagen and
protein polysaccharide.
These cells have
numerous mitochondria,
a well formed Golgi-
apparatus and large
amounts of granular
endoplasmic reticulum.
Histological observation of areas of root resorption has
shown that cementoblasts can arise wherever viable
dentin is exposed to the soft tissue of the periodontal
ligament. Induction of cementoblasts from periodontal
ligament cells can apparently take place throughout life,
as evidenced by physiological areas of cemental repair.
• ADAPTATION
• IDIOPATHIC
• Cementum resorption appears microscopically as
baylike concavities in the root surface.
• Multinucleated giant cells and large mononuclear
macrophages are generally found adjacent to
cementum undergoing active resorption.
• Cementum repair can occur in devitalized as well
as in vital teeth.
• Resorption occurs most commonly in apical third
then middle third followed by gingival third.
DEVELOPMENTAL
A N O M A LIES
A SSO C IA T E D W I T H
CEMENTOGENISIS
C EM EN T I C L E S
• Are small, globular masses of cementum
found in approx 35% of human roots.
• May not be always attached to the cementum
surface but may be located free in Pdl.
• These may result from microtrauma, when
extra stress on sharpeys fibers causes a tear in
the cementum.
• Are more commonly found in apical & middle
third of root and in root furcation areas
• May develop from calcified epithelial rests;
around small
• spicules of cementum or alveolar bone
traumatically displaced
• into the periodontal ligament; from
calcified
• Sharpey's fibers; and from calcified,
thrombosed vessels
• within the periodontal ligament
EN A M EL PEA R L S
It is largely an age
related
phenomenon
It can be –
Localized to one
tooth
Generalized- affect
• If the overgrowth improves the functional
qualities of the cementum, it is termed as
cementum hypertrophy.
Other systemic
include acromegaly calcinosis,
disturbances thyroid goiter, ,
arthritis etc.
TREATMENT:
Hypercementosis itself does not need treatment.
Hypophosphatasia
Hypophosphatasia is due to an inborn error of
metabolism.The basic disorder is a deficiency of enzyme
alkaline phosphatase in serum or tissues.
Cemental
Tear
ANKYLOSIS
Fusion of cementum and alveolar bone and obliteration of
the periodontal ligament is called ankylosis. Results in
resorption of root and its replacement by bone tissue.
This condition is uncommon.
Occurs in teeth with cemental resorption.
It represents a form of abnormal repair.
ANKYLOSIS CAN ALSO
OCCUR AFTER:
Chronic periapical infection
Tooth reimplantation
Occlusal trauma
Treatment:
No predictable treatment can be suggested.
Alvelodental
Gomphosis
ligament
Dental
Pericementum
periosteum
ELECTRON MICROSCOPIC
STRUCTURE