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Acute suppurative otitis media is an inflammation of the muco-periosteal lining of the middle ear cleft produced by pus forming organisms lasting up to 3wks. Middle ear cleft consists of:
a) b) c) d) Eustachian tube. Tympanic cavity. Mastoid antrum. Mastoid air cells.
i.
AETIOLOGY:
Route of infection:
Via the eustachian tube. Via the tympanic membrane. Blood-borne infection. Age . Socio-economic factors. Climate. Racial factors. Nasopharyngeal tissue masses. Other nasopharyngeal masses:
Polyps. Teratoma. Angiofibroma. Lymphoma. Carcinoma.
ii.
Predisposing factors:
Respiratory disease. Allergy. Pre-existing middle ear effusion. Immunodeficiency syndromes. Chronic systemic disorders. Cleft palate. Primary ciliary dyskinesia.
PATHOLOGY:
Microbiology:
Haemolytic streptococcus. Staphylococcus aureus. Streptococcus pneumoniae. Haemophilus influenzae. Viruses , paving way for pyococcal invasion. Stage of tubo-tympanitis. Stage of acute serous inflammation. Stage of acute suppurative inflammation. Stage of resolution.
Spread of infection:
Retrograde thrombophlebitis. Bone necrosis. Congenital dehiscences. Fracture lines. 1.Mastoiditis . 2.Intracranially causing extradural or subdural abscess, meningitis ,brain abscess, lateral sinus thrombosis & otitic hydrocephalus. 3.To labyrinth causing suppurative labyrinthitis. 4.To facial nerve canal causing facial paralysis. 5.To the neck producing Bezolds or Citellis abscess. 6.To the petrous apex causing petrositis & Gradenigos Synd.
2. LOCAL:
Deep seated throbbing pain in ear. Deafness. Aural discharge. H/O previous upper respiratory tract infection. 5. Complain of giddiness.
HYPERAEMIC BULGING TM & INJECTION OF THE VASCULATURE ALONG THE MALLEUS HANDLE
1. 2.
I.
SIGNS: GENERAL:
Temperature: 103 - 104F. Pulse rate proportionately increased.
LOCAL:
Stage of tubotympanitis:
a. b. c. d. e. Leash of blood vessels along the handle of malleus. Injection of pars flaccida. Retracted ear drum. Immobility of the ear drum. Ipsilateral conductive deafness.
II.
a. b. c. d. e. f.
III.
a. b. c. d.
After perforation:
Pus in EAC. Perforation in the TM ( small central perforation in the posterior segment of pars tensa ) . Injection of the residual ear drum. Ipsilateral conductive deafness.
III.
a. b.
Stage of resolution:
Dry perforation in the ear drum. Ipsilateral conductive deafness.
III.
1. 2. 3.
Examination of Nose & throat. Variation in clinical picture in any infection is due to:
Differing virulence of invading microorganisms. Varying host defence. Effectiveness of & compliance in Rx. CHRONIC MYRINGITIS
SIGNS OF COMPLICATIONS:
Tenderness & oedema over the mastoid process. Protuberance of pinna. Sagging of posterosuperior canal wall. Granulation tissue pouting through the perforation. Aural discharge persisting for 3-4wks. Neck stiffness. Examination of CNS to rule out impending I/C complications. Nystagmus in patients with vertigo should be sought. Fistula test carried out.
INVESTIGATIONS
Microbiology:
Ear swab for C/S.
Blood studies:
FBC.
Audiometry:
PTA. Tympanometry.
DIAGNOSIS:
H/O earache. Deafness. Otorrhoea. Preceding H/O URTI. Inflammatory changes found on clinical examination. Otitis externa. Tympanic membrane hyperaemia. Otitis media with effusion ( OME ) . Herpes zoster oticus. Myringitis haemorrhagica bullosa. Other conditions.
DIFFERENTIAL DIAGNOSIS:
AC.OM BULLOUS MYRINGITIS
SEQUELAE:
Persistence of a sterile middle ear effusion. High tone sensorineural deafness. Persistent perforation. Extensive scarring of TM, middle ear adhesions & resorption of ossicles ( Adhesive otitis media ).
TREATMENT: 1. Curative:
Medical :
1. 2. 3. 4. 5. General. Analgesics. Topical. Antibiotics. Decongestants.
Surgical :
1. Myringotomy.
2. 3. 4.
STEPS OF MYRINGOTOMY STAGE 1: BEFORE MYRINGOTOMY 1 STAGE 2: MYRINGOTOME MAKING INCISION STAGE 3: FOLLOWING MYRINGOTOMY PURULENT EXUDATE&BLOOD FLOW INTO EAC 3 4 STAGE 4: ONE MONTH FOLLOWING MYRINGOTOMY 2
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