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DEFINITION
It refers to the gamut of artificial means used to support ventilation and oxygenation. They encompass all forms of Positive Pressure Ventilation and those modes used to increase airway pressure above atmospheric during spontaneous ventilation
GOALS OF VENTILATION Increase efficiency of breathing Increase oxygenation Improve ventilation/perfusion relationships Decrease work of breathing
Types of Systems
Negative Pressure Ventilator Iron lung Allows long-term ventilation without artificial airway Maintains normal intrathoracic hemodynamics Uncomfortable, limits access to patient
Types of Systems Positive Pressure Ventilator Uses pressures above atmospheric pressure to push air into lungs Requires use of artificial airway Types Pressure cycled Time cycled Volume cycled
Positive Pressure Ventilators Pressure Cycled Terminates inspiration at preset pressure Small, portable, inexpensive Ventilation volume can vary with changes in airway resistance, pulmonary compliance Used for short-term support of patients with no pre-existing thoracic or pulmonary problems
ASSIST
Supported breath is triggered when patient initiates a breath that develops negative pressure below a threshold termed sensitivity
Tidal volume is preset while peak airway pressure is variable Airway pressure is preset while tidal volume is variable Tidal volume delivered then converted to expiration May be prolonged by inspiratory pause declines to predetermined level
LIMIT
VOLUME
PRESSURE
CYCLE OFF
VOLUME TIME
VENTILATOR MODES
Defined by inspiratory events while expiration is treated as independent entity
Otherwise similar to CMV Better tolerated in light sedation Cannot be used in sedated or paralyzed Work of breathing is greater Can predispose to alkalosis if resp. rate is high
Assist Mode
Assist/Control (A/C)
Patient triggers machine to deliver breaths but machine has preset backup rate Patient initiates breath--machine delivers tidal volume If patient does not breathe fast enough, machine takes over at preset rate Tachypnea patients may hyperventilate dangerously
A/C mode
Assist Mode
INTERMITTENT MANDATORY VENTILATION (IMV) Combination of CMV and Spontaneous modified circuit allows continuous flow that allows patient to breathe between machine delivered breath with minimal work of breathing Delivery is regardless of the stage of respiration
Patient breathes on own Machine delivers breaths at preset intervals Patient determines tidal volume of spontaneous breaths Used to wean patients from ventilators patient machine dyssynchrony may lead to lung over distension or fighting with the ventilator Patients with weak respiratory muscles may tire from breathing against machines resistance Largely abandoned in favor of SIMV
However if patient does not breathe then IMV delivers Relative work of breathing is less Does not contribute to central hyperventilation syndrome Does not require sedation or paralysis In COPD patients, hypercarbia is preserved Popular mode for weaning of patients
SIMV mode
PRESSURE SUPPORT VENTILATION Applicable in spontaneous ventilation Initiation of respiration cause rapid flow of gas until selected pressure is crossed and continues until inspiration drive is stopped Advantage over SIMV being support is provided for each breath Requires spontaneous ventilation but spontaneous breath in SIMV mode are also supported Cannot be used in low pulmonary compliance Central hyperventilation syndrome along with resp.alkalosis may develop Monitoring of tidal volume is required
POSITIVE END EXPIRATORY PRESSURE commonly kept at 3 5 cm of water as a substitute for physiological PEEP provided by closed glottis primary goals include: 1. increase functional Residual Capacity 2. distends patent alveoli 3. recruits previously collapsed alveoli 4. In pulmonary edema, may redistribute extra vascular lung water from alveolar capillary interstitium to peribronchial and perihilar interstitium
Disadvantages 1. Alveolar distention and pulmonary dead space increases air trapping, CO2 retention and hypercapnia 2. Not useful in diseased lung 3. Not tolerated in awake patients 4. Increased work of breathing 5. Increased chances of barotrauma Contraindications 1. Severe hemodynamic instability 2. Acute bronchospasm 3. Severe Emphysema 4. Pneumothorax Suspected or present
In patients who are hemodynamically unstable, ensures effective ventilation reducing the number of variables in management
SURGICAL COMPLICATIONS
Hemorrhage Cardiac contusion following TEE, Phrenic Nerve injury
OPERATIVE CATASTROPHE
Cardiac arrest, Malignant hyperthermia, ABO mismatch Gastric Acid aspiration, Major anaphylactic reaction
Ventilator Settings
Tidal volume--10 to 15ml/kg (std = 12 ml/kg) Respiratory rate--initially 10 to 16/minute FiO2--0.21 to 1.0 depending on disease process 100% causes oxygen toxicity and atelectasis in less than 24 hours 40% is safe indefinitely PEEP can be added to stay below 40% Goal is to achieve a PaO2 >60 I:E Ratio--1:2 is good starting point Obstructive disease requires longer expirations Restrictive disease requires longer inspirations
Changes are made according to Arterial Blood Gas reports Maintain with ACV, use sedatives & muscle relaxants if needed Switch to Synchronized Intermittent Mandatory Ventilation (SIMV) mode, reduce respiratory rate gradually and finally switch to Spontaneous mode Extubate the patients when the criteria's are met.
Ventilator Settings
Ancillary adjustments
Inspiratory flow time Temperature adjustments Humidity Trigger sensitivity Peak airway pressure limits Sighs
For Extubation
Vital Capacity > 10 15ml / kg Negative Inspiratory Force > 25 cm water Spontaneous Respiratory Rate < 30 / min D(A-a)O2 <350 TORR ON 100% O2
low chance with peak airway pressure < 45 cm of water high tidal volume may injure alveolar capillary membrane
Oxygen toxicity
incidence and rapidity of oxygen related injury increases with FIO2 > 60%
Ventilator Complications
Renal malfunction Gastric hemorrhage Pulmonary atelectasis Infection Oxygen toxicity Loss of respiratory muscle tone increased intra cranial pressure
Ventilator complications Acute Respiratory Insufficiency present when there is evidence of inadequate oxygenation (PaO2 <60 mm Hg on FIO2 0.5) or Ventilation (PCO2 >50 mm Hg) during mechanical ventilatory support.
Causes: -Ventilator malfunction, improper setting - Endotracheal tube malfunction - Pulmonary Problems Atelectasis, Pulmonary edema, Pneumonia, Pneumothorax, Haemothorax - Low cardiac output states - Aspiration Pneumonia
Management: - Examine the ventilator setting and function, ET Tube, ABGs, CXR - Hand ventilate with 100% O2, increase FIO2 in ventilator until problem is corrected - May require repositioning of ET tube, insertion of Intra thoracic tube - Asses and optimise hemodyanmics - Add Inverse Ratio Ventilation with PEEP increment - Consider sedation and Paralysis in patient Ventilator synchronicity - Treat identifiable causes
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