Cardiomyopathy Disease of the heart muscle Cause is unknown Occurs in only 10-20 per 100,000 Results in 30,000 deaths/year 3 types Dilated – both ventricles Hypertrophic – usually die by age 40 Restrictive – rarest Cardiomyopathy Characterized by left and right ventricular failure Some may be asymptomatic for years and others have acute onset Stroke volume and cardiac output are decreased Atypical chest pain which occurs at rest Progressive and chronic disease Cardiomyopathy, cont. Signs and symptoms are dependent upon the type Dilated Dyspnea Fatigue Left sided heart failure Cardiomyopathy Mitral regurgitation (S1 and S2 sounds heard) Hypertropic Syncope Ankle edema Orthopnea Angina Restrictive Exercise intolerance Dyspnea Fatigue Right sided heart failure S3 and S4 Cardiomyopathy, cont. Diagnosis Echo - primary Angiography Radionuclide imaging Dysrhythmias Decreased CO with restrictive Cardiomyopathy, cont Interventions Drugs Diuretics, vasodilators, cardiac glycosides, beta blockers, anticoagulants Surgery Excision of the hypertrophied muscle Mitral valve replacement Cardiomyoplasty – chest muscle wrapped around the heart Heart transplant Nursing Care Assess Dyspnea Cough Edema Dysrhythmias Decreased CO Need lots of family support and teaching about the disease Heart Transplant Transplanted form a donor with comparable weight and ABO compatibility into a recipient less than 6 hours after procurement Donor must be free of infection, no chest trauma and be declared brain dead, and no malignancies Most of the cases of transplant are to patients with cardiomyopathy Patients with a history of noncompliance, depression or inability to cope with stress are not considered good candidates Heart Transplant, cont. Recipient is prepared for Open Heart Surgery and placed on cardiopulmonary bypass and the anterior portions of the patients heart are removed and replaced with the donor heart Post op care is similar to CABG patients Must be protected from infection by isolation Must receive immunosuppressant drugs for life, as well as steroids (Solu-Medrol, CellCept, Prograf, Imuran, Sandimmune) Watch for rejection – temp, malaise, fatigue, dysrhythmias Monitored by endocardial biopsies Complications include Hypertension, ^ cholesterol, obesity, and malignancies Congestive Heart Failure/Left Sided Heart Failure Causes the most hospitalizations in patients over the age of 65 5 million people on the US are living with heart failure Inadequacy of the heart to pump blood throughout the body effectively This deficit causes insufficient perfusion of body tissues with nutrients and oxygen Causes of heart failure Coronary artery disease Acute MI Cardiomyopathy Hypertension COPD Anemia Fluid volume overload Disease of the heart valves CHF, cont. 2 ventricles pump independently Can have right or left sided failure Usually the left side fails 1st and progresses to failure of both ventricles May be acute or chronic May be mild or severe May be systolic or diastolic failure May cause pulmonary edema or enlarged liver Causes retention of sodium and water by the kidneys CHF, cont. Right sided failure May be caused by left ventricle failure, RV infarct, or Pulmonary hypertension Right ventricle is unable to empty completely Increased volume and pressure develops in the systemic veins and systemic vascular congestion develops with peripheral edema Patient may gain fluid weight and have nausea/anorexia, ascites may develop High output failure Caused by increased metabolic needs Septicemia, anemia, and hyperthyroidism CHF, cont. Compensation – how the body responds to maintain adequate cardiac output Sympathetic Renal Ventricular hypertrophy CHF, cont. Diagnostic tests History and physical Chest x-ray shows cardiomegaly with hazy lung fields Echocardiogram will show enlarged heart and poor contraction of ventricles BUN and creatinine ^ Na and Hct may be decreased due to dilution SAO2 may be decreased LFT’s may be elevated B-type Natriuretic peptide (BNP) – produced and released by the ventricles increases CHF, cont. Objective symptoms Objective symptoms Left sided failure Right sided failure Anxious Weight gain Pale Pitting, dependent edema Tachycardia JVD Dyspnea, with crackles, Ascites wheezes Decreased UOP Orthopnea Distended neck veins Non-productive cough N/V, anorexia Later productive cough with frothy, bloody sputum Oliguria Nursing Assessment Vital signs with both apical and radial pulse HOB elevated Peripheral pulses JVD CVP Orientation with GCS Assess for crackles and wheezes Dependent edema Weight Accurate I/O Abdominal girth Assess for client and family emotional status CHF, cont. Medical treatment Treat the cause – hypertension, rhythm problems, valve repair Drugs – cardiac glycosides, diuretics, inotropic agents, vasodilators, ACE inhibitors, beta blockers, Natrecor Diet – restrictions of sodium and increase of K if diuresis is occuring Restriciton of H2O Surgery Cardiomyoplasty Heart transplant Heart reduction surgery Nursing Interventions Client education for home care Must adjust lifestyle May need O2 S/S to report to provider Weight control – contact physician if more than 2 lb weight gain in a week Dietary management Medication review Exercise regimen Need to work with client to balance activity and rest periods Monitor for complications Many have outpatient CHF clinics